Nogo-A knockdown inhibits hypoxia/reoxygenation-induced activation of mitochondrial-dependent apoptosis in cardiomyocytes.
about
Pathogenic properties of the N-terminal region of cardiac myosin binding protein-C in vitroA novel approach to minimize false discovery rate in genome-wide data analysis.RNA-binding protein RBM20 represses splicing to orchestrate cardiac pre-mRNA processing.Lin28a protects against hypoxia/reoxygenation induced cardiomyocytes apoptosis by alleviating mitochondrial dysfunction under high glucose/high fat conditions.Lycopene protects against hypoxia/reoxygenation-induced apoptosis by preventing mitochondrial dysfunction in primary neonatal mouse cardiomyocytes.Exenatide protects against hypoxia/reoxygenation-induced apoptosis by improving mitochondrial function in H9c2 cells.A hypertrophic cardiomyopathy-associated MYBPC3 mutation common in populations of South Asian descent causes contractile dysfunctionEffects of Senegenin against hypoxia/reoxygenation-induced injury in PC12 cells.Endothelial Nogo-B regulates sphingolipid biosynthesis to promote pathological cardiac hypertrophy during chronic pressure overload.Downregulation of GSTK1 Is a Common Mechanism Underlying Hypertrophic Cardiomyopathy.Cardioprotection by combination of three compounds from ShengMai preparations in mice with myocardial ischemia/reperfusion injury through AMPK activation-mediated mitochondrial fission.Nogo-C regulates cardiomyocyte apoptosis during mouse myocardial infarctionRole of Mitochondria-Associated Endoplasmic Reticulum Membrane in Inflammation-Mediated Metabolic DiseasesClematichinenoside (AR) Attenuates Hypoxia/Reoxygenation-Induced H9c2 Cardiomyocyte Apoptosis via a Mitochondria-Mediated Signaling Pathway.Hypoxia promotes AMP-activated protein kinase (AMPK) and induces apoptosis in mouse osteoblasts.Pyrroloquinoline quinone inhibits oxygen/glucose deprivation-induced apoptosis by activating the PI3K/AKT pathway in cardiomyocytes.Asiatic Acid Attenuates Myocardial Ischemia/Reperfusion Injury via Akt/GSK-3β/HIF-1α Signaling in Rat H9c2 Cardiomyocytes.Clinical implications of plasma Nogo-A levels in patients with coronary heart disease.Exploring the Crosstalk Between and Splicing Machinery Gene Mutations in Dilated Cardiomyopathy
P2860
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P2860
Nogo-A knockdown inhibits hypoxia/reoxygenation-induced activation of mitochondrial-dependent apoptosis in cardiomyocytes.
description
2011 nî lūn-bûn
@nan
2011 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մարտին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Nogo-A knockdown inhibits hypo ...... t apoptosis in cardiomyocytes.
@ast
Nogo-A knockdown inhibits hypo ...... t apoptosis in cardiomyocytes.
@en
Nogo-A knockdown inhibits hypo ...... t apoptosis in cardiomyocytes.
@nl
type
label
Nogo-A knockdown inhibits hypo ...... t apoptosis in cardiomyocytes.
@ast
Nogo-A knockdown inhibits hypo ...... t apoptosis in cardiomyocytes.
@en
Nogo-A knockdown inhibits hypo ...... t apoptosis in cardiomyocytes.
@nl
prefLabel
Nogo-A knockdown inhibits hypo ...... t apoptosis in cardiomyocytes.
@ast
Nogo-A knockdown inhibits hypo ...... t apoptosis in cardiomyocytes.
@en
Nogo-A knockdown inhibits hypo ...... t apoptosis in cardiomyocytes.
@nl
P2093
P2860
P1476
Nogo-A knockdown inhibits hypo ...... nt apoptosis in cardiomyocytes
@en
P2093
G L Kartje
J L Martin
J P Sarkey
P P de Tombe
P2860
P304
P356
10.1016/J.YJMCC.2011.03.004
P577
2011-03-17T00:00:00Z