Interaction of Yersinia pestis with macrophages: limitations in YopJ-dependent apoptosis.
about
Cell death programs in Yersinia immunity and pathogenesisYersinia controls type III effector delivery into host cells by modulating Rho activityYersinia type III effectors perturb host innate immune responsesExploitation of eukaryotic ubiquitin signaling pathways by effectors translocated by bacterial type III and type IV secretion systemsReduced secretion of YopJ by Yersinia limits in vivo cell death but enhances bacterial virulence.Yersinia pestis endowed with increased cytotoxicity is avirulent in a bubonic plague model and induces rapid protection against pneumonic plague.The NlpD lipoprotein is a novel Yersinia pestis virulence factor essential for the development of plague.Neutrophils are resistant to Yersinia YopJ/P-induced apoptosis and are protected from ROS-mediated cell death by the type III secretion systemImmune defense against pneumonic plaguePKC-η-MARCKS Signaling Promotes Intracellular Survival of Unopsonized Burkholderia thailandensisYopJ-promoted cytotoxicity and systemic colonization are associated with high levels of murine interleukin-18, gamma interferon, and neutrophils in a live vaccine model of Yersinia pseudotuberculosis infectionA Yersinia effector with enhanced inhibitory activity on the NF-κB pathway activates the NLRP3/ASC/caspase-1 inflammasome in macrophagesGenome-wide mutant fitness profiling identifies nutritional requirements for optimal growth of Yersinia pestis in deep tissue.YopJ-induced caspase-1 activation in Yersinia-infected macrophages: independent of apoptosis, linked to necrosis, dispensable for innate host defense.How the structural gene products of Yersinia pestis relate to virulence.Early apoptosis of macrophages modulated by injection of Yersinia pestis YopK promotes progression of primary pneumonic plagueDestabilization of YopE by the ubiquitin-proteasome pathway fine-tunes Yop delivery into host cells and facilitates systemic spread of Yersinia enterocolitica in host lymphoid tissuec-KIT signaling is targeted by pathogenic Yersinia to suppress the host immune response.YopP-expressing variant of Y. pestis activates a potent innate immune response affording cross-protection against yersiniosis and tularemia [corrected].Disruption of fas-fas ligand signaling, apoptosis, and innate immunity by bacterial pathogensCircumventing Y. pestis Virulence by Early Recruitment of Neutrophils to the Lungs during Pneumonic Plague.Comparative Proteomic Studies of Yersinia pestis Strains Isolated from Natural Foci in the Republic of Georgia.Macrophage cell death upon intracellular bacterial infection.Interaction between Yersinia pestis and the host immune systemUncovering an Important Role for YopJ in the Inhibition of Caspase-1 in Activated Macrophages and Promoting Yersinia pseudotuberculosis Virulence.Yersinia pestis type III secretion system-dependent inhibition of human polymorphonuclear leukocyte functionCaspase-1 activation in macrophages infected with Yersinia pestis KIM requires the type III secretion system effector YopJ.Manipulation of Interleukin-1β and Interleukin-18 Production by Yersinia pestis Effectors YopJ and YopM and Redundant Impact on Virulence.Modulation of host immune defenses by Aeromonas and Yersinia species: convergence on toxins secreted by various secretion systems.Innate immune response during Yersinia infection: critical modulation of cell death mechanisms through phagocyte activation.Activation and Evasion of Inflammasomes by Yersinia.RcsB positively regulates the Yersinia Ysc-Yop type III secretion system by activating expression of the master transcriptional regulator LcrF.Serogroup-related escape of Yersinia enterocolitica YopE from degradation by the ubiquitin-proteasome pathway.Physiology of Yersinia pestis.Discordance in the effects of Yersinia pestis on the dendritic cell functions manifested by induction of maturation and paralysis of migration.Yersinia pestis versus Yersinia pseudotuberculosis: effects on host macrophages.Yersinia pestis can bypass protective antibodies to LcrV and activation with gamma interferon to survive and induce apoptosis in murine macrophagesDisparity between Yersinia pestis and Yersinia enterocolitica O:8 in YopJ/YopP-dependent functions.The fraction 1 and V protein antigens of Yersinia pestis activate dendritic cells to induce primary T cell responses.Necroptosis of infiltrated macrophages drives Yersinia pestis dispersal within buboes
P2860
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P2860
Interaction of Yersinia pestis with macrophages: limitations in YopJ-dependent apoptosis.
description
2006 nî lūn-bûn
@nan
2006 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Interaction of Yersinia pestis with macrophages: limitations in YopJ-dependent apoptosis.
@ast
Interaction of Yersinia pestis with macrophages: limitations in YopJ-dependent apoptosis.
@en
Interaction of Yersinia pestis with macrophages: limitations in YopJ-dependent apoptosis.
@nl
type
label
Interaction of Yersinia pestis with macrophages: limitations in YopJ-dependent apoptosis.
@ast
Interaction of Yersinia pestis with macrophages: limitations in YopJ-dependent apoptosis.
@en
Interaction of Yersinia pestis with macrophages: limitations in YopJ-dependent apoptosis.
@nl
prefLabel
Interaction of Yersinia pestis with macrophages: limitations in YopJ-dependent apoptosis.
@ast
Interaction of Yersinia pestis with macrophages: limitations in YopJ-dependent apoptosis.
@en
Interaction of Yersinia pestis with macrophages: limitations in YopJ-dependent apoptosis.
@nl
P2093
P2860
P356
P1476
Interaction of Yersinia pestis with macrophages: limitations in YopJ-dependent apoptosis.
@en
P2093
Avigdor Shafferman
Avital Tidhar
Ayelet Zauberman
Baruch Velan
Emanuelle Mamroud
Eytan Elhanany
Raphael Ber
Sara Cohen
Yehuda Flashner
P2860
P304
P356
10.1128/IAI.00097-06
P407
P577
2006-06-01T00:00:00Z