Biological consequences of free radical-damaged DNA bases.
about
Senataxin, defective in ataxia oculomotor apraxia type 2, is involved in the defense against oxidative DNA damageMutational analysis of the damage-recognition and catalytic mechanism of human SMUG1 DNA glycosylaseThe crystal structure of human endonuclease VIII-like 1 (NEIL1) reveals a zincless finger motif required for glycosylase activityNovel high-resolution characterization of ancient DNA reveals C > U-type base modification events as the sole cause of post mortem miscoding lesionsRepair activity of base and nucleotide excision repair enzymes for guanine lesions induced by nitrosative stress.Close encounters for the first time: Helicase interactions with DNA damageInflammation-induced cell proliferation potentiates DNA damage-induced mutations in vivoStructural Characterization of a Mouse Ortholog of Human NEIL3 with a Marked Preference for Single-Stranded DNAStructural Characterization of Viral Ortholog of Human DNA Glycosylase NEIL1 Bound to Thymine Glycol or 5-Hydroxyuracil-containing DNAReplication stress in Mammalian cells and its consequences for mitosisSubstrate specificity of human endonuclease III (hNTH1). Effect of human APE1 on hNTH1 activityDifferential specificity of human and Escherichia coli endonuclease III and VIII homologues for oxidative base lesionsOxanine DNA glycosylase activity from Mammalian alkyladenine glycosylaseDNA methylation of oxidative stress genes and cancer risk in the Normative Aging StudyNewly engineered magnetic erythrocytes for sustained and targeted delivery of anti-cancer therapeutic compoundsUnexpected role for Helicobacter pylori DNA polymerase I as a source of genetic variabilityThe oxidative DNA glycosylases of Mycobacterium tuberculosis exhibit different substrate preferences from their Escherichia coli counterpartsApplication of Holistic Liquid Chromatography-High Resolution Mass Spectrometry Based Urinary Metabolomics for Prostate Cancer Detection and Biomarker DiscoveryThe mouse ortholog of NEIL3 is a functional DNA glycosylase in vitro and in vivoFANCJ helicase uniquely senses oxidative base damage in either strand of duplex DNA and is stimulated by replication protein A to unwind the damaged DNA substrate in a strand-specific manner.Mechanistic and biological aspects of helicase action on damaged DNA.Two glycosylase families diffusively scan DNA using a wedge residue to probe for and identify oxidatively damaged bases.Distinct roles of RECQ1 in the maintenance of genomic stability.Modulation of UvrD helicase activity by covalent DNA-protein cross-links.Mitochondrial DNA alterations and reduced mitochondrial function in agingMechanisms of free radical-induced damage to DNA.Mitochondrial DNA damage is associated with damage accrual and disease duration in patients with systemic lupus erythematosusError-Prone Translesion DNA Synthesis by Escherichia coli DNA Polymerase IV (DinB) on Templates Containing 1,2-dihydro-2-oxoadenineEnhanced radiation therapy with internalized polyelectrolyte modified nanoparticlesSingle-stranded breaks in DNA but not oxidative DNA base damages block transcriptional elongation by RNA polymerase II in HeLa cell nuclear extracts.Neil3 and NEIL1 DNA glycosylases remove oxidative damages from quadruplex DNA and exhibit preferences for lesions in the telomeric sequence context.Structural/functional analysis of the human OXR1 protein: identification of exon 8 as the anti-oxidant encoding function.RNA editing changes the lesion specificity for the DNA repair enzyme NEIL1Molecular and cellular functions of the FANCJ DNA helicase defective in cancer and in Fanconi anemiaMutagenicity of furan in female Big Blue B6C3F1 miceEffect of edaravone on radiation-induced brain necrosis in patients with nasopharyngeal carcinoma after radiotherapy: a randomized controlled trialSignificant disparity in base and sugar damage in DNA resulting from neutron and electron irradiationDehydration, deamination and enzymatic repair of cytosine glycols from oxidized poly(dG-dC) and poly(dI-dC)Mitochondrial longevity pathwaysGenomic DNA of Nostoc commune (Cyanobacteria) becomes covalently modified during long-term (decades) desiccation but is protected from oxidative damage and degradation
P2860
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P2860
Biological consequences of free radical-damaged DNA bases.
description
2002 nî lūn-bûn
@nan
2002 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Biological consequences of free radical-damaged DNA bases.
@ast
Biological consequences of free radical-damaged DNA bases.
@en
Biological consequences of free radical-damaged DNA bases.
@nl
type
label
Biological consequences of free radical-damaged DNA bases.
@ast
Biological consequences of free radical-damaged DNA bases.
@en
Biological consequences of free radical-damaged DNA bases.
@nl
prefLabel
Biological consequences of free radical-damaged DNA bases.
@ast
Biological consequences of free radical-damaged DNA bases.
@en
Biological consequences of free radical-damaged DNA bases.
@nl
P1476
Biological consequences of free radical-damaged DNA bases
@en
P2093
Susan S Wallace
P356
10.1016/S0891-5849(02)00827-4
P577
2002-07-01T00:00:00Z