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A unique C-terminal domain allows retention of matrix metalloproteinase-27 in the endoplasmic reticulumEndometriosis, angiogenesis and tissue factorRegulation of Matrix Metalloproteinase-2 Activity by COX-2-PGE2-pAKT Axis Promotes Angiogenesis in EndometriosisSelective inhibition of prostaglandin E2 receptors EP2 and EP4 inhibits adhesion of human endometriotic epithelial and stromal cells through suppression of integrin-mediated mechanismsNovel therapies targeting endometriosisEMMPRIN is secreted by human uterine epithelial cells in microvesicles and stimulates metalloproteinase production by human uterine fibroblast cellsNeutralizing TIMP1 restores fecundity in a rat model of endometriosis and treating control rats with TIMP1 causes anomalies in ovarian function and embryo development.Dioxin may promote inflammation-related development of endometriosis.Inhibition of histone deacetylase activity in human endometrial stromal cells promotes extracellular matrix remodelling and limits embryo invasion.Endometrial expression of relaxin and relaxin receptor in endometriosisMatrix metalloproteinase-27 is expressed in CD163+/CD206+ M2 macrophages in the cycling human endometrium and in superficial endometriotic lesions.Coding regions of INHBA, SFRP4 and HOXA10 are not implicated in familial endometriosis linked to chromosome 7p13-15.Immune interactions in endometriosis.A critical period of progesterone withdrawal precedes menstruation in macaques.TIMP1 contributes to ovarian anomalies in both an MMP-dependent and -independent manner in a rat model.Association between matrix metalloproteinase-2 and matrix metalloproteinase-9 polymorphisms and endometriosis: A systematic review and meta-analysis.Simvastatin decreases invasiveness of human endometrial stromal cells.Cellular and molecular basis for endometriosis-associated infertility.Investigational drugs for endometriosis.A new isoform of steroid receptor coactivator-1 is crucial for pathogenic progression of endometriosisThe role of endometrium in endometriosis.Nuclear factor-kappab (NF-kappaB): an unsuspected major culprit in the pathogenesis of endometriosis that is still at large?Interplay between Misplaced Müllerian-Derived Stem Cells and Peritoneal Immune Dysregulation in the Pathogenesis of Endometriosis.Simvastatin protects against the development of endometriosis in a nude mouse modelReduced fecundity in female rats with surgically induced endometriosis and in their daughters: a potential role for tissue inhibitors of metalloproteinase 1The role of iron in the pathogenesis of endometriosis.Anti-Endometriotic Effects of Pueraria Flower Extract in Human Endometriotic Cells and Mice.Abnormal regulation of chemokine TECK and its receptor CCR9 in the endometriotic milieu is involved in pathogenesis of endometriosis by way of enhancing invasiveness of endometrial stromal cells.Prostaglandin E2: the master of endometriosis?The role of prostaglandin E2 in endometriosis.MAP kinases and the inflammatory signaling cascade as targets for the treatment of endometriosis?Leptin promotes human endometriotic cell migration and invasion by up-regulating MMP-2 through the JAK2/STAT3 signaling pathway.Inhibition of proliferation of endometrial stromal cells by trichostatin A, RU486, CDB-2914, N-acetylcysteine, and ICI 182780.Adenoviral vector encoding soluble Flt-1 engineered human endometrial mesenchymal stem cells effectively regress endometriotic lesions in NOD/SCID mice.Somatostatin analogs regress endometriotic implants in rats by decreasing implant levels of vascular endothelial growth factor and matrix metaloproteinase 9.Gonadotropin-releasing hormone analogues reduce the proliferation of endometrial stromal cells but not endometriotic cells.Increased circulating MMP-2 levels in infertile patients with moderate and severe pelvic endometriosis.Stimulation of macrophage migration inhibitory factor expression in endometrial stromal cells by interleukin 1, beta involving the nuclear transcription factor NFkappaB.Protein-protein interaction analysis to identify biomarker networks for endometriosis.Interleukin 6 secretion from alternatively activated macrophages promotes the migration of endometriotic epithelial cells.
P2860
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P2860
description
2003 nî lūn-bûn
@nan
2003 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Matrix metalloproteinases and endometriosis.
@ast
Matrix metalloproteinases and endometriosis.
@en
type
label
Matrix metalloproteinases and endometriosis.
@ast
Matrix metalloproteinases and endometriosis.
@en
prefLabel
Matrix metalloproteinases and endometriosis.
@ast
Matrix metalloproteinases and endometriosis.
@en
P2093
P356
P1476
Matrix metalloproteinases and endometriosis.
@en
P2093
Grant R Yeaman
Kaylon L Bruner-Tran
Kevin G Osteen
P304
P356
10.1055/S-2003-41322
P577
2003-05-01T00:00:00Z