Molecular factors and biochemical pathways induced by febrile temperature in intraerythrocytic Plasmodium falciparum parasites.
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Ambivalent Outcomes of Cell Apoptosis: A Barrier or Blessing in Malaria ProgressionExpansion of Lysine-rich Repeats in Plasmodium Proteins Generates Novel Localisation Sequences that Target the Periphery of the Host ErythrocyteA secretory multifunctional serine protease, DegP of Plasmodium falciparum, plays an important role in thermo-oxidative stress, parasite growth and developmentA conserved domain targets exported PHISTb family proteins to the periphery of Plasmodium infected erythrocytesPlasmodium falciparum Plasmodium helical interspersed subtelomeric proteins contribute to cytoadherence and anchor P. falciparum erythrocyte membrane protein 1 to the host cell cytoskeletonSir2a regulates rDNA transcription and multiplication rate in the human malaria parasite Plasmodium falciparumTurning up the heat: heat stress induces markers of programmed cell death in Plasmodium falciparum in vitro.SURFIN4.1, a schizont-merozoite associated protein in the SURFIN family of Plasmodium falciparum.Deciphering the ubiquitin-mediated pathway in apicomplexan parasites: a potential strategy to interfere with parasite virulence.Dynamic RNA profiling in Plasmodium falciparum synchronized blood stages exposed to lethal doses of artesunate.Molecular correlates of experimental cerebral malaria detectable in whole bloodThe effect of mimicking febrile temperature and drug stress on malarial development.Differential, positional-dependent transcriptional response of antigenic variation (var) genes to biological stress in Plasmodium falciparum.Plasmodium falciparum spermidine synthase inhibition results in unique perturbation-specific effects observed on transcript, protein and metabolite levels.Cerebral malaria: insights from host-parasite protein-protein interactionsComparative gene expression profiling of P. falciparum malaria parasites exposed to three different histone deacetylase inhibitorsImpact of protozoan cell death on parasite-host interactions and pathogenesisMetabolomics and malaria biologyGlobal response of Plasmodium falciparum to hyperoxia: a combined transcriptomic and proteomic approach.Plasmodium Helical Interspersed Subtelomeric (PHIST) Proteins, at the Center of Host Cell Remodeling.A programmed cell death pathway in the malaria parasite Plasmodium falciparum has general features of mammalian apoptosis but is mediated by clan CA cysteine proteases.Drug-induced permeabilization of parasite's digestive vacuole is a key trigger of programmed cell death in Plasmodium falciparumOn Programmed Cell Death in Plasmodium falciparum: Status Quo.An integrative analysis of small molecule transcriptional responses in the human malaria parasite Plasmodium falciparumTranscriptional variation in the malaria parasite Plasmodium falciparumTransposons to toxins: the provenance, architecture and diversification of a widespread class of eukaryotic effectorsDetection of viable plasmodium ookinetes in the midguts of anopheles coluzzi using PMA-qrtPCR.Sunlight inhibits growth and induces markers of programmed cell death in Plasmodium falciparum in vitro.Epigenetic dysregulation of virulence gene expression in severe Plasmodium falciparum malaria.Possible association of the Plasmodium falciparum T1526C resa2 gene mutation with severe malaria.Cytoadherence and severe malaria.Molecular Markers of Radiation Induced Attenuation in Intrahepatic Plasmodium falciparum Parasites.Radiation-induced cellular and molecular alterations in asexual intraerythrocytic Plasmodium falciparum.The microneme proteins CTRP and SOAP are not essential for Plasmodium berghei ookinete to oocyst transformation in vitro in a cell free system.Refractive index maps and membrane dynamics of human red blood cells parasitized by Plasmodium falciparum.Host biomarkers and biological pathways that are associated with the expression of experimental cerebral malaria in mice.Febrile temperature leads to significant stiffening of Plasmodium falciparum parasitized erythrocytes.A cross strain Plasmodium falciparum microarray optimized for the transcriptome analysis of Plasmodium falciparum patient derived isolates.Co-inhibition of Plasmodium falciparum S-adenosylmethionine decarboxylase/ornithine decarboxylase reveals perturbation-specific compensatory mechanisms by transcriptome, proteome, and metabolome analyses.Co-ordinated stage-dependent enhancement of Plasmodium falciparum antioxidant enzymes and heat shock protein expression in parasites growing in oxidatively stressed or G6PD-deficient red blood cells.
P2860
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P2860
Molecular factors and biochemical pathways induced by febrile temperature in intraerythrocytic Plasmodium falciparum parasites.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Molecular factors and biochemi ...... asmodium falciparum parasites.
@ast
Molecular factors and biochemi ...... asmodium falciparum parasites.
@en
type
label
Molecular factors and biochemi ...... asmodium falciparum parasites.
@ast
Molecular factors and biochemi ...... asmodium falciparum parasites.
@en
prefLabel
Molecular factors and biochemi ...... asmodium falciparum parasites.
@ast
Molecular factors and biochemi ...... asmodium falciparum parasites.
@en
P2093
P2860
P356
P1476
Molecular factors and biochemi ...... asmodium falciparum parasites.
@en
P2093
Babita Mahajan
Hong Zheng
J David Haynes
J Kathleen Moch
Miranda S M Oakley
Rick Fairhurst
Sanjai Kumar
Thomas F McCutchan
P2860
P304
P356
10.1128/IAI.01236-06
P407
P577
2007-02-05T00:00:00Z