Current trends in targeted therapies for glioblastoma multiforme
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A systematic review of microRNA in glioblastoma multiforme: micro-modulators in the mesenchymal mode of migration and invasionTargeting the epithelial to mesenchymal transition in glioblastoma: the emerging role of MET signalingGlioma initiating cells form a differentiation niche via the induction of extracellular matrices and integrin αVExternal beam re-irradiation, combination chemoradiotherapy, and particle therapy for the treatment of recurrent glioblastomaA nanoparticle carrying the p53 gene targets tumors including cancer stem cells, sensitizes glioblastoma to chemotherapy and improves survival.Integration of pathway structure information into a reweighted partial Cox regression approach for survival analysis on high-dimensional gene expression data.A tree shrew glioblastoma model recapitulates features of human glioblastomaEphrin-B3 supports glioblastoma growth by inhibiting apoptosis induced by the dependence receptor EphA4Metabolic targeting of EGFRvIII/PDK1 axis in temozolomide resistant glioblastoma.The Jak2 small molecule inhibitor, G6, reduces the tumorigenic potential of T98G glioblastoma cells in vitro and in vivo.KML001, a telomere-targeting drug, sensitizes glioblastoma cells to temozolomide chemotherapy and radiotherapy through DNA damage and apoptosis.Tumor treating fields: a new frontier in cancer therapy.Nexus of signaling and endocytosis in oncogenesis driven by non-small cell lung cancer-associated epidermal growth factor receptor mutants.N-(2-hydroxyphenyl)acetamide (NA-2) and Temozolomide synergistically induce apoptosis in human glioblastoma cell line U87.An integrated mRNA and microRNA expression signature for glioblastoma multiforme prognosis.The challenges and the promise of molecular targeted therapy in malignant gliomas.Precursor States of Brain Tumor Initiating Cell Lines Are Predictive of Survival in Xenografts and Associated with Glioblastoma Subtypes.Repurposing antipsychotics as glioblastoma therapeutics: Potentials and challengesmiR-135b suppresses tumorigenesis in glioblastoma stem-like cells impairing proliferation, migration and self-renewal.Expression of the miR-302/367 cluster in glioblastoma cells suppresses tumorigenic gene expression patterns and abolishes transformation related phenotypes.Transmembrane protein CD9 is glioblastoma biomarker, relevant for maintenance of glioblastoma stem cells.A Thermodynamic-Based Interpretation of Protein Expression Heterogeneity in Different Glioblastoma Multiforme Tumors Identifies Tumor-Specific Unbalanced Processes.Mitochondrial Dysfunction in Gliomas: Pharmacotherapeutic Potential of Natural Compounds.Signal transduction molecule patterns indicating potential glioblastoma therapy approaches.Multivoxel magnetic resonance spectroscopy identifies enriched foci of cancer stem-like cells in high-grade gliomas.3-Deoxyschweinfurthin B Lowers Cholesterol Levels by Decreasing Synthesis and Increasing Export in Cultured Cancer Cell Lines.Autoimmunity as a double agent in tumor killing and cancer promotion.Isoform-level gene signature improves prognostic stratification and accurately classifies glioblastoma subtypes.Molecular targets in glioblastoma.MicroR-545 mediates colorectal cancer cells proliferation through up-regulating epidermal growth factor receptor expression in HOTAIR long non-coding RNA dependent.miR-577 inhibits glioblastoma tumor growth via the Wnt signaling pathway.Spatiotemporal genomic architecture informs precision oncology in glioblastoma.Glioblastoma Multiforme: A Review of its Epidemiology and Pathogenesis through Clinical Presentation and TreatmentCausal Mechanistic Regulatory Network for Glioblastoma Deciphered Using Systems Genetics Network Analysis.Small molecule inhibition of Axl receptor tyrosine kinase potently suppresses multiple malignant properties of glioma cells.Immunoexcitatory mechanisms in glioma proliferation, invasion and occasional metastasis.Interferon-β and temozolomide combination therapy for temozolomide monotherapy-refractory malignant gliomas.Decoding the cancer stem cell hypothesis in glioblastoma.Glioblastoma multiforme from diagnosis to death: a prospective, hospital-based, cohort, pilot feasibility study of patient reported symptoms and needs.ATP7B expression in human glioblastoma is related to temozolomide resistance.
P2860
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P2860
Current trends in targeted therapies for glioblastoma multiforme
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
Current trends in targeted therapies for glioblastoma multiforme
@ast
Current trends in targeted therapies for glioblastoma multiforme
@en
type
label
Current trends in targeted therapies for glioblastoma multiforme
@ast
Current trends in targeted therapies for glioblastoma multiforme
@en
prefLabel
Current trends in targeted therapies for glioblastoma multiforme
@ast
Current trends in targeted therapies for glioblastoma multiforme
@en
P2093
P2860
P356
P1476
Current trends in targeted therapies for glioblastoma multiforme
@en
P2093
Atsushi Natsume
Fumiharu Ohka
Toshihiko Wakabayashi
P2860
P304
P356
10.1155/2012/878425
P577
2012-03-05T00:00:00Z