Deviant energetic metabolism of glycolytic cancer cells.
about
The clinical relevance of the metabolism of prostate cancer; zinc and tumor suppression: connecting the dotsCancer as a metabolic diseaseMolecular cloning and characterization of a cellular phosphoprotein that interacts with a conserved C-terminal domain of adenovirus E1A involved in negative modulation of oncogenic transformationMetabolic targeting of lactate efflux by malignant glioma inhibits invasiveness and induces necrosis: an in vivo studyFunctional roles of the tetramer organization of malic enzymeExpanding antitumor therapeutic windows by targeting cancer-specific nicotinamide adenine dinucleotide phosphate-biogenesis pathwaysThe genetic/metabolic transformation concept of carcinogenesisGlucose catabolism in cancer cells. Isolation, sequence, and activity of the promoter for type II hexokinaseDeterminants of the dual cofactor specificity and substrate cooperativity of the human mitochondrial NAD(P)+-dependent malic enzyme: functional roles of glutamine 362Metabolic remodeling of malignant gliomas for enhanced sensitization during radiotherapy: an in vitro studyEnergy metabolism in tumor cellsLactate and malignant tumors: a therapeutic target at the end stage of glycolysisGlucose regulates protein catabolism in ras-transformed fibroblasts through a lysosomal-dependent proteolytic pathwayGlutamine deprivation induces abortive s-phase rescued by deoxyribonucleotides in k-ras transformed fibroblasts.Glutamine metabolism is essential for human cytomegalovirus infectionInduction of erythroid differentiation in human erythroleukemia cells by depletion of malic enzyme 2Fumarate analogs act as allosteric inhibitors of the human mitochondrial NAD(P)+-dependent malic enzyme.Metabolic targeting of malignant tumors: small-molecule inhibitors of bioenergetic flux.Knockdown of malic enzyme 2 suppresses lung tumor growth, induces differentiation and impacts PI3K/AKT signaling.Does the oxidative/glycolytic ratio determine proliferation or death in immune recognition?Determinants of nucleotide-binding selectivity of malic enzymeThe intermediary metabolism of the prostate: a key to understanding the pathogenesis and progression of prostate malignancyPrognostic significance of the standardized uptake value of pre-therapeutic (18)F-FDG PET in patients with malignant lymphoma.MicroRNA-126 suppresses mesothelioma malignancy by targeting IRS1 and interfering with the mitochondrial functionDeficiency in glutamine but not glucose induces MYC-dependent apoptosis in human cells.Bioenergetic and antiapoptotic properties of mitochondria from cultured human prostate cancer cell lines PC-3, DU145 and LNCaP.Rapid analysis of glycolytic and oxidative substrate flux of cancer cells in a microplateOncogenic K-Ras decouples glucose and glutamine metabolism to support cancer cell growth.Metabolic remodeling precedes mitochondrial outer membrane permeabilization in human glioma xenograft cellsA small-molecule inhibitor suppresses the tumor-associated mitochondrial NAD(P)+-dependent malic enzyme (ME2) and induces cellular senescence.Mitochondrial function, zinc, and intermediary metabolism relationships in normal prostate and prostate cancer.Differential Utilization of Dietary Fatty Acids in Benign and Malignant Cells of the Prostate.The metabolic profile of tumors depends on both the responsible genetic lesion and tissue type.Antigen receptor-mediated changes in glucose metabolism in B lymphocytes: role of phosphatidylinositol 3-kinase signaling in the glycolytic control of growth.pH, Lactate, and Hypoxia: Reciprocity in Regulating High-Affinity Monocarboxylate Transporter Expression in Glioblastoma'Why do tumour cells glycolyse?': from glycolysis through citrate to lipogenesis.Redox regulation of the epigenetic landscape in cancer: a role for metabolic reprogramming in remodeling the epigenomeSilencing of IkBβ mRNA causes disruption of mitochondrial retrograde signaling and suppression of tumor growth in vivo.Nitric oxide released from JS-K induces cell death by mitotic catastrophe as part of necrosis in glioblastoma multiforme.MicroRNA-126 induces autophagy by altering cell metabolism in malignant mesothelioma.
P2860
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P2860
Deviant energetic metabolism of glycolytic cancer cells.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Deviant energetic metabolism of glycolytic cancer cells.
@ast
Deviant energetic metabolism of glycolytic cancer cells.
@en
type
label
Deviant energetic metabolism of glycolytic cancer cells.
@ast
Deviant energetic metabolism of glycolytic cancer cells.
@en
prefLabel
Deviant energetic metabolism of glycolytic cancer cells.
@ast
Deviant energetic metabolism of glycolytic cancer cells.
@en
P1433
P1476
Deviant energetic metabolism of glycolytic cancer cells.
@en
P2093
Baggetto LG
P304
P356
10.1016/0300-9084(92)90016-8
P577
1992-11-01T00:00:00Z