Development of mechanical hypersensitivity in rats during heroin and ethanol dependence: alleviation by CRF₁ receptor antagonism.
about
Neural mechanisms of pain and alcohol dependenceAnimal models of post-traumatic stress disorder and recent neurobiological insights.Neuropeptide Y (NPY) in the extended amygdala is recruited during the transition to alcohol dependenceAnticipatory 50 kHz ultrasonic vocalizations are associated with escalated alcohol intake in dependent rats.Predator odor stress alters corticotropin-releasing factor-1 receptor (CRF1R)-dependent behaviors in rats.Nicotine dependence produces hyperalgesia: role of corticotropin-releasing factor-1 receptors (CRF1Rs) in the central amygdala (CeA).Corticotropin-releasing factor (CRF) and α 2 adrenergic receptors mediate heroin withdrawal-potentiated startle in ratsChronic CRF1 receptor blockade reduces heroin intake escalation and dependence-induced hyperalgesiaClarifying the roles of homeostasis and allostasis in physiological regulation.Structural reorganization of pyramidal neurons in the medial prefrontal cortex of alcohol dependent rats is associated with altered glial plasticity.Pain is associated with risky drinking over time among HIV-infected persons in St. Petersburg, Russia.Increased alcohol consumption in urocortin 3 knockout mice is unaffected by chronic inflammatory pain.Hypocretin receptor 2 antagonism dose-dependently reduces escalated heroin self-administration in rats.Protracted abstinence from chronic ethanol exposure alters the structure of neurons and expression of oligodendrocytes and myelin in the medial prefrontal cortex.Inflammatory Pain Promotes Increased Opioid Self-Administration: Role of Dysregulated Ventral Tegmental Area μ Opioid Receptors.Corticosteroid-dependent plasticity mediates compulsive alcohol drinking in rats.Divergent regulation of distinct glucocorticoid systems in alcohol dependence.The affective dimension of pain as a risk factor for drug and alcohol addiction.Chronic intermittent voluntary alcohol drinking induces hyperalgesia in Sprague-Dawley rats.Alcohol dependence as a chronic pain disorderThe NK1 receptor antagonist L822429 reduces heroin reinforcement.11β-hydroxysteroid dehydrogenase inhibition as a new potential therapeutic target for alcohol abuseLong-term antagonism of κ opioid receptors prevents escalation of and increased motivation for heroin intake.Neuronal extracellular signal-regulated kinase (ERK) activity as marker and mediator of alcohol and opioid dependence.The Prefrontal Cortex as a Critical Gate of Negative Affect and Motivation in Alcohol Use Disorder.Involvement of delta opioid receptors in alcohol withdrawal-induced mechanical allodynia in male C57BL/6 mice.Pain and Risk Behaviors Among HIV-Infected Persons in St. Petersburg, Russia.Traumatic stress reactivity promotes excessive alcohol drinking and alters the balance of prefrontal cortex-amygdala activityElectroacupuncture Attenuates Hyperalgesia in Rats Withdrawn from Chronic Alcohol Drinking via Habenular Mu Opioid Receptors.Opiate dependence induces cell type-specific plasticity of intrinsic membrane properties in the rat juxtacapsular bed nucleus of stria terminalis (jcBNST).Withdrawal from Chronic Nicotine Exposure Produces Region-Specific Tolerance to Alcohol-Stimulated GluA1 Phosphorylation.Neurobiological Correlates of Pain Avoidance-Like Behavior in Morphine-Dependent and Non-Dependent Rats.Corticotropin-Releasing Factor (CRF) Neurocircuitry and Neuropharmacology in Alcohol Drinking.Compulsive-Like Sufentanil Vapor Self-Administration in Rats.Voluntary induction and maintenance of alcohol dependence in rats using alcohol vapor self-administration.Negative reinforcement via motivational withdrawal is the driving force behind the transition to addiction.Inhibition of Glyoxalase 1 reduces alcohol self-administration in dependent and nondependent rats.Systemic and Intra-Habenular Activation of the Orphan G Protein-Coupled Receptor GPR139 Decreases Compulsive-Like Alcohol Drinking and Hyperalgesia in Alcohol-Dependent Rats.An enzymatic approach reverses nicotine dependence, decreases compulsive-like intake, and prevents relapse
P2860
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P2860
Development of mechanical hypersensitivity in rats during heroin and ethanol dependence: alleviation by CRF₁ receptor antagonism.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Development of mechanical hype ...... n by CRF₁ receptor antagonism.
@ast
Development of mechanical hype ...... n by CRF₁ receptor antagonism.
@en
type
label
Development of mechanical hype ...... n by CRF₁ receptor antagonism.
@ast
Development of mechanical hype ...... n by CRF₁ receptor antagonism.
@en
prefLabel
Development of mechanical hype ...... n by CRF₁ receptor antagonism.
@ast
Development of mechanical hype ...... n by CRF₁ receptor antagonism.
@en
P2093
P2860
P1433
P1476
Development of mechanical hype ...... n by CRF₁ receptor antagonism.
@en
P2093
George F Koob
Gery Schulteis
Joel E Schlosburg
Kaushik K Misra
Leandro F Vendruscolo
Paula E Park
Scott Edwards
Sunmee Wee
P2860
P304
P356
10.1016/J.NEUROPHARM.2011.11.006
P577
2011-11-23T00:00:00Z