Defects in NADPH Oxidase Genes NOX1 and DUOX2 in Very Early Onset Inflammatory Bowel Disease
about
Genetic disorders coupled to ROS deficiencyCardiac Cell Senescence and Redox Signaling.NOX2-dependent regulation of inflammationGenetic Complexity of Crohn's Disease in Two Large Ashkenazi Jewish Families.Deficiency in Duox2 activity alleviates ileitis in GPx1- and GPx2-knockout mice without affecting apoptosis incidence in the crypt epithelium.NADPH oxidase-1 deficiency offers little protection in Salmonella typhimurium-induced typhlitis in miceAquaporin-3 mediates hydrogen peroxide-dependent responses to environmental stress in colonic epithelia.ROS in gastrointestinal inflammation: Rescue Or Sabotage?Incidence and Phenotype at Diagnosis of Very-early-onset Compared with Later-onset Paediatric Inflammatory Bowel Disease: A Population-based Study [1988-2011].Therapeutic potential of NADPH oxidase 1/4 inhibitors.Pathobiology of neutrophil-epithelial interactions.Neutrophils and the inflammatory tissue microenvironment in the mucosa.Cell Biology of Tight Junction Barrier Regulation and Mucosal Disease.Paradoxical roles of dual oxidases in cancer biology.Epithelial Reactive Oxygen Species and Risk for Very Early Onset Inflammatory Bowel Disease.NOX1 loss-of-function genetic variants in patients with inflammatory bowel disease.p40 phox -Deficient Mice Exhibit Impaired Bacterial Clearance and Enhanced Pro-inflammatory Responses during Salmonella enterica serovar Typhimurium Infection.Cell-specific Activation of the Nrf2 Antioxidant Pathway Increases Mucosal Inflammation in Acute but Not in Chronic Colitis.Epithelial Organization: The Gut and Beyond.NADPH Oxidase Deficiency: A Multisystem Approach.Clinical and Genomic Correlates of Neutrophil Reactive Oxygen Species Production in Pediatric Patients with Crohn's Disease.Analysis of the Human Mucosal Response to Cholera Reveals Sustained Activation of Innate Immune Signaling Pathways.DUAL OXIDASE: A NOVEL THERAPEUTIC TARGET IN ALLERGIC DISEASE.Quantitative interaction analysis permits molecular insights into functional NOX4 NADPH oxidase heterodimer assembly.Long-term Inflammation Transforms Intestinal Epithelial Cells of Colonic Organoids.NoxO1 Controls Proliferation of Colon Epithelial Cells.Hydrogen peroxide production by lactobacilli promotes epithelial restitution during colitis.Increased risk for inflammatory bowel disease in congenital hypothyroidism supports the existence of a shared susceptibility factor.
P2860
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P2860
Defects in NADPH Oxidase Genes NOX1 and DUOX2 in Very Early Onset Inflammatory Bowel Disease
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
@zh-my
2015年学术文章
@zh-sg
2015年學術文章
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name
Defects in NADPH Oxidase Genes ...... set Inflammatory Bowel Disease
@ast
Defects in NADPH Oxidase Genes ...... set Inflammatory Bowel Disease
@en
type
label
Defects in NADPH Oxidase Genes ...... set Inflammatory Bowel Disease
@ast
Defects in NADPH Oxidase Genes ...... set Inflammatory Bowel Disease
@en
prefLabel
Defects in NADPH Oxidase Genes ...... set Inflammatory Bowel Disease
@ast
Defects in NADPH Oxidase Genes ...... set Inflammatory Bowel Disease
@en
P2093
P2860
P921
P1476
Defects in NADPH Oxidase Genes ...... set Inflammatory Bowel Disease
@en
P2093
Abdul Elkadri
Aleixo A Muise
Anne M Griffiths
Billy Bourke
Conghui H Guo
Cornelia Thoeni
Dermot P B McGovern
Gabriella Aviello
Holm H Uhlig
Inga Peter
P2860
P304
P356
10.1016/J.JCMGH.2015.06.005
P577
2015-09-01T00:00:00Z