ACE2 and Microbiota: Emerging Targets for Cardiopulmonary Disease Therapy

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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ACE2 and Microbiota: Emerging Targets for Cardiopulmonary Disease Therapy

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ACE2 and Microbiota: Emerging Targets for Cardiopulmonary Disease Therapy

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ACE2 and Microbiota: Emerging Targets for Cardiopulmonary Disease Therapy

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ACE2 and Microbiota: Emerging Targets for Cardiopulmonary Disease Therapy

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ACE2 and Microbiota: Emerging Targets for Cardiopulmonary Disease Therapy

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ACE2 and Microbiota: Emerging Targets for Cardiopulmonary Disease Therapy

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P1476

ACE2 and Microbiota: Emerging Targets for Cardiopulmonary Disease Therapy

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P2093

Colleen T Cole-Jeffrey

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Meng Liu

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Michael J Katovich

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Mohan K Raizada

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Vinayak Shenoy

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P2860

Heart Disease and Stroke Statistics--2015 Update: A Report From the American Heart Association

A human homolog of angiotensin-converting enzyme. Cloning and functional expression as a captopril-insensitive carboxypeptidase

2013 ACCF/AHA Guideline for the Management of Heart Failure: A Report of the American College of Cardiology Foundation/American Heart Association Task Force on Practice Guidelines

A novel angiotensin-converting enzyme-related carboxypeptidase (ACE2) converts angiotensin I to angiotensin 1-9

Angiotensin-converting enzyme 2 overexpression in the subfornical organ prevents the angiotensin II-mediated pressor and drinking responses and is associated with angiotensin II type 1 receptor downregulation

Determining the transgene containment level provided by chloroplast transformation

ACE2 gene expression is up-regulated in the human failing heart

Evaluation of the functional efficacy of an antioxidative probiotic in healthy volunteers

Angiotensin-converting enzyme inhibitors reduce mortality in hypertension: a meta-analysis of randomized clinical trials of renin-angiotensin-aldosterone system inhibitors involving 158,998 patients

Upregulation of Angiotensin (1-7)-Mediated Signaling Preserves Endothelial Function Through Reducing Oxidative Stress in Diabetes.

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540-550

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scholarly article

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10.1097/FJC.0000000000000307

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English

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2015-08-28T00:00:00Z

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26322922

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4807023

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