Tissue inflammation and nitric oxide-mediated alterations in cardiovascular function are major determinants of endotoxin-induced insulin resistance
about
Emerging role of chemokine CC motif ligand 4 related mechanisms in diabetes mellitus and cardiovascular disease: friends or foes?Acute low-dose endotoxin treatment results in improved whole-body glucose homeostasis in mice.Advanced glycation end-products decreases expression of endothelial nitric oxide synthase through oxidative stress in human coronary artery endothelial cellsEnhanced Glucose Transport, but not Phosphorylation Capacity, Ameliorates Lipopolysaccharide-Induced Impairments in Insulin-Stimulated Muscle Glucose Uptake.Glucose intolerance after chronic stress is related with downregulated PPAR-γ in adipose tissue.Upregulation of inducible NO synthase by exogenous adenosine in vascular smooth muscle cells activated by inflammatory stimuli in experimental diabetes.Voluntary running exercise protects against sepsis-induced early inflammatory and pro-coagulant responses in aged mice.
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P2860
Tissue inflammation and nitric oxide-mediated alterations in cardiovascular function are major determinants of endotoxin-induced insulin resistance
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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2015年论文
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name
Tissue inflammation and nitric ...... xin-induced insulin resistance
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Tissue inflammation and nitric ...... xin-induced insulin resistance
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Tissue inflammation and nitric ...... xin-induced insulin resistance
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Tissue inflammation and nitric ...... xin-induced insulin resistance
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Tissue inflammation and nitric ...... xin-induced insulin resistance
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Tissue inflammation and nitric ...... xin-induced insulin resistance
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Tissue inflammation and nitric ...... xin-induced insulin resistance
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Lawrence M House
Louise Lantier
Owen P McGuinness
Robert T Morris
Travis J Cyphert
Yolanda F Otero
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P2888
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10.1186/S12933-015-0223-2
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2015-05-20T00:00:00Z
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1019440382