WISP1/CCN4: a potential target for inhibiting prostate cancer growth and spread to bone.
about
Eyeing the Cyr61/CTGF/NOV (CCN) group of genes in development and diseases: highlights of their structural likenesses and functional dissimilaritiesStem cell guidance through the mechanistic target of rapamycinNovel applications of trophic factors, Wnt and WISP for neuronal repair and regeneration in metabolic diseaseWISP1: Clinical insights for a proliferative and restorative member of the CCN familyWNT-1 inducible signaling pathway protein-1 enhances growth and tumorigenesis in human breast cancer.Controlled delivery of BID protein fused with TAT peptide sensitizes cancer cells to apoptosis.Targeting WISP1 to sensitize esophageal squamous cell carcinoma to irradiation.Matricellular proteins in drug delivery: Therapeutic targets, active agents, and therapeutic localizationMatricellular proteins as regulators of cancer metastasis to bone.CCN family of proteins: critical modulators of the tumor cell microenvironment.The CCN family proteins: modulators of bone development and novel targets in bone-associated tumorsDifferential Expression of Ccn4 and Other Genes Between Metastatic and Non-metastatic EL4 Mouse Lymphoma Cells.High expression of WISP1 in colon cancer is associated with apoptosis, invasion and poor prognosis.Cyr61/CTGF/Nov family proteins in gastric carcinogenesis.Genetics of breast cancer bone metastasis: a sequential multistep pattern.Targeting molecules to medicine with mTOR, autophagy and neurodegenerative disorders.Human pancreatic cancer progression: an anarchy among CCN-siblings.Dual effect of WISP-1 in diverse pathological processes.WISP-1 overexpression upregulates cell proliferation in human salivary gland carcinomas via regulating MMP-2 expression.Knockdown of WISP1 inhibit proliferation and induce apoptosis in ALL Jurkat cells.CCN4/WISP-1 positively regulates chondrogenesis by controlling TGF-β3 function.CCN4/WISP1 controls cutaneous wound healing by modulating proliferation, migration and ECM expression in dermal fibroblasts via α5β1 and TNFα.The role of CCN4/WISP-1 in the cancerous phenotype
P2860
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P2860
WISP1/CCN4: a potential target for inhibiting prostate cancer growth and spread to bone.
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2013 nî lūn-bûn
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2013 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի օգոստոսին հրատարակված գիտական հոդված
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年论文
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WISP1/CCN4: a potential target for inhibiting prostate cancer growth and spread to bone.
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WISP1/CCN4: a potential target for inhibiting prostate cancer growth and spread to bone.
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WISP1/CCN4: a potential target for inhibiting prostate cancer growth and spread to bone.
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label
WISP1/CCN4: a potential target for inhibiting prostate cancer growth and spread to bone.
@ast
WISP1/CCN4: a potential target for inhibiting prostate cancer growth and spread to bone.
@en
WISP1/CCN4: a potential target for inhibiting prostate cancer growth and spread to bone.
@nl
prefLabel
WISP1/CCN4: a potential target for inhibiting prostate cancer growth and spread to bone.
@ast
WISP1/CCN4: a potential target for inhibiting prostate cancer growth and spread to bone.
@en
WISP1/CCN4: a potential target for inhibiting prostate cancer growth and spread to bone.
@nl
P2093
P2860
P50
P1433
P1476
WISP1/CCN4: a potential target for inhibiting prostate cancer growth and spread to bone.
@en
P2093
Aaron C Brown
Agnes D Berendsen
Alfredo Molinolo
Azusa Maeda
Larry W Fisher
Luis F de Castro
Marian F Young
Mitsuaki Ono
P2860
P304
P356
10.1371/JOURNAL.PONE.0071709
P407
P577
2013-08-14T00:00:00Z