The unfolded protein response is shaped by the NMD pathway - sprookjes - yvandenbroek - TriplyDB

The unfolded protein response is shaped by the NMD pathway

The unfolded protein response is shaped by the NMD pathway

http://www.wikidata.org/entity/Q35595494

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Nonsense-mediated mRNA decay: novel mechanistic insights and biological impact Mechanism and regulation of the nonsense-mediated decay pathway The role of nucleotide composition in premature termination codon recognition.Tissue-specific variation in nonsense mutant transcript level and drug-induced read-through efficiency in the Cln1(R151X) mouse model of INCL Targeting MYC in cancer therapy: RNA processing offers new opportunities.The Antagonistic Gene Paralogs Upf3a and Upf3b Govern Nonsense-Mediated RNA Decay.Proteostasis control by the unfolded protein response Nonsense-mediated mRNA decay: an intricate machinery that shapes transcriptomes.The RNA Surveillance Factor UPF1 Represses Myogenesis via Its E3 Ubiquitin Ligase Activity.Physiological and pathophysiological role of nonsense-mediated mRNA decay.Stress and the nonsense-mediated RNA decay pathway.Control of gene expression through the nonsense-mediated RNA decay pathway.Regulation of the unfolded protein response by noncoding RNA.Proteomic Analysis Reveals Branch-specific Regulation of the Unfolded Protein Response by Nonsense-mediated mRNA Decay.RNA metabolism: putting the brake on the UPR.A Role for the Nonsense-Mediated mRNA Decay Pathway in Maintaining Genome Stability in Caenorhabditis elegans.Nonsense-mediated mRNA decay at the crossroads of many cellular pathways Erratum: Proteostasis control by the unfolded protein response.The nonsense-mediated RNA decay pathway is disrupted in inflammatory myofibroblastic tumors.RNA degradation drives stem cell differentiation.A Upf3b-mutant mouse model with behavioral and neurogenesis defects.Inhibition of nonsense-mediated RNA decay by ER stress.RNA decay, evolution, and the testis.Nuclear mRNA degradation tunes the gain of the unfolded protein response in Saccharomyces cerevisiae.p38 MAPK inhibits nonsense-mediated RNA decay in response to persistent DNA damage in noncycling cells.An RNA decay factor wears a new coat: UPF3B modulates translation termination.Nonsense-mediated mRNA decay: a 'nonsense' pathway makes sense in stem cell biology.Nonsense-mediated mRNA Decay and Cancer.Endoplasmic reticulum stress in the development of multiple myeloma and drug resistance.ICE1 promotes the link between splicing and nonsense-mediated mRNA decay.The loss of SMG1 causes defects in quality control pathways in Physcomitrella patens.

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P2860

The unfolded protein response is shaped by the NMD pathway

http://www.wikidata.org/entity/Q35595494

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2015 nî lūn-bûn

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The unfolded protein response is shaped by the NMD pathway

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The unfolded protein response is shaped by the NMD pathway

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The unfolded protein response is shaped by the NMD pathway

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The unfolded protein response is shaped by the NMD pathway

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Chih-Hong Lou

http://www.w3.org/2001/XMLSchema#string

Heike Kroeger

http://www.w3.org/2001/XMLSchema#string

Jonathan H Lin

http://www.w3.org/2001/XMLSchema#string

Lulu Huang

http://www.w3.org/2001/XMLSchema#string

Miles F Wilkinson

http://www.w3.org/2001/XMLSchema#string

Rachid Karam

http://www.w3.org/2001/XMLSchema#string

P2860

ER stress induces cleavage of membrane-bound ATF6 by the same proteases that process SREBPs

XBP1 mRNA is induced by ATF6 and spliced by IRE1 in response to ER stress to produce a highly active transcription factor

BAX inhibitor-1 is a negative regulator of the ER stress sensor IRE1alpha

Proapoptotic BAX and BAK modulate the unfolded protein response by a direct interaction with IRE1alpha

A competition between stimulators and antagonists of Upf complex recruitment governs human nonsense-mediated mRNA decay

Exon-junction complex components specify distinct routes of nonsense-mediated mRNA decay with differential cofactor requirements

The spliceosome deposits multiple proteins 20-24 nucleotides upstream of mRNA exon-exon junctions

Identification of an Ire1alpha endonuclease specific inhibitor with cytotoxic activity against human multiple myeloma

Sustained translational repression by eIF2α-P mediates prion neurodegeneration

Mutations in UPF3B, a member of the nonsense-mediated mRNA decay complex, cause syndromic and nonsyndromic mental retardation

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scholarly article

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10.15252/EMBR.201439696

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5

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16

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2015-03-25T00:00:00Z

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274091339

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25807986

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4428047

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