Inflammatory response following intranasal infection with Mycobacterium avium complex: role of T-cell subsets and gamma interferon.
about
Comparing adjuvanted H28 and modified vaccinia virus ankara expressingH28 in a mouse and a non-human primate tuberculosis modelAdjuvant interferon gamma in patients with pulmonary atypical Mycobacteriosis: a randomized, double-blind, placebo-controlled study.CD4+ T cells but Not CD8+ or gammadelta+ lymphocytes are required for host protection against Mycobacterium avium infection and dissemination through the intestinal route.Host defense against Mycobacterium avium does not have an absolute requirement for major histocompatibility complex class I-restricted T cells.Non-major histocompatibility complex control of antibody isotype and Th1 versus Th2 cytokines during experimental infection of mice with Mycobacterium avium.Infection with Mycobacterium ulcerans induces persistent inflammatory responses in mice.In vitro and in vivo activities of gatifloxacin against Mycobacterium tuberculosis.Interleukin-2 and loss of immunity in experimental Mycobacterium avium infectionGeneralized immunological decline during long-term experimental infection with Mycobacterium avium.Endogenous interleukin-12 is involved in resistance of mice to Mycobacterium avium complex infection.Involvement of reactive oxygen intermediates in tumor necrosis factor alpha-dependent bacteriostasis of Mycobacterium avium.Intranasal infection of beige mice with Mycobacterium avium complex: role of neutrophils and natural killer cells.Cytokine secretion and adhesion molecule expression by granuloma T lymphocytes in Mycobacterium avium infectionLack of the Transcription Factor Hypoxia-Inducible Factor 1α (HIF-1α) in Macrophages Accelerates the Necrosis of Mycobacterium avium-Induced Granulomas.Mycolactone-mediated inhibition of tumor necrosis factor production by macrophages infected with Mycobacterium ulcerans has implications for the control of infectionT cells from lungs and livers of Francisella tularensis-immune mice control the growth of intracellular bacteriaVirulence and immune response induced by Mycobacterium avium complex strains in a model of progressive pulmonary tuberculosis and subcutaneous infection in BALB/c miceBystander activation of CD8+ T lymphocytes during experimental mycobacterial infection.Interaction of Mycobacterium avium with human monocyte-derived dendritic cellsPhenotypic changes in T cell populations during the reactivation of tuberculosis in mice.Reactivation of tuberculosis is associated with a shift from type 1 to type 2 cytokines.Cytokine production by CD4 and CD8 T cells during the growth of Mycobacterium tuberculosis in mice.
P2860
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P2860
Inflammatory response following intranasal infection with Mycobacterium avium complex: role of T-cell subsets and gamma interferon.
description
1995 nî lūn-bûn
@nan
1995 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Inflammatory response followin ...... subsets and gamma interferon.
@ast
Inflammatory response followin ...... subsets and gamma interferon.
@en
type
label
Inflammatory response followin ...... subsets and gamma interferon.
@ast
Inflammatory response followin ...... subsets and gamma interferon.
@en
prefLabel
Inflammatory response followin ...... subsets and gamma interferon.
@ast
Inflammatory response followin ...... subsets and gamma interferon.
@en
P2860
P1476
Inflammatory response followin ...... subsets and gamma interferon.
@en
P2093
P2860
P304
P407
P577
1995-06-01T00:00:00Z