Regulation of nucleotide metabolism by mutant p53 contributes to its gain-of-function activities.
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Mutant p53 and ETS2, a Tale of ReciprocityMutant p53 Drives Cancer by Subverting Multiple Tumor Suppression PathwaysMutant p53: One, No One, and One Hundred Thousandp53 as a Regulator of Lipid Metabolism in CancerMutant p53 Protein and the Hippo Transducers YAP and TAZ: A Critical Oncogenic Node in Human Cancers.Systematic screening of isogenic cancer cells identifies DUSP6 as context-specific synthetic lethal target in melanoma.The ETS family of oncogenic transcription factors in solid tumours.The Emerging Hallmarks of Cancer MetabolismCharacterization of cancer-associated missense mutations in MDM2.Mutant p53 protects ETS2 from non-canonical COP1/DET1 dependent degradation.Stromal ETS2 Regulates Chemokine Production and Immune Cell Recruitment during Acinar-to-Ductal Metaplasia.Microphthalmia-associated transcription factor suppresses invasion by reducing intracellular GTP pools.Transcriptional Regulation by Wild-Type and Cancer-Related Mutant Forms of p53.Cancer cell metabolism: the essential role of the nonessential amino acid, glutamine.Molecularly targeted therapies for p53-mutant cancers.Learning causal networks with latent variables from multivariate information in genomic data.Control of Nucleotide Metabolism Enables Mutant p53's Oncogenic Gain-of-Function Activity.Effect of Mutant p53 Proteins on Glycolysis and Mitochondrial Metabolism.Proteasome machinery is instrumental in a common gain-of-function program of the p53 missense mutants in cancer.SUMO2 modification of Aurora B and its impact on follicular development and atresia in the mouse ovary.Potential therapeutic targets of TP53 gene in the context of its classically canonical functions and its latest non-canonical functions in human cancer.KRAB-type zinc-finger proteins PITA and PISA specifically regulate p53-dependent glycolysis and mitochondrial respiration.Paris Polyphylla-Derived Saponins Inhibit Growth of Bladder Cancer Cells by Inducing Mutant P53 Degradation While Up-Regulating CDKN1A Expression.Emerging Non-Canonical Functions and Regulation of p53.Deoxyribonucleotide Triphosphate Metabolism in Cancer and Metabolic Disease.dNTP metabolism links mechanical cues and YAP/TAZ to cell growth and oncogene-induced senescence.Complexes formed by mutant p53 and their roles in breast cancer.Wild-type p53 oligomerizes more efficiently than p53 hot-spot mutants and overcomes mutant p53 gain-of-function via a "dominant-positive" mechanism
P2860
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P2860
Regulation of nucleotide metabolism by mutant p53 contributes to its gain-of-function activities.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Regulation of nucleotide metab ...... s gain-of-function activities.
@ast
Regulation of nucleotide metab ...... s gain-of-function activities.
@en
type
label
Regulation of nucleotide metab ...... s gain-of-function activities.
@ast
Regulation of nucleotide metab ...... s gain-of-function activities.
@en
prefLabel
Regulation of nucleotide metab ...... s gain-of-function activities.
@ast
Regulation of nucleotide metab ...... s gain-of-function activities.
@en
P2093
P2860
P356
P1476
Regulation of nucleotide metab ...... s gain-of-function activities.
@en
P2093
Adriano Chan
Damian G Romero
Elizabeth Dimitrova
Gerard R Boss
Gopalakrishnan Ramakrishnan
Kounosuke Watabe
Krishna C Vallabhaneni
Krishna M Chauhan
Luis A Martinez
Madhusudhan Kollareddy
P2860
P2888
P356
10.1038/NCOMMS8389
P407
P577
2015-06-12T00:00:00Z
P6179
1039946560