Canonical Wnts function as potent regulators of osteogenesis by human mesenchymal stem cells. - sprookjes - yvandenbroek - TriplyDB

Canonical Wnts function as potent regulators of osteogenesis by human mesenchymal stem cells.

Canonical Wnts function as potent regulators of osteogenesis by human mesenchymal stem cells.

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Mutations in WNT1 cause different forms of bone fragility Potential mechanisms underlying the Runx2 induced osteogenesis of bone marrow mesenchymal stem cells The role of microRNAs in bone remodeling Osteogenic differentiation of amniotic fluid mesenchymal stromal cells and their bone regeneration potential Expression of osterix Is Regulated by FGF and Wnt/β-Catenin Signalling during Osteoblast Differentiation Single-pulsed electromagnetic field therapy increases osteogenic differentiation through Wnt signaling pathway and sclerostin downregulation.WNT1 mutations in families affected by moderately severe and progressive recessive osteogenesis imperfecta.Wnt10b deficiency results in age-dependent loss of bone mass and progressive reduction of mesenchymal progenitor cells Wnt/β-catenin pathway regulates cementogenic differentiation of adipose tissue-deprived stem cells in dental follicle cell-conditioned medium Pharmaceutical modulation of canonical Wnt signaling in multipotent stromal cells for improved osteoinductive therapy.Comparative analysis of mouse-induced pluripotent stem cells and mesenchymal stem cells during osteogenic differentiation in vitro Canonical and noncanonical Wnts use a common mechanism to activate completely unrelated coreceptors.The role of small molecules in musculoskeletal regeneration.The effect of low-frequency electromagnetic field on human bone marrow stem/progenitor cell differentiation.Glycogen synthase kinase 3β promotes osteogenic differentiation of murine adipose-derived stromal cells High throughput transcriptome profiling of lithium stimulated human mesenchymal stem cells reveals priming towards osteoblastic lineage.Wnt signaling and the control of human stem cell fate.High-frequency canonical Wnt activation in multiple sarcoma subtypes drives proliferation through a TCF/β-catenin target gene, CDC25A.Microbioreactor array screening of Wnt modulators and microenvironmental factors in osteogenic differentiation of mesenchymal progenitor cells.Inhibition of adipocytogenesis by canonical WNT signaling in human mesenchymal stem cells MiR-27a targets sFRP1 in hFOB cells to regulate proliferation, apoptosis and differentiation.Wnts enhance neurotrophin-induced neuronal differentiation in adult bone-marrow-derived mesenchymal stem cells via canonical and noncanonical signaling pathways MiR-637 maintains the balance between adipocytes and osteoblasts by directly targeting Osterix.Signature of microRNA expression during osteogenic differentiation of bone marrow MSCs reveals a putative role of miR-335-5p in osteoarthritis.Connexin 43 deficiency desensitizes bone to the effects of mechanical unloading through modulation of both arms of bone remodeling Spatial and temporal localization of WNT signaling proteins in a mouse model of distraction osteogenesis DKK1 rescues osteogenic differentiation of mesenchymal stem cells isolated from periodontal ligaments of patients with diabetes mellitus induced periodontitis Epigenetic Library Screen Identifies Abexinostat as Novel Regulator of Adipocytic and Osteoblastic Differentiation of Human Skeletal (Mesenchymal) Stem Cells Aberrant activation of Wnt/β-catenin signaling drives proliferation of bone sarcoma cells.Bioreactors to influence stem cell fate: augmentation of mesenchymal stem cell signaling pathways via dynamic culture systems.Cyclic AMP signaling in bone marrow stromal cells has reciprocal effects on the ability of mesenchymal stem cells to differentiate into mature osteoblasts versus mature adipocytes.TCF3, a novel positive regulator of osteogenesis, plays a crucial role in miR-17 modulating the diverse effect of canonical Wnt signaling in different microenvironments.Activation of GLP-1 Receptor Promotes Bone Marrow Stromal Cell Osteogenic Differentiation through β-Catenin Transient Canonical Wnt Stimulation Enriches Human Bone Marrow Mononuclear Cell Isolates for Osteoprogenitors.Wnt signaling behaves as a "master regulator" in the osteogenic and adipogenic commitment of human amniotic fluid mesenchymal stem cells.A clinically relevant model of osteoinduction: a process requiring calcium phosphate and BMP/Wnt signalling.Development of stepwise osteogenesis-mimicking matrices for the regulation of mesenchymal stem cell functions.Molecular control of nitric oxide synthesis through eNOS and caveolin-1 interaction regulates osteogenic differentiation of adipose-derived stem cells by modulation of Wnt/β-catenin signaling.Angled Growth of the Dental Lamina Is Accompanied by Asymmetrical Expression of the WNT Pathway Receptor Frizzled 6.Estrogen receptor α in osteocytes regulates trabecular bone formation in female mice.

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P2860

Canonical Wnts function as potent regulators of osteogenesis by human mesenchymal stem cells.

http://www.wikidata.org/entity/Q34974487

description

2009 nî lūn-bûn

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2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2009 թվականի ապրիլին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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Canonical Wnts function as pot ...... human mesenchymal stem cells.

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Canonical Wnts function as pot ...... human mesenchymal stem cells.

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Canonical Wnts function as pot ...... human mesenchymal stem cells.

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type

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Canonical Wnts function as pot ...... human mesenchymal stem cells.

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Canonical Wnts function as pot ...... human mesenchymal stem cells.

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Canonical Wnts function as pot ...... human mesenchymal stem cells.

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Canonical Wnts function as pot ...... human mesenchymal stem cells.

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Canonical Wnts function as pot ...... human mesenchymal stem cells.

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Canonical Wnts function as pot ...... human mesenchymal stem cells.

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Gal Akiri

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Guizhong Liu

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Huifang Qiao

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Randy Arroyave

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Sapna Vijayakumar

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Stuart A Aaronson

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P2860

Inhibition of adipogenesis by Wnt signaling

Osteocyte control of bone formation via sclerostin, a novel BMP antagonist

A mutation in the LDL receptor-related protein 5 gene results in the autosomal dominant high-bone-mass trait

Lrp5 controls bone formation by inhibiting serotonin synthesis in the duodenum

Multilineage potential of adult human mesenchymal stem cells

The receptor tyrosine kinase Ror2 is involved in non-canonical Wnt5a/JNK signalling pathway

LDL receptor-related protein 5 (LRP5) affects bone accrual and eye development

High bone density due to a mutation in LDL-receptor-related protein 5

Wnt/beta-catenin signaling in mesenchymal progenitors controls osteoblast and chondrocyte differentiation during vertebrate skeletogenesis

Canonical Wnt/beta-catenin signaling prevents osteoblasts from differentiating into chondrocytes

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