Intensive care unit-acquired weakness (ICUAW) and muscle wasting in critically ill patients with severe sepsis and septic shock - sprookjes - yvandenbroek - TriplyDB

Intensive care unit-acquired weakness (ICUAW) and muscle wasting in critically ill patients with severe sepsis and septic shock

Intensive care unit-acquired weakness (ICUAW) and muscle wasting in critically ill patients with severe sepsis and septic shock

http://www.wikidata.org/entity/Q34697264

about

Inspiratory muscle training and physical training for reducing neuromuscular dysfunction in critically ill adults in intensive care units Dysfunction of respiratory muscles in critically ill patients on the intensive care unit Utility of blood procalcitonin concentration in the management of cancer patients with infections Use of miRNAs as biomarkers in sepsis Highlights of mechanistic and therapeutic cachexia and sarcopenia research 2010 to 2012 and their relevance for cardiology.The Sick and the Weak: Neuropathies/Myopathies in the Critically Ill.The ChIP-seq-defined networks of Bcl-3 gene binding support its required role in skeletal muscle atrophy.Associations between muscle-related cytokines and selected patient outcomes in the ICU Sarcopenia in stroke-facts and numbers on muscle loss accounting for disability after stroke.Non-sedation versus sedation with a daily wake-up trial in critically ill patients receiving mechanical ventilation--effects on physical function: study protocol for a randomized controlled trial: a substudy of the NONSEDA trial.Human limb skeletal muscle wasting and architectural remodeling during five to ten days intubation and ventilation in critical care - an observational study using ultrasound.Poly (ADP-ribose) synthetase inhibitor has a heart protective effect in a rat model of experimental sepsis.Functional Status in ICU Survivors and Out of Hospital Outcomes: A Cohort Study Cardiovascular Function in Intensive Care Medicine or Homo Mensura Est Assessment and predictors of physical functioning post-hospital discharge in survivors of critical illness.Single baseline serum creatinine measurements predict mortality in critically ill patients hospitalized for acute heart failure.Delirium in the ICU and subsequent long-term disability among survivors of mechanical ventilation.Premorbid obesity, but not nutrition, prevents critical illness-induced muscle wasting and weakness.Neuromuscular blockade and skeletal muscle weakness in critically ill patients: time to rethink the evidence?Assessment of intensive care unit-acquired weakness in young and old mice: An E. coli septic peritonitis model.Cachexia: clinical features when inflammation drives malnutrition.Measurement of muscle strength with handheld dynamometer in Intensive Care Unit Response: "Commentary: A Hypothesis for Examining Skeletal Muscle Biopsy-Derived Sarcolemmal nNOSµ as Surrogate for Enteric nNOSα Function". nNOS(skeletal muscle) may be Evidentiary for Enteric NO-Transmission Despite nNOSµ/α Differences.Lipopolysaccharide inhibits myogenic differentiation of C2C12 myoblasts through the Toll-like receptor 4-nuclear factor-κB signaling pathway and myoblast-derived tumor necrosis factor-α Sarcopenia in critically ill patients.Publication trends in cachexia and sarcopenia in elderly heart failure patients.Critical Illness Myopathy (CIM) and Ventilator-Induced Diaphragm Muscle Dysfunction (VIDD): Acquired Myopathies Affecting Contractile Proteins.Insulin ameliorating endotoxaemia-induced muscle wasting is associated with the alteration of hypothalamic neuropeptides and inflammation in rats.Moving upwards - the journal of cachexia, sarcopenia and muscle in 2016.Translational evidence for two distinct patterns of neuroaxonal injury in sepsis: a longitudinal, prospective translational study.[Intensive care unit-acquired weakness in the critically ill : critical illness polyneuropathy and critical illness myopathy].Nurses' perceptions of unpleasant symptoms and signs in ventilated and sedated patients.Outcomes of ICU Patients With a Discharge Diagnosis of Critical Illness Polyneuromyopathy: A Propensity-Matched Analysis.The Japanese Clinical Practice Guidelines for Management of Sepsis and Septic Shock 2016 (J-SSCG 2016).Protein Kinetics and Metabolic Effects Related to Disease States in the Intensive Care Unit.

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Intensive care unit-acquired weakness (ICUAW) and muscle wasting in critically ill patients with severe sepsis and septic shock

http://www.wikidata.org/entity/Q34697264

description

2010 nî lūn-bûn

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2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Intensive care unit-acquired w ...... severe sepsis and septic shock

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Intensive care unit-acquired w ...... severe sepsis and septic shock

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Intensive care unit-acquired weakness

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Intensive care unit-acquired w ...... severe sepsis and septic shock

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Intensive care unit-acquired w ...... severe sepsis and septic shock

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Intensive care unit-acquired weakness

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Intensive care unit-acquired w ...... severe sepsis and septic shock

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Intensive care unit-acquired w ...... severe sepsis and septic shock

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Intensive care unit-acquired weakness

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Journal of Cachexia, Sarcopenia and Muscle

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Intensive care unit-acquired w ...... severe sepsis and septic shock

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Jeffrey Bierbrauer

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The initiation factor eIF3-f is a major target for atrogin1/MAFbx function in skeletal muscle atrophy

Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophy

Early goal-directed therapy in the treatment of severe sepsis and septic shock

Epidemiology of severe sepsis in the United States: analysis of incidence, outcome, and associated costs of care

Hydrocortisone therapy for patients with septic shock

Persistent paralysis in critically ill patients after long-term administration of vecuronium

Identification of ubiquitin ligases required for skeletal muscle atrophy

The epidemiology of sepsis in the United States from 1979 through 2000

Efficacy and safety of recombinant human activated protein C for severe sepsis

FoxO3 coordinately activates protein degradation by the autophagic/lysosomal and proteasomal pathways in atrophying muscle cells

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