A CD36-dependent signaling cascade is necessary for macrophage foam cell formation. - sprookjes - yvandenbroek - TriplyDB

A CD36-dependent signaling cascade is necessary for macrophage foam cell formation.

A CD36-dependent signaling cascade is necessary for macrophage foam cell formation.

http://www.wikidata.org/entity/Q35758193

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CD36 participates in a signaling pathway that regulates ROS formation in murine VSMCs.CD36 protein is involved in store-operated calcium flux, phospholipase A2 activation, and production of prostaglandin E2 CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer Uptake of oxidized low density lipoprotein by CD36 occurs by an actin-dependent pathway distinct from macropinocytosis Absence of CD36 protects against atherosclerosis in ApoE knock-out mice with no additional protection provided by absence of scavenger receptor A I/II CD36: implications in cardiovascular disease Perspectives for Monocyte/Macrophage-Based Diagnostics of Chronic Inflammation SR-BI: Linking Cholesterol and Lipoprotein Metabolism with Breast and Prostate Cancer Generation and biological activities of oxidized phospholipids CD9 tetraspanin interacts with CD36 on the surface of macrophages: a possible regulatory influence on uptake of oxidized low density lipoprotein HSP90 inhibition by 17-DMAG attenuates oxidative stress in experimental atherosclerosis Luminal lipid regulates CD36 levels and downstream signaling to stimulate chylomicron synthesis CD36 is a novel serum amyloid A (SAA) receptor mediating SAA binding and SAA-induced signaling in human and rodent cells Novel functions of PXR in cardiometabolic disease.Apoptotic cell recognition receptors and scavenger receptors.The role of oxidized phospholipids in atherosclerosis.Cytoskeletal control of CD36 diffusion promotes its receptor and signaling function.Oxidized LDL/CD36 interaction induces loss of cell polarity and inhibits macrophage locomotion.Functionalization of gadolinium metallofullerenes for detecting atherosclerotic plaque lesions by cardiovascular magnetic resonance.CD36-specific antibodies block release of HIV-1 from infected primary macrophages and its transmission to T cells.Vav Guanine nucleotide exchange factors regulate atherosclerotic lesion development in mice.Exosome poly-ubiquitin inhibits platelet activation, downregulates CD36 and inhibits pro-atherothombotic cellular functions Oxidized Lipoprotein Uptake Through the CD36 Receptor Activates the NLRP3 Inflammasome in Human Retinal Pigment Epithelial Cells.A specific CD36-dependent signaling pathway is required for platelet activation by oxidized low-density lipoprotein.Soluble CD36 ectodomain binds negatively charged diacylglycerol ligands and acts as a co-receptor for TLR2.A concerted kinase interplay identifies PPARgamma as a molecular target of ghrelin signaling in macrophages.Glucose-oxidized low-density lipoproteins enhance insulin-like growth factor I-stimulated smooth muscle cell proliferation by inhibiting integrin-associated protein cleavage Enhanced macrophage tribbles-1 expression in murine experimental atherosclerosis 4,4'-Diaponeurosporene-Producing Bacillus subtilis Increased Mouse Resistance against Salmonella typhimurium Infection in a CD36-Dependent Manner Inflammation, atherosclerosis, and arterial thrombosis: role of the scavenger receptor CD36 CD36, a scavenger receptor involved in immunity, metabolism, angiogenesis, and behavior.Type 2 scavenger receptor CD36 in platelet activation: the role of hyperlipemia and oxidative stress.Fusobacterium nucleatum-associated beta-defensin inducer (FAD-I): identification, isolation, and functional evaluation CD36/fatty acid translocase, an inflammatory mediator, is involved in hyperlipidemia-induced exacerbation in ischemic brain injury.Lipid-induced insulin resistance is associated with increased monocyte expression of scavenger receptor CD36 and internalization of oxidized LDL.Cancer stem cell-specific scavenger receptor CD36 drives glioblastoma progression.PKCδ-IRAK1 axis regulates oxidized LDL-induced IL-1β production in monocytes.CD36, a scavenger receptor implicated in atherosclerosis.Vav guanine nucleotide exchange factors link hyperlipidemia and a prothrombotic state CD36 deletion improves recovery from spinal cord injury.

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P2860

A CD36-dependent signaling cascade is necessary for macrophage foam cell formation.

http://www.wikidata.org/entity/Q35758193

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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2006年论文

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2006年论文

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name

A CD36-dependent signaling cascade is necessary for macrophage foam cell formation.

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A CD36-dependent signaling cascade is necessary for macrophage foam cell formation.

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type

label

A CD36-dependent signaling cascade is necessary for macrophage foam cell formation.

@ast

A CD36-dependent signaling cascade is necessary for macrophage foam cell formation.

@en

prefLabel

A CD36-dependent signaling cascade is necessary for macrophage foam cell formation.

@ast

A CD36-dependent signaling cascade is necessary for macrophage foam cell formation.

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J.CMET.2006.06.007

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Cell Metabolism

P1476

A CD36-dependent signaling cascade is necessary for macrophage foam cell formation.

@en

P2093

David J Lennon

http://www.w3.org/2001/XMLSchema#string

Evgeny A Podrez

http://www.w3.org/2001/XMLSchema#string

Maria Febbraio

http://www.w3.org/2001/XMLSchema#string

Roy L Silverstein

http://www.w3.org/2001/XMLSchema#string

S Ohidar Rahaman

http://www.w3.org/2001/XMLSchema#string

Stanley L Hazen

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P2860

Macrophage scavenger receptor CD36 is the major receptor for LDL modified by monocyte-generated reactive nitrogen species

Scavenger receptors class A-I/II and CD36 are the principal receptors responsible for the uptake of modified low density lipoprotein leading to lipid loading in macrophages

Oxidized LDL regulates macrophage gene expression through ligand activation of PPARgamma

PPARgamma promotes monocyte/macrophage differentiation and uptake of oxidized LDL

Membrane glycoprotein IV (CD36) is physically associated with the Fyn, Lyn, and Yes protein-tyrosine kinases in human platelets

Targeted disruption of the class B scavenger receptor CD36 protects against atherosclerotic lesion development in mice

Atherosclerosis

Signal transduction by the JNK group of MAP kinases

Src-related protein tyrosine kinases are physically associated with the surface antigen CD36 in human dermal microvascular endothelial cells

Evidence for the presence of oxidatively modified low density lipoprotein in atherosclerotic lesions of rabbit and man

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10.1016/J.CMET.2006.06.007

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3

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16950138

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1855263

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