Anthrax lethal factor represses glucocorticoid and progesterone receptor activity.
about
Binary bacterial toxins: biochemistry, biology, and applications of common Clostridium and Bacillus proteinsCellular and physiological effects of anthrax exotoxin and its relevance to disease.Glucocorticoid regulation of inflammation and its functional correlates: from HPA axis to glucocorticoid receptor dysfunctionAn overview of anthrax infection including the recently identified form of disease in injection drug usersAnthrax toxin receptor 2-dependent lethal toxin killing in vivoRole of N-terminal amino acids in the potency of anthrax lethal factor.Endocrine perturbation increases susceptibility of mice to anthrax lethal toxin.Analysis of severe adverse events related to the use of mifepristone as an abortifacient.Neural regulation of innate immunity: a coordinated nonspecific host response to pathogens.Respiratory syncytial virus represses glucocorticoid receptor-mediated gene activation.Anthrax lethal toxin disrupts the endothelial permeability barrier through blocking p38 signaling.Anthrax lethal toxin induces endothelial barrier dysfunctionThe glucocorticoid receptor: a revisited target for toxinsContribution of lethal toxin and edema toxin to the pathogenesis of anthrax meningitis.The large clostridial toxins from Clostridium sordellii and C. difficile repress glucocorticoid receptor activity.Brain-immune interactions and disease susceptibility.G protein beta interacts with the glucocorticoid receptor and suppresses its transcriptional activity in the nucleus.Differential modulation of glucocorticoid and progesterone receptor transactivation.The role of glucocorticoids and progestins in inflammatory, autoimmune, and infectious disease.Potential biological targets of Bacillus anthracis in anti-infective approaches against the threat of bioterrorism.Bacillus anthracis lethal toxin represses MMTV promoter activity through transcription factorsCellular and systemic effects of anthrax lethal toxin and edema toxin.MAPping innate immunity.Murine macrophage transcriptional responses to Bacillus anthracis infection and intoxication.Lethality during continuous anthrax lethal toxin infusion is associated with circulatory shock but not inflammatory cytokine or nitric oxide release in rats.
P2860
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P2860
Anthrax lethal factor represses glucocorticoid and progesterone receptor activity.
description
2003 nî lūn-bûn
@nan
2003 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Anthrax lethal factor represses glucocorticoid and progesterone receptor activity.
@ast
Anthrax lethal factor represses glucocorticoid and progesterone receptor activity.
@en
type
label
Anthrax lethal factor represses glucocorticoid and progesterone receptor activity.
@ast
Anthrax lethal factor represses glucocorticoid and progesterone receptor activity.
@en
prefLabel
Anthrax lethal factor represses glucocorticoid and progesterone receptor activity.
@ast
Anthrax lethal factor represses glucocorticoid and progesterone receptor activity.
@en
P2093
P2860
P356
P1476
Anthrax lethal factor represses glucocorticoid and progesterone receptor activity.
@en
P2093
Esther M Sternberg
Jeanette I Webster
Leonardo H Tonelli
S Stoney Simons
P2860
P304
P356
10.1073/PNAS.1036973100
P407
P577
2003-04-30T00:00:00Z