Bile acid recognition by the Clostridium difficile germinant receptor, CspC, is important for establishing infection. - sprookjes - yvandenbroek - TriplyDB

Bile acid recognition by the Clostridium difficile germinant receptor, CspC, is important for establishing infection.

Bile acid recognition by the Clostridium difficile germinant receptor, CspC, is important for establishing infection.

http://www.wikidata.org/entity/Q34722536

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Epigenetic Regulation of Enteric Neurotransmission by Gut Bacteria Gastrointestinal dysbiosis and the use of fecal microbial transplantation in Clostridium difficile infection Germination of spores of Bacillus species: what we know and do not know Clostridium difficile spore biology: sporulation, germination, and spore structural proteins Cross-talk between bile acids and intestinal microbiota in host metabolism and health Characterization of the Dynamic Germination of Individual Clostridium difficile Spores Using Raman Spectroscopy and Differential Interference Contrast Microscopy.Bacterial genome engineering and synthetic biology: combating pathogens Identification of a Novel Lipoprotein Regulator of Clostridium difficile Spore Germination Functional Intestinal Bile Acid 7α-Dehydroxylation by Clostridium scindens Associated with Protection from Clostridium difficile Infection in a Gnotobiotic Mouse Model Chemical and Stress Resistances of Clostridium difficile Spores and Vegetative Cells Functional genomics reveals that Clostridium difficile Spo0A coordinates sporulation, virulence and metabolism From stool transplants to next-generation microbiota therapeutics Muricholic acids inhibit Clostridium difficile spore germination and growth Genome-wide analysis of cell type-specific gene transcription during spore formation in Clostridium difficile.Microbiota transplantation restores normal fecal bile acid composition in recurrent Clostridium difficile infection.Role of the intestinal microbiota in resistance to colonization by Clostridium difficile.Harnessing microbiota to kill a pathogen: Fixing the microbiota to treat Clostridium difficile infections.SpoIIID-mediated regulation of σK function during Clostridium difficile sporulation.Functional characterisation of germinant receptors in Clostridium botulinum and Clostridium sporogenes presents novel insights into spore germination systems.The rest of the story: the microbiome and gastrointestinal infections Clostridium sordellii genome analysis reveals plasmid localized toxin genes encoded within pathogenicity loci Effects of surotomycin on Clostridium difficile viability and toxin production in vitro Clostridium difficile drug pipeline: challenges in discovery and development of new agents Changes in Colonic Bile Acid Composition following Fecal Microbiota Transplantation Are Sufficient to Control Clostridium difficile Germination and Growth.Spore Cortex Hydrolysis Precedes Dipicolinic Acid Release during Clostridium difficile Spore Germination.Consequences of bile salt biotransformations by intestinal bacteria.Colonization Resistance of the Gut Microbiota against Clostridium difficile.Administration of probiotic kefir to mice with Clostridium difficile infection exacerbates disease.Understanding the mechanisms of faecal microbiota transplantation.Clostridium difficile colitis: pathogenesis and host defence Clostridium difficile virulence factors: Insights into an anaerobic spore-forming pathogen.Natural products as mediators of disease.A genetic switch controls the production of flagella and toxins in Clostridium difficile Regulation of Clostridium difficile spore germination by the CspA pseudoprotease domain Immunogenicity and protective efficacy of Clostridium difficile spore proteins.Reexamining the Germination Phenotypes of Several Clostridium difficile Strains Suggests Another Role for the CspC Germinant Receptor.Culturing of 'unculturable' human microbiota reveals novel taxa and extensive sporulation.Characterization of Clostridium difficile Spores Lacking Either SpoVAC or Dipicolinic Acid Synthetase.Detecting Cortex Fragments During Bacterial Spore Germination.Germinants and Their Receptors in Clostridia.

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P2860

Bile acid recognition by the Clostridium difficile germinant receptor, CspC, is important for establishing infection.

http://www.wikidata.org/entity/Q34722536

description

2013 nî lūn-bûn

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2013年の論文

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Bile acid recognition by the C ...... nt for establishing infection.

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Bile acid recognition by the C ...... nt for establishing infection.

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Bile acid recognition by the C ...... nt for establishing infection.

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Bile acid recognition by the C ...... nt for establishing infection.

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Bile acid recognition by the C ...... nt for establishing infection.

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Bile acid recognition by the C ...... nt for establishing infection.

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Bile acid recognition by the C ...... ant for establishing infection

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Charlotte A Allen

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Michael B Francis

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Ritu Shrestha

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P2860

Toxin B is essential for virulence of Clostridium difficile

Structural and functional analysis of the CspB protease required for Clostridium spore germination

Studies on transformation of Escherichia coli with plasmids

Metabolism of bile salts in mice influences spore germination in Clostridium difficile

Bile salt biotransformations by human intestinal bacteria

Targeted and random bacterial gene disruption using a group II intron (targetron) vector containing a retrotransposition-activated selectable marker.

Increase in Clostridium difficile-related mortality rates, United States, 1999-2004.

Genetic requirements for induction of germination of spores of Bacillus subtilis by Ca(2+)-dipicolinate.

The anti-sigma factor TcdC modulates hypervirulence in an epidemic BI/NAP1/027 clinical isolate of Clostridium difficile.

The role of toxin A and toxin B in Clostridium difficile infection.

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Clostridium difficile

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