Moderation of adult depression by a polymorphism in the FKBP5 gene and childhood physical abuse in the general population - sprookjes - yvandenbroek - TriplyDB

Moderation of adult depression by a polymorphism in the FKBP5 gene and childhood physical abuse in the general population

Moderation of adult depression by a polymorphism in the FKBP5 gene and childhood physical abuse in the general population

http://www.wikidata.org/entity/Q35171326

about

Steroid Receptor-Associated Immunophilins: A Gateway to Steroid Signalling Molecular and genetic basis of depression Genetic determinants of depression: recent findings and future directions Schizophrenia in the spectrum of gene-stress interactions: the FKBP5 example Glucocorticoids, epigenetic control and stress resilience The Genetics of Stress-Related Disorders: PTSD, Depression, and Anxiety Disorders Brain-derived neurotrophic factor/FK506-binding protein 5 genotype by childhood trauma interactions do not impact on hippocampal volume and cognitive performance Abuse-related trauma forward medical care in a randomly sampled nationwide population Dexamethasone Treatment Leads to Enhanced Fear Extinction and Dynamic Fkbp5 Regulation in Amygdala Drug addiction and stress-response genetic variability: association study in African Americans.Role of Environmental Confounding in the Association between FKBP5 and First-Episode Psychosis Antidepressant treatment response is modulated by genetic and environmental factors and their interactions.Allele-specific FKBP5 DNA demethylation mediates gene-childhood trauma interactions The common functional FKBP5 variant rs1360780 is associated with altered cognitive function in aged individuals Stress-related genes and heroin addiction: a role for a functional FKBP5 haplotype CREB1 gene polymorphisms combined with environmental risk factors increase susceptibility to major depressive disorder (MDD).FKBP5 genotype-dependent DNA methylation and mRNA regulation after psychosocial stress in remitted depression and healthy controls Life course epidemiology of trauma and related psychopathology in civilian populations.Gene-environment interaction in major depression: focus on experience-dependent biological systems.Glucocorticoid-induced loss of DNA methylation in non-neuronal cells and potential involvement of DNMT1 in epigenetic regulation of Fkbp5.Cross-cultural gene- environment interactions in depression, post-traumatic stress disorder, and the cortisol awakening response: FKBP5 polymorphisms and childhood trauma in South Asia.Glucocorticoid sensitizers Bag1 and Ppid are regulated by adolescent stress in a sex-dependent manner Developmental pathways from child maltreatment to adolescent marijuana dependence: Examining moderation by FK506 binding protein 5 gene (FKBP5).Gene-Stress-Epigenetic Regulation of FKBP5: Clinical and Translational Implications RDoC and translational perspectives on the genetics of trauma-related psychiatric disorders.Genetic association of FKBP5 and CRHR1 with cortisol response to acute psychosocial stress in healthy adults.The role of limbic system irritability in linking history of childhood maltreatment and psychiatric outcomes in low-income, high-risk women: moderation by FK506 binding protein 5 haplotype Cannabinoid receptor genotype moderation of the effects of childhood physical abuse on anhedonia and depression.HPA AXIS RELATED GENES AND RESPONSE TO PSYCHOLOGICAL THERAPIES: GENETICS AND EPIGENETICS.Epigenetic Biomarkers as Predictors and Correlates of Symptom Improvement Following Psychotherapy in Combat Veterans with PTSD.Prevalence of bullying victimisation amongst first-episode psychosis patients and unaffected controls FKBP5 and emotional neglect interact to predict individual differences in amygdala reactivity.Mind and body: how the health of the body impacts on neuropsychiatry.Gene-environment interactions and intermediate phenotypes: early trauma and depression.DNA methylation and childhood maltreatment: from animal models to human studies.Childhood maltreatment and stress-related psychopathology: the epigenetic memory hypothesis The biological effects of childhood trauma.Gene-environment interaction in major depression and antidepressant treatment response.Stress-related depression: neuroendocrine, genetic, and therapeutical aspects.Resilience and measured gene-environment interactions.

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Moderation of adult depression by a polymorphism in the FKBP5 gene and childhood physical abuse in the general population

http://www.wikidata.org/entity/Q35171326

description

2011 nî lūn-bûn

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2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2011年の論文

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Moderation of adult depression ...... buse in the general population

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Moderation of adult depression ...... buse in the general population

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Moderation of adult depression ...... buse in the general population

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Moderation of adult depression ...... buse in the general population

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Moderation of adult depression ...... buse in the general population

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Moderation of adult depression ...... buse in the general population

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Carsten Spitzer

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Matthias Nauck

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P2860

Association of FKBP5 polymorphisms and childhood abuse with risk of posttraumatic stress disorder symptoms in adults

Influence of life stress on depression: moderation by a polymorphism in the 5-HTT gene

Polymorphisms in FKBP5 are associated with increased recurrence of depressive episodes and rapid response to antidepressant treatment

Interaction of FKBP5, a stress-related gene, with childhood trauma increases the risk for attempting suicide.

Is FKBP5 a genetic marker of affective psychosis? A case control study and analysis of disease related traits.

The rationale for corticotropin-releasing hormone receptor (CRH-R) antagonists to treat depression and anxiety.

The corticosteroid receptor hypothesis of depression.

Interaction of FKBP5 with childhood adversity on risk for post-traumatic stress disorder.

Cerebrospinal fluid corticotropin-releasing factor and perceived early-life stress in depressed patients and healthy control subjects.

The role of childhood trauma in the neurobiology of mood and anxiety disorders: preclinical and clinical studies.

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