Hydrogen peroxide differentially modulates cardiac myocyte nitric oxide synthesis.
about
Implications of glial nitric oxide in neurodegenerative diseasesNitric oxide synthases in heart failureAbnormal Ca(2+) cycling in failing ventricular myocytes: role of NOS1-mediated nitroso-redox balanceSpecific visualization of nitric oxide in the vasculature with two-photon microscopy using a copper based fluorescent probeRole of Ca2+ in the control of H2O2-modulated phosphorylation pathways leading to eNOS activation in cardiac myocytesMeasurement of Reactive Oxygen Species, Reactive Nitrogen Species, and Redox-Dependent Signaling in the Cardiovascular System: A Scientific Statement From the American Heart Association.Inhibition of cellular methyltransferases promotes endothelial cell activation by suppressing glutathione peroxidase 1 protein expression.Resveratrol decreases fructose-induced oxidative stress, mediated by NADPH oxidase via an AMPK-dependent mechanismAcute mechanical stretch promotes eNOS activation in venous endothelial cells mainly via PKA and Akt pathways.Nitric oxide-dependent activation of CaMKII increases diastolic sarcoplasmic reticulum calcium release in cardiac myocytes in response to adrenergic stimulation.P2X4 receptor-eNOS signaling pathway in cardiac myocytes as a novel protective mechanism in heart failure.Oxidative stress induces phosphorylation of neuronal NOS in cardiomyocytes through AMP-activated protein kinase (AMPK)Role of reactive oxygen and nitrogen species in the vascular responses to inflammation.Cluster Differentiating 36 (CD36) Deficiency Attenuates Obesity-Associated Oxidative Stress in the Heart.Treatment with Rhus tripartita extract curtails isoproterenol-elicited cardiotoxicity and oxidative stress in rats.Angiotensin-II and MARCKS: a hydrogen peroxide- and RAC1-dependent signaling pathway in vascular endothelium.A universally conserved ATPase regulates the oxidative stress response in Escherichia coliAlterations in glutathione redox metabolism, oxidative stress, and mitochondrial function in the left ventricle of elderly Zucker diabetic fatty rat heart.Nitric oxide in skeletal muscle: role on mitochondrial biogenesis and function.ROS and endothelial nitric oxide synthase (eNOS)-dependent trafficking of angiotensin II type 2 receptor begets neuronal NOS in cardiac myocytes.Resveratrol Inhibition of Rac1-Derived Reactive Oxygen Species by AMPK Decreases Blood Pressure in a Fructose-Induced Rat Model of Hypertension.Differential Roles of Protein Complexes NOX1-NOXO1 and NOX2-p47phox in Mediating Endothelial Redox Responses to Oscillatory and Unidirectional Laminar Shear Stress.Effects of Diabetes on Salivary Gland Protein Expression of Tetrahydrobiopterin and Nitric Oxide Synthesis and Function.Loss of angiotensin-converting enzyme-2 exacerbates diabetic cardiovascular complications and leads to systolic and vascular dysfunction: a critical role of the angiotensin II/AT1 receptor axisLoss of Apelin exacerbates myocardial infarction adverse remodeling and ischemia-reperfusion injury: therapeutic potential of synthetic Apelin analogues.Caveolin-1 is a critical determinant of autophagy, metabolic switching, and oxidative stress in vascular endothelium.Central role for hydrogen peroxide in P2Y1 ADP receptor-mediated cellular responses in vascular endotheliumCrosstalk between AMPK activation and angiotensin II-induced hypertrophy in cardiomyocytes: the role of mitochondria.AMP-activated protein kinase regulation and biological actions in the heart.Posttranslational modifications of cardiac ryanodine receptors: Ca(2+) signaling and EC-coupling.Pharmacological approaches to the treatment of oxidative stress-induced cardiovascular dysfunctions.Nitric Oxide-Induced Calcium Release: Activation of Type 1 Ryanodine Receptor, a Calcium Release Channel, through Non-Enzymatic Post-Translational Modification by Nitric Oxide.ROS signalling between endothelial cells and cardiac cells.Berberine Pretreatment Confers Cardioprotection Against Ischemia-Reperfusion Injury in a Rat Model of Type 2 Diabetes.Redox homeostasis and age-related deficits in neuromuscular integrity and function.Modulation of stretch-induced myocyte remodeling and gene expression by nitric oxide: a novel role for lipoma preferred partner in myofibrillogenesis.Formation of protein S-nitrosylation by reactive oxygen species.Insulin-dependent metabolic and inotropic responses in the heart are modulated by hydrogen peroxide from NADPH-oxidase isoforms NOX2 and NOX4.
P2860
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P2860
Hydrogen peroxide differentially modulates cardiac myocyte nitric oxide synthesis.
description
2011 nî lūn-bûn
@nan
2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Hydrogen peroxide differentially modulates cardiac myocyte nitric oxide synthesis.
@ast
Hydrogen peroxide differentially modulates cardiac myocyte nitric oxide synthesis.
@en
type
label
Hydrogen peroxide differentially modulates cardiac myocyte nitric oxide synthesis.
@ast
Hydrogen peroxide differentially modulates cardiac myocyte nitric oxide synthesis.
@en
prefLabel
Hydrogen peroxide differentially modulates cardiac myocyte nitric oxide synthesis.
@ast
Hydrogen peroxide differentially modulates cardiac myocyte nitric oxide synthesis.
@en
P2093
P2860
P356
P1476
Hydrogen peroxide differentially modulates cardiac myocyte nitric oxide synthesis.
@en
P2093
Hermann Kalwa
Juliano L Sartoretto
Michael D Pluth
Stephen J Lippard
Thomas Michel
P2860
P304
15792-15797
P356
10.1073/PNAS.1111331108
P407
P577
2011-09-06T00:00:00Z