Inflammatory signaling in human tuberculosis granulomas is spatially organized.
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Granulomas and Inflammation: Host-Directed Therapies for Tuberculosis.Dissection of the host-pathogen interaction in human tuberculosis using a bioengineered 3-dimensional modelIntracellular growth of Mycobacterium tuberculosis after macrophage cell death leads to serial killing of host cells.Hypervirulent Mycobacterium tuberculosis strain triggers necrotic lung pathology associated with enhanced recruitment of neutrophils in resistant C57BL/6 miceA Bioengineered Three-Dimensional Cell Culture Platform Integrated with Microfluidics To Address Antimicrobial Resistance in Tuberculosis.The bacillary and macrophage response to hypoxia in tuberculosis and the consequences for T cell antigen recognition.Chemical activation of adenylyl cyclase Rv1625c inhibits growth of Mycobacterium tuberculosis on cholesterol and modulates intramacrophage signaling.Protein kinase C-delta (PKCδ), a marker of inflammation and tuberculosis disease progression in humans, is important for optimal macrophage killing effector functions and survival in mice.Nitric oxide prevents a pathogen-permissive granulocytic inflammation during tuberculosis.Macrophage form, function, and phenotype in mycobacterial infection: lessons from tuberculosis and other diseases.The Wnt Blows: On the Functional Role of Wnt Signaling in Mycobacterium tuberculosis Infection and BeyondUnravelling the networks dictating host resistance versus tolerance during pulmonary infections.Crystal nephropathies: mechanisms of crystal-induced kidney injury.Integrating Lung Physiology, Immunology, and Tuberculosis.Mycobacterium tuberculosis in the Face of Host-Imposed Nutrient Limitation.Kunkel Lecture: Fundamental immunodeficiency and its correction.NOS2-deficient mice with hypoxic necrotizing lung lesions predict outcomes of tuberculosis chemotherapy in humans.The Capacity of Mycobacterium tuberculosis To Survive Iron Starvation Might Enable It To Persist in Iron-Deprived Microenvironments of Human Granulomas.HIV-Associated Cardiovascular Disease: Role of Connexin 43.Mycobacterium tuberculosis DosR Regulon Gene Rv2004c Encodes a Novel Antigen with Pro-inflammatory Functions and Potential Diagnostic Application for Detection of Latent Tuberculosis.Mass spectrometry-based proteomic exploration of the human immune system: focus on the inflammasome, global protein secretion, and T cells.Macrophage Epithelial Reprogramming Underlies Mycobacterial Granuloma Formation and Promotes Infection.Utility of Macrophage-activated Marker CD163 for Diagnosis and Prognosis in Pulmonary Tuberculosis.The ins and outs of the Mycobacterium tuberculosis-containing vacuole.Comparison of Surface Proteomes of Adherence Variants of Listeria Monocytogenes Using LC-MS/MS for Identification of Potential Surface Adhesins.Mycobacterial infection induces higher interleukin-1β and dysregulated lung inflammation in mice with defective leukocyte NADPH oxidase.The Troika Host-Pathogen-Extrinsic Factors in Tuberculosis: Modulating Inflammation and Clinical Outcomes.Tuberculosis Infectiousness and Host Susceptibility.Immunological roulette: Luck or something more? Considering the connections between host and environment in TB.Deletion of TGF-β1 Increases Bacterial Clearance by Cytotoxic T Cells in a Tuberculosis Granuloma Model.Spatial Tissue Proteomics Quantifies Inter- and Intratumor Heterogeneity in Hepatocellular Carcinoma (HCC).Gene Enrichment Analysis Reveals Major Regulators of Mycobacterium tuberculosis Gene Expression in Two Models of Antibiotic Tolerance.Growth of Mycobacterium tuberculosis in vivo segregates with host macrophage metabolism and ontogeny.Macrophage Heterogeneity in the Immunopathogenesis of Tuberculosis.A Beneficial Effect of Low-Dose Aspirin in a Murine Model of Active Tuberculosis.Transferring knowledge of bacterial protein interaction networks to predict pathogen targeted human genes and immune signaling pathways: a case study on M. tuberculosis.Storage lipid studies in tuberculosis reveal that foam cell biogenesis is disease-specificHuman Monocytic Suppressive Cells Promote Replication of and Alter Stability of Generated GranulomasThe Immune Mechanisms of Lung Parenchymal Damage in Tuberculosis and the Role of Host-Directed TherapyCombining discovery and targeted proteomics reveals a prognostic signature in oral cancer
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P2860
Inflammatory signaling in human tuberculosis granulomas is spatially organized.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Inflammatory signaling in human tuberculosis granulomas is spatially organized.
@ast
Inflammatory signaling in human tuberculosis granulomas is spatially organized.
@en
type
label
Inflammatory signaling in human tuberculosis granulomas is spatially organized.
@ast
Inflammatory signaling in human tuberculosis granulomas is spatially organized.
@en
prefLabel
Inflammatory signaling in human tuberculosis granulomas is spatially organized.
@ast
Inflammatory signaling in human tuberculosis granulomas is spatially organized.
@en
P2093
P2860
P50
P356
P1433
P1476
Inflammatory signaling in human tuberculosis granulomas is spatially organized.
@en
P2093
Brendan Prideaux
Clifton E Barry
Eliseo A Eugenin
Eric J Rubin
Jin Hee Kim
Kirti Sharma
Laura E Via
Matthias Mann
Pei-Yu Chen
Ravikiran M Raju
P2860
P2888
P304
P356
10.1038/NM.4073
P407
P577
2016-04-04T00:00:00Z
P6179
1003867873