Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion. - sprookjes - yvandenbroek - TriplyDB

Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.

Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.

http://www.wikidata.org/entity/Q34941213

about

Inflammation in Sickle Cell Disease: Differential and Down-Expressed Plasma Levels of Annexin A1 Protein Genetic and biochemical markers of hydroxyurea therapeutic response in sickle cell anemia.Apolipoprotein A-I and serum amyloid A plasma levels are biomarkers of acute painful episodes in patients with sickle cell disease Telomere length correlates with disease severity and inflammation in sickle cell disease.Nitric oxide protection against murine cerebral malaria is associated with improved cerebral microcirculatory physiology.Exogenous nitric oxide decreases brain vascular inflammation, leakage and venular resistance during Plasmodium berghei ANKA infection in mice.Endothelial activation by platelets from sickle cell anemia patients.Interethnic diversity of the CD209 (rs4804803) gene promoter polymorphism in African but not American sickle cell disease Urinary Bladder Dysfunction in Transgenic Sickle Cell Disease Mice The XmnI polymorphic site 5' to the gene G(γ) in a Brazilian patient with sickle cell anaemia - fetal haemoglobin concentration, haematology and clinical features.Carbon-fiber microelectrode amperometry reveals sickle-cell-induced inflammation and chronic morphine effects on single mast cells Role of innate immunity-triggered pathways in the pathogenesis of Sickle Cell Disease: a meta-analysis of gene expression studies Pulmonary function indices in children with sickle cell anemia in Enugu, south-east Nigeria Sickling cells, cyclic nucleotides, and protein kinases: the pathophysiology of urogenital disorders in sickle cell anemia.Opioid patient controlled analgesia use during the initial experience with the IMPROVE PCA trial: a phase III analgesic trial for hospitalized sickle cell patients with painful episodes Impact of iron overload on interleukin-10 levels, biochemical parameters and oxidative stress in patients with sickle cell anemia Acute silent cerebral ischemic events in children with sickle cell anemia.Role of oxidative stress in the pathogenesis of sickle cell disease.Musculoskeletal complications among children with sickle cell admitted in university of Nigeria teaching hospital ituku - ozalla enugu: a 58 month review.Hypoxia and inflammation in children with sickle cell disease: implications for hippocampal functioning and episodic memory.Role of Exercise-Induced Oxidative Stress in Sickle Cell Trait and Disease.Update on sickle cell disease.Red cell DAMPs and inflammation.Relation between glutathione S-transferase genes (GSTM1, GSTT1, and GSTP1) polymorphisms and clinical manifestations of sickle cell disease in Egyptian patients.Epinephrine modulates BCAM/Lu and ICAM-4 expression on the sickle cell trait red blood cell membrane Simvastatin abrogates inflamed neutrophil adhesive properties, in association with the inhibition of Mac-1 integrin expression and modulation of Rho kinase activity.Increased oxidative stress alters nucleosides metabolite levels in sickle cell anemia.Sleep-disordered breathing in patients with sickle cell disease.Treating pain in sickle cell disease with opioids: clinical advances, ethical pitfalls.Optimizing Outcomes in Pedicle and Free Flap Reconstruction in Patients With Sickle Cell Trait.Influence of the βs haplotype and α-thalassemia on stroke development in a Brazilian population with sickle cell anaemia.Endothelial activation and inflammation biomarkers in children and adolescents with sickle cell disease.Pain in sickle cell disease: the future of acute treatment.Sickle cell disease: a natural model of acute and chronic pain.Association of the SOD2 Polymorphism (Val16Ala) and SOD Activity with Vaso-occlusive Crisis and Acute Splenic Sequestration in Children with Sickle Cell Anemia.A common molecular signature of patients with sickle cell disease revealed by microarray meta-analysis and a genome-wide association study Low nitric oxide level is implicated in sickle cell disease and its complications in Ghana

P2860

Q28553088-64E237AC-2284-4918-8B8A-D16D61EF6F54 Q31141526-D27DDB7D-ED14-4FC3-A8A6-B12442F0526F Q33551127-15858B26-51BB-46AB-A2A3-450253BF4F36 Q33758996-D07C1420-C8F3-45BA-B6EA-282B20E3C8D8 Q34833840-AE709E1A-98DD-4727-9818-049C021E4FC9 Q35054703-FE8CC04C-0E28-411F-8BF2-3719612D2646 Q35097680-22263284-FAA4-41BB-A349-C2CF958C0494 Q35145919-00F53CD3-06A4-4C88-A81E-A2F176993E1B Q35737367-6F87C74F-92E8-4C1B-AAD2-F36922FC7B45 Q35817814-379A340E-0B11-4F04-9797-59A30134F24A Q35836675-927AC39B-8763-4EAF-BE7B-A3A21D5230E0 Q35863787-0D178BBE-634D-4E0D-B113-C0BA0FAB99AD Q35996218-2B241C5B-175F-4092-A980-5C11CD3A7E29 Q36056143-33DA376A-FE8F-4267-AEA7-1595516DA03E Q36069493-27D2306D-254C-4147-BFD1-E787BDE2E03B Q36753262-3C982184-1BFA-443A-8A8D-F941383D4DD6 Q36914931-E68A7F33-B9B9-4B6E-A4A8-46F84210C5DD Q37963311-904CC02A-7D94-4BD5-A02B-BA8A739386AE Q38174899-B0679F32-5730-4679-A7FE-9E7834DD9BD6 Q38205407-39FF184D-4C56-4A96-B656-68E13D27DCE8 Q38669072-EC1A4BFD-EB55-4686-BC1A-E20605BFFDE3 Q38806417-E64439D5-5C92-4F71-BC30-6F8C99DDD5FE Q38850672-F9548693-5AA3-4A40-B45C-644F4BD09A79 Q38992024-CF08B41E-F5A6-46B0-B89A-133FB9304B0D Q42503214-6253C147-27AB-4D83-9CC4-C7A7329D35C3 Q46017785-5E6B23CC-D859-4894-B5BC-05FBBD43F017 Q46416731-4E6588F2-14D0-477A-ACEF-48DB8195C01A Q47656335-8C6180E3-FE00-4D37-8885-AE7143B3E466 Q48070421-FC0EE9C8-329A-48AA-B33A-A6711AEDE640 Q49166208-0A8BC6A4-4992-4B15-BFC4-74A360051677 Q50479766-07C23859-90B2-4E4C-8CF8-D9086EB89263 Q50872342-55151F20-5F35-4F2B-B22A-9A17FB47A8F2 Q51701658-5C85F2FF-2A41-43BF-B04E-DA63094D7F38 Q55053106-EA3E46CD-BBC0-4E07-B5EA-F77A9EB77717 Q55241232-3F945D72-8B2D-4599-AB22-75A4BFE2E1F3 Q56965330-9DFE48C8-9D02-493A-81D5-39D028598787 Q58761537-62FBAD79-26EB-4EE8-A794-C82001F8B24F

