miR-200 regulates PDGF-D-mediated epithelial-mesenchymal transition, adhesion, and invasion of prostate cancer cells. - sprookjes - yvandenbroek - TriplyDB

miR-200 regulates PDGF-D-mediated epithelial-mesenchymal transition, adhesion, and invasion of prostate cancer cells.

miR-200 regulates PDGF-D-mediated epithelial-mesenchymal transition, adhesion, and invasion of prostate cancer cells.

http://www.wikidata.org/entity/Q34988143

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Wnt signaling pathways in urological cancers: past decades and still growing Cancer Stem Cells and Epithelial-to-Mesenchymal Transition (EMT)-Phenotypic Cells: Are They Cousins or Twins?The miRacle in Pancreatic Cancer by miRNAs: Tiny Angels or Devils in Disease Progression Mechanisms of TGFβ-Induced Epithelial-Mesenchymal Transition Induced Pluripotent Stem Cell Therapies for Cervical Spinal Cord Injury Revisiting Epithelial-to-Mesenchymal Transition in Liver Fibrosis: Clues for a Better Understanding of the "Reactive" Biliary Epithelial Phenotype Platelet-derived growth factors (PDGFs) in glomerular and tubulointerstitial fibrosis Emerging roles of PDGF-D in EMT progression during tumorigenesis MicroRNAs that affect prostate cancer: emphasis on prostate cancer in African Americans The miR-200 and miR-221/222 microRNA families: opposing effects on epithelial identity A Quest to Identify Prostate Cancer Circulating Biomarkers with a Bench-to-Bedside Potential Regulating miRNA by natural agents as a new strategy for cancer treatment microRNA-200b as a Switch for Inducible Adult Angiogenesis MicroRNAs targeting prostate cancer stem cells Key nodes of a microRNA network associated with the integrated mesenchymal subtype of high-grade serous ovarian cancer The Regulatory Role of MicroRNAs in EMT and Cancer Role for DNA methylation in the regulation of miR-200c and miR-141 expression in normal and cancer cells Anti-tumor activity of a novel compound-CDF is mediated by regulating miR-21, miR-200, and PTEN in pancreatic cancer TMPRSS2/ERG promotes epithelial to mesenchymal transition through the ZEB1/ZEB2 axis in a prostate cancer model Hypoxia-induced aggressiveness of pancreatic cancer cells is due to increased expression of VEGF, IL-6 and miR-21, which can be attenuated by CDF treatment Comprehensive study of gene and microRNA expression related to epithelial-mesenchymal transition in prostate cancer Circadian Rhythm Disruption in Cancer Biology Platelet-derived growth factor-D promotes fibrogenesis of cardiac fibroblasts.Epithelial mesenchymal-like transition occurs in a subset of cells in castration resistant prostate cancer bone metastases.Epithelial to mesenchymal transition is mechanistically linked with stem cell signatures in prostate cancer cells Epithelial-to-mesenchymal transition in prostate cancer: paradigm or puzzle?Module network inference from a cancer gene expression data set identifies microRNA regulated modules Endometrial miR-200c is altered during transformation into cancerous states and targets the expression of ZEBs, VEGFA, FLT1, IKKβ, KLF9, and FBLN5.Androgen receptor as a driver of therapeutic resistance in advanced prostate cancer.Up-regulation of sonic hedgehog contributes to TGF-β1-induced epithelial to mesenchymal transition in NSCLC cells Cross-talk between miRNA and Notch signaling pathways in tumor development and progression.Phenethyl isothiocyanate (PEITC) suppresses prostate cancer cell invasion epigenetically through regulating microRNA-194.Pancreatic cancer stem cells and EMT in drug resistance and metastasis.Implication of microRNAs in drug resistance for designing novel cancer therapy Emerging roles of PDGF-D signaling pathway in tumor development and progression.PDGF: the nuts and bolts of signalling toolbox.MicroRNAs associated with metastatic prostate cancer MicroRNAs and prostate cancer.MicroRNA roles in beta-catenin pathway.Targeting miRNAs involved in cancer stem cell and EMT regulation: An emerging concept in overcoming drug resistance.

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miR-200 regulates PDGF-D-mediated epithelial-mesenchymal transition, adhesion, and invasion of prostate cancer cells.

http://www.wikidata.org/entity/Q34988143

description

2009 nî lūn-bûn

@nan

2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

miR-200 regulates PDGF-D-media ...... sion of prostate cancer cells.

@ast

miR-200 regulates PDGF-D-media ...... sion of prostate cancer cells.

@en

miR-200 regulates PDGF-D-media ...... sion of prostate cancer cells.

@nl

type

label

miR-200 regulates PDGF-D-media ...... sion of prostate cancer cells.

@ast

miR-200 regulates PDGF-D-media ...... sion of prostate cancer cells.

@en

miR-200 regulates PDGF-D-media ...... sion of prostate cancer cells.

@nl

prefLabel

miR-200 regulates PDGF-D-media ...... sion of prostate cancer cells.

@ast

miR-200 regulates PDGF-D-media ...... sion of prostate cancer cells.

@en

miR-200 regulates PDGF-D-media ...... sion of prostate cancer cells.

@nl

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miR-200 regulates PDGF-D-media ...... sion of prostate cancer cells.

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Aamir Ahmad

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Dejuan Kong

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Fazlul H Sarkar

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Hyeong-Reh Choi Kim

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Sanjeev Banerjee

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Yiwei Li

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Zhiwei Wang

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P2860

The zinc-finger protein slug causes desmosome dissociation, an initial and necessary step for growth factor-induced epithelial-mesenchymal transition

Platelet-derived growth factor D is activated by urokinase plasminogen activator in prostate carcinoma cells.

The miR-200 family inhibits epithelial-mesenchymal transition and cancer cell migration by direct targeting of E-cadherin transcriptional repressors ZEB1 and ZEB2

The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2

A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

SIP1/ZEB2 induces EMT by repressing genes of different epithelial cell-cell junctions

The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

DeltaEF1 is a transcriptional repressor of E-cadherin and regulates epithelial plasticity in breast cancer cells

Vascular endothelial growth factor mediates intracrine survival in human breast carcinoma cells through internally expressed VEGFR1/FLT1

DEAD-box RNA helicase subunits of the Drosha complex are required for processing of rRNA and a subset of microRNAs

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1712-1721

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10.1002/STEM.101

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8

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27

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Fazlul H. Sarkar

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2009-08-01T00:00:00Z

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26309064

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19544444

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3400149

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