Novel role for mitochondria: protein kinase Ctheta-dependent oxidative signaling organelles in activation-induced T-cell death. - sprookjes - yvandenbroek - TriplyDB

Novel role for mitochondria: protein kinase Ctheta-dependent oxidative signaling organelles in activation-induced T-cell death.

Novel role for mitochondria: protein kinase Ctheta-dependent oxidative signaling organelles in activation-induced T-cell death.

http://www.wikidata.org/entity/Q35856966

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Reactive Oxygen Species Regulate T Cell Immune Response in the Tumor Microenvironment Clinical significance of T cell metabolic reprogramming in cancer T cells and reactive oxygen species Redox regulation of T-cell function: from molecular mechanisms to significance in human health and disease The role of selenium in inflammation and immunity: from molecular mechanisms to therapeutic opportunities Partial complex I deficiency due to the CNS conditional ablation of Ndufa5 results in a mild chronic encephalopathy but no increase in oxidative damage.A respiratory chain controlled signal transduction cascade in the mitochondrial intermembrane space mediates hydrogen peroxide signaling ROS function in redox signaling and oxidative stress VHL-dependent alterations in the secretome of renal cell carcinoma: Association with immune cell response?Investigation of the role of mitochondrial DNA in multiple sclerosis susceptibility.Mitochondrial ion channels/transporters as sensors and regulators of cellular redox signaling.The nonphagocytic NADPH oxidase Duox1 mediates a positive feedback loop during T cell receptor signaling.A transcriptome signature of endothelial lymphatic cells coexists with the chronic oxidative stress signature in radiation-induced post-radiotherapy breast angiosarcomas.TCR-triggered extracellular superoxide production is not required for T-cell activation.Granzyme B-induced mitochondrial ROS are required for apoptosis Different Blood Cell-Derived Transcriptome Signatures in Cows Exposed to Vaccination Pre- or Postpartum.Programmed death-1 controls T cell survival by regulating oxidative metabolism.High-Dose Nicotinamide Suppresses ROS Generation and Augments Population Expansion during CD8(+) T Cell Activation DOR activation inhibits anoxic/ischemic Na+ influx through Na+ channels via PKC mechanisms in the cortex NADH oxidase-dependent CD39 expression by CD8(+) T cells modulates interferon gamma responses via generation of adenosine.Mitochondrial respiratory complex I: structure, function and implication in human diseases.Redox control of leukemia: from molecular mechanisms to therapeutic opportunities.Critical role for BIM in T cell receptor restimulation-induced death Uncoupling protein 2 regulates metabolic reprogramming and fate of antigen-stimulated CD8+ T cells.NOTCH1 promotes T cell leukemia-initiating activity by RUNX-mediated regulation of PKC-θ and reactive oxygen species.Dimethyl fumarate restores apoptosis sensitivity and inhibits tumor growth and metastasis in CTCL by targeting NF-κB.Role of apoptosis-inducing factor (Aif) in the T cell lineage.Reactive oxygen species in the immune system.Mitochondria as oxidative signaling organelles in T-cell activation: physiological role and pathological implications.Redox regulation of T-cell receptor signaling.Host-pathogen interactions in malaria: cross-kingdom signaling and mitochondrial regulation.Pathophysiological mechanisms and therapeutic potentials of macrophages in non-alcoholic steatohepatitis.Mitochondrial H2O2 generated from electron transport chain complex I stimulates muscle differentiation.Roles of reactive oxygen species in cell signaling pathways and immune responses to viral infections.TNF-α mediates mitochondrial uncoupling and enhances ROS-dependent cell migration via NF-κB activation in liver cells.Redox balance of mouse medullary CD4 single-positive thymocytes.The Wilms' tumor suppressor WT1 enhances CD95L expression and promotes activation-induced cell death in leukemic T cells.The role of metabolic reprogramming in T cell fate and function.Granzyme A cleaves a mitochondrial complex I protein to initiate caspase-independent cell death.Lyssavirus phosphoproteins increase mitochondrial complex I activity and levels of reactive oxygen species.

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P2860

Novel role for mitochondria: protein kinase Ctheta-dependent oxidative signaling organelles in activation-induced T-cell death.

http://www.wikidata.org/entity/Q35856966

description

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Novel role for mitochondria: p ...... tivation-induced T-cell death.

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Novel role for mitochondria: p ...... tivation-induced T-cell death.

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Novel role for mitochondria: p ...... tivation-induced T-cell death.

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Molecular and Cellular Biology

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Novel role for mitochondria: p ...... tivation-induced T-cell death.

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Dorothee Süss

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Karsten Gülow

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Marcin Kaminski

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Michael Kiessling

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Peter H Krammer

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P2860

CIA30 complex I assembly factor: a candidate for human complex I deficiency?

A PAK1-PIX-PKL complex is activated by the T-cell receptor independent of Nck, Slp-76 and LAT.

Hydrogen peroxide release from mouse peritoneal macrophages: dependence on sequential activation and triggering

Free radicals in the physiological control of cell function

Delayed mitochondrial dysfunction in excitotoxic neuron death: cytochrome c release and a secondary increase in superoxide production

Antiarthritic activity of the newly developed neutrophil oxidative burst antagonist apocynin

LAT is required for TCR-mediated activation of PLCgamma1 and the Ras pathway

Protein kinase Cdelta targets mitochondria, alters mitochondrial membrane potential, and induces apoptosis in normal and neoplastic keratinocytes when overexpressed by an adenoviral vector

Protein kinase C theta affects Ca2+ mobilization and NFAT cell activation in primary mouse T cells

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3625-3639

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10.1128/MCB.02295-06

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10

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27

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2007-03-05T00:00:00Z

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17339328

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1900004

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