P2860

Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.

http://www.wikidata.org/entity/Q34941213

description

2009 nî lūn-bûn

@nan

2009 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年学术文章

@wuu

2009年学术文章

@zh-cn

2009年学术文章

@zh-hans

2009年学术文章

@zh-my

2009年学术文章

@zh-sg

2009年學術文章

@yue

name

Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.

@ast

Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.

@en

Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.

@nl

type

label

Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.

@ast

Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.

@en

Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.

@nl

prefLabel

Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.

@ast

Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.

@en

Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.

@nl

P1433

Q34941213-3964D83E-0F7F-4916-AB1D-A88D48A0487F

P1476

Q34941213-8D261F78-3693-48BD-B705-3F0D4A511B99

P2093

Q34941213-43D262D3-8045-4DC4-BE10-A47C45F2EBD1

Q34941213-5732851F-AEAF-47A6-85FB-BF3D24701EB3

Q34941213-9AE8DE28-9267-4601-B945-75979C6D02AA

P2860

Q34941213-027CD1B6-B25E-461B-8F57-89EE214E68F9

Q34941213-02E11FB6-634D-4A6E-90C5-A974B2DAE4D7

Q34941213-03150C98-476A-4A91-BEAA-FEBBA52ACAEC

Q34941213-04D68131-CBD8-4273-B8E9-D465EF8B3CA3

Q34941213-079756BF-05DD-4F23-821B-E2CA25CC40A6

Q34941213-0E73B066-8139-4D92-A8D9-8A6C3675FF4D

Q34941213-0F758DDB-1F96-4EAC-9EE8-130D81672003

Q34941213-0FCF95B8-8929-4917-9F60-BFE809C4320C

Q34941213-140A3373-42A2-4330-9072-E95274BED398

Q34941213-16384C34-1528-4DD9-B91C-90E0ADEE96F4

P304

Q34941213-A4D22D3B-93C7-4DD1-8FE9-428DC0A919E8

P31

Q34941213-E526E592-674B-42E0-993E-BD8836CC6C32

P356

Q34941213-02755654-2B26-424A-83A8-68C192E3D8E2

P433

Q34941213-99E96AD3-8691-4439-8697-F3A521808917

P478

Q34941213-71EE233B-67E5-4A27-A6A1-88643C961536

P577

Q34941213-740BD23B-8914-4F05-8639-13E0BE45321B

P5875

Q34941213-2FC7F933-746F-4390-923B-89884D613BC1

P698

Q34941213-68249397-8432-4251-936E-B1C6F5C9A4D9

P921

Q34941213-6F818906-B32A-42AA-A4EC-70CB17AEF0E1

Q34941213-731A1C9D-2F6D-4D19-9D6C-FF6212CB292A

P356

03630260802625709

P1433

P1476

Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.

@en

P2093

Carla F Franco-Penteado

http://www.w3.org/2001/XMLSchema#string

Fernando F Costa

http://www.w3.org/2001/XMLSchema#string

Nicola Conran

http://www.w3.org/2001/XMLSchema#string

P2860

Sickle-cell disease

Nitric oxide regulates human eosinophil adhesion mechanisms in vitro by changing integrin expression and activity on the eosinophil cell surface

The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine

Primary role for adherent leukocytes in sickle cell vascular occlusion: a new paradigm

Treatment with NS3623, a novel Cl-conductance blocker, ameliorates erythrocyte dehydration in transgenic SAD mice: a possible new therapeutic approach for sickle cell disease.

Management of sickle cell disease.

Oral magnesium pidolate: effects of long-term administration in patients with sickle cell disease.

Oxygen radical inhibition of nitric oxide-dependent vascular function in sickle cell disease.

Hemolysis-associated endothelial dysfunction mediated by accelerated NO inactivation by decompartmentalized oxyhemoglobin.

Cell-free hemoglobin limits nitric oxide bioavailability in sickle-cell disease.

P304

1-16

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1080/03630260802625709

http://www.w3.org/2001/XMLSchema#string

P433

1

http://www.w3.org/2001/XMLSchema#string

P478

33

http://www.w3.org/2001/XMLSchema#string

P577

2009-01-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

23996649

http://www.w3.org/2001/XMLSchema#string

P698

19205968

http://www.w3.org/2001/XMLSchema#string

P921

pathophysiology

sickle-cell disease