Hepatic SOCS3 expression is strongly associated with non-response to therapy and race in HCV and HCV/HIV infection
about
Host restriction factors for hepatitis C virusDendritic cells: The warriors upfront-turned defunct in chronic hepatitis C infectionInnate immune responses in hepatitis C virus infectionSuppressor of cytokine signaling 3 suppresses hepatitis C virus replication in an mTOR-dependent manner.Induction and evasion of innate antiviral responses by hepatitis C virus.Silencing of microRNA-122 enhances interferon-α signaling in the liver through regulating SOCS3 promoter methylation.Saponin inhibits hepatitis C virus propagation by up-regulating suppressor of cytokine signaling 2Loss and dysregulation of Th17 cells during HIV infection.Insulin resistance predicts re-treatment failure in an efficacy study of peginterferon-α-2a and ribavirin in HIV/HCV co-infected patients.Viral exploitation of host SOCS protein functionsAssociation of genetic variants with rapid fibrosis: progression after liver transplantation for hepatitis C.The role of hepatic expression of STAT1, SOCS3 and PIAS1 in the response of chronic hepatitis C patients to therapySOCS3 mRNA expression and polymorphisms as pretreatment predictor of response to HCV genotype 3a IFN-based treatment.SOCS3, a Major Regulator of Infection and Inflammation.Interferon combination therapy for HIV/hepatitis C virus coinfection.Steatosis and insulin resistance in response to treatment of chronic hepatitis C.Immune Response in Hepatitis B Virus Infection.Hepatitis C virus induces a prediabetic state by directly impairing hepatic glucose metabolism in mice.SOCS3 revisited: a broad regulator of disease, now ready for therapeutic use?Effects of hepatitis C virus on suppressor of cytokine signaling mRNA levels: comparison between different genotypes and core protein sequence analysis.Hepatic IFNL4 expression is associated with non-response to interferon-based therapy through the regulation of basal interferon-stimulated gene expression in chronic hepatitis C patients.Association of suppressor of cytokine signalling 3 polymorphisms with insulin resistance in patients with chronic hepatitis C.Recipient IL28B polymorphism is an important independent predictor of posttransplant diabetes mellitus in liver transplant patients with chronic hepatitis C.Transcriptional response of MxA, PKR and SOCS3 to interferon-based therapy in HCV genotype 4-infected patients and contribution of p53 to host antiviral response.Intrahepatic mRNA levels of SOCS1 and SOCS3 are associated with cirrhosis but do not predict virological response to therapy in chronic hepatitis C.Effect of branched-chain amino acid supplementation on insulin resistance and quality of life in chronic hepatitis C patients.Genetic variants of interferon-stimulated genes and IL-28B as host prognostic factors of response to combination treatment for chronic hepatitis C.
P2860
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P2860
Hepatic SOCS3 expression is strongly associated with non-response to therapy and race in HCV and HCV/HIV infection
description
2009 nî lūn-bûn
@nan
2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Hepatic SOCS3 expression is st ...... e in HCV and HCV/HIV infection
@ast
Hepatic SOCS3 expression is st ...... e in HCV and HCV/HIV infection
@en
type
label
Hepatic SOCS3 expression is st ...... e in HCV and HCV/HIV infection
@ast
Hepatic SOCS3 expression is st ...... e in HCV and HCV/HIV infection
@en
prefLabel
Hepatic SOCS3 expression is st ...... e in HCV and HCV/HIV infection
@ast
Hepatic SOCS3 expression is st ...... e in HCV and HCV/HIV infection
@en
P2093
P2860
P1476
Hepatic SOCS3 expression is st ...... e in HCV and HCV/HIV infection
@en
P2093
Atul K Bhan
Carolina B De Sa Borges
Ethan Weinberg
Kyung-Ah Kim
Raymond T Chung
Run-Xuan Shao
Yoshitaka Kamegaya
P2860
P304
P356
10.1016/J.JHEP.2008.12.021
P50
P577
2009-02-14T00:00:00Z