Developmental bisphenol A (BPA) exposure leads to sex-specific modification of hepatic gene expression and epigenome at birth that may exacerbate high-fat diet-induced hepatic steatosis.
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Endocrine causes of nonalcoholic fatty liver diseaseThe Role of Epigenetics in the Latent Effects of Early Life Exposure to Obesogenic Endocrine Disrupting ChemicalsTris(1,3-dichloro-2-propyl)phosphate Induces Genome-Wide Hypomethylation within Early Zebrafish EmbryosEndocrine-disrupting chemicals and fatty liver disease.Polycystic ovary syndrome (PCOS) and endocrine disrupting chemicals (EDCs).Molecular targets of developmental exposure to bisphenol A in diabesity: a focus on endoderm-derived organs.Early life programming and the risk of non-alcoholic fatty liver disease.Maternal bisphenol A exposure alters rat offspring hepatic and skeletal muscle insulin signaling protein abundance.Effects of Endocrine-Disrupting Chemicals and Epigenetic Modifications in Ovarian Cancer: A Review.Effects of Low-Dose Developmental Bisphenol A Exposure on Metabolic Parameters and Gene Expression in Male and Female Fischer 344 Rat Offspring.The Plasticizer Bisphenol A Perturbs the Hepatic Epigenome: A Systems Level Analysis of the miRNome.Epigenetic impact of endocrine disrupting chemicals on lipid homeostasis and atherosclerosis: a pregnane X receptor-centric view.Sex-Specific Modulation of Fetal Adipogenesis by Gestational Bisphenol A and Bisphenol S Exposure.Hepatic Lipid Accumulation and Nrf2 Expression following Perinatal and Peripubertal Exposure to Bisphenol A in a Mouse Model of Nonalcoholic Liver Disease.Adipose Tissue as a Site of Toxin Accumulation.Persistent Endocrine-Disrupting Chemicals and Fatty Liver Disease.Bisphenol a increases risk for presumed non-alcoholic fatty liver disease in Hispanic adolescents in NHANES 2003-2010.Sexually Dimorphic Effects of Early-Life Exposures to Endocrine Disruptors: Sex-Specific Epigenetic Reprogramming as a Potential Mechanism.Endocrine Disruptors and Developmental Origins of Nonalcoholic Fatty Liver Disease.Perinatal bisphenol A exposure increases atherosclerosis in adult male PXR-humanized mice.Obesity Pathogenesis: An Endocrine Society Scientific Statement.Maternal levels of endocrine disrupting chemicals in the first trimester of pregnancy are associated with infant cord blood DNA methylation.Bisphenol-A and metabolic diseases: epigenetic, developmental and transgenerational basis.
P2860
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P2860
Developmental bisphenol A (BPA) exposure leads to sex-specific modification of hepatic gene expression and epigenome at birth that may exacerbate high-fat diet-induced hepatic steatosis.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
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2015年论文
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name
Developmental bisphenol A (BPA ...... iet-induced hepatic steatosis.
@ast
Developmental bisphenol A (BPA ...... iet-induced hepatic steatosis.
@en
type
label
Developmental bisphenol A (BPA ...... iet-induced hepatic steatosis.
@ast
Developmental bisphenol A (BPA ...... iet-induced hepatic steatosis.
@en
prefLabel
Developmental bisphenol A (BPA ...... iet-induced hepatic steatosis.
@ast
Developmental bisphenol A (BPA ...... iet-induced hepatic steatosis.
@en
P2093
P2860
P1476
Developmental bisphenol A (BPA ...... iet-induced hepatic steatosis.
@en
P2093
Nicki J Engeseth
Rita S Strakovsky
Stéphane Lezmi
William G Helferich
P2860
P304
P356
10.1016/J.TAAP.2015.02.021
P577
2015-03-05T00:00:00Z