Amniotic fluid inhibits Toll-like receptor 4 signaling in the fetal and neonatal intestinal epithelium. - sprookjes - yvandenbroek - TriplyDB

Amniotic fluid inhibits Toll-like receptor 4 signaling in the fetal and neonatal intestinal epithelium.

Amniotic fluid inhibits Toll-like receptor 4 signaling in the fetal and neonatal intestinal epithelium.

http://www.wikidata.org/entity/Q36094184

about

Amniotic fluid: Source of trophic factors for the developing intestine Ontogeny of intestinal epithelial innate immune responses Necrotizing enterocolitis: new insights into pathogenesis and mechanisms Ciprofloxacin modulates cytokine/chemokine profile in serum, improves bone marrow repopulation, and limits apoptosis and autophagy in ileum after whole body ionizing irradiation combined with skin-wound trauma Discovery and validation of a new class of small molecule Toll-like receptor 4 (TLR4) inhibitors Reactive oxygen species are required for driving efficient and sustained aerobic glycolysis during CD4+ T cell activation.Human perinatal immunity in physiological conditions and during infection.Animal models of gastrointestinal and liver diseases. Animal models of necrotizing enterocolitis: pathophysiology, translational relevance, and challenges.Lactobacillus rhamnosus HN001 decreases the severity of necrotizing enterocolitis in neonatal mice and preterm piglets: evidence in mice for a role of TLR9.Evidence-based feeding strategies before and after the development of necrotizing enterocolitis.Update in pathogenesis and prospective in treatment of necrotizing enterocolitis.A holobiont birth narrative: the epigenetic transmission of the human microbiome Transforming growth factor-β2 and endotoxin interact to regulate homeostasis via interleukin-8 levels in the immature intestine.Necrotizing enterocolitis in newborns: update in pathophysiology and newly emerging therapeutic strategies.Maturation of the enteric mucosal innate immune system during the postnatal period.Mucosal Immune Development in Early Life: Setting the Stage.Factors influencing gastrointestinal tract and microbiota immune interaction in preterm infants.Breast milk protects against the development of necrotizing enterocolitis through inhibition of Toll-like receptor 4 in the intestinal epithelium via activation of the epidermal growth factor receptor The human milk oligosaccharide 2'-fucosyllactose attenuates the severity of experimental necrotising enterocolitis by enhancing mesenteric perfusion in the neonatal intestine Toll-like receptor 4 is expressed on intestinal stem cells and regulates their proliferation and apoptosis via the p53 up-regulated modulator of apoptosis Toll-like receptor 4-mediated lymphocyte influx induces neonatal necrotizing enterocolitis.A critical role for TLR4 induction of autophagy in the regulation of enterocyte migration and the pathogenesis of necrotizing enterocolitis Mechanisms of gut barrier failure in the pathogenesis of necrotizing enterocolitis: Toll-like receptors throw the switch.Innate immune signaling in the pathogenesis of necrotizing enterocolitis.Endothelial TLR4 activation impairs intestinal microcirculatory perfusion in necrotizing enterocolitis via eNOS-NO-nitrite signaling.Short-term surgical outcomes of preterm infants with necrotizing enterocolitis: A single-center experience.Amniotic fluid-borne hepatocyte growth factor protects rat pups against experimental necrotizing enterocolitis The Role of Mucosal Immunity in the Pathogenesis of Necrotizing Enterocolitis.Toll-like receptor 4-mediated endoplasmic reticulum stress in intestinal crypts induces necrotizing enterocolitis.ErbB receptors and their growth factor ligands in pediatric intestinal inflammation.Stem cells as a potential therapy for necrotizing enterocolitis.Toll-like receptor regulation of intestinal development and inflammation in the pathogenesis of necrotizing enterocolitis.Pathogenesis of necrotizing enterocolitis: modeling the innate immune response.Protective effects of amniotic fluid in the setting of necrotizing enterocolitis.Inflammation and Apoptosis: Dual Mediator Role for Toll-like Receptor 4 in the Development of Necrotizing Enterocolitis.LPS Induces Hyper-Permeability of Intestinal Epithelial Cells.Experimental Anti-Inflammatory Drug Semapimod Inhibits TLR Signaling by Targeting the TLR Chaperone gp96.Surfactant protein-D attenuates the lipopolysaccharide-induced inflammation in human intestinal cells overexpressing toll-like receptor 4.Probiotics, Prebiotics, and Synbiotics for the Prevention of Necrotizing Enterocolitis.Cytokines and growth factors in the developing intestine and during necrotizing enterocolitis.

P2860

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P2860

Amniotic fluid inhibits Toll-like receptor 4 signaling in the fetal and neonatal intestinal epithelium.

http://www.wikidata.org/entity/Q36094184

description

2012 nî lūn-bûn

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

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name

Amniotic fluid inhibits Toll-l ...... eonatal intestinal epithelium.

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Amniotic fluid inhibits Toll-l ...... eonatal intestinal epithelium.

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Amniotic fluid inhibits Toll-l ...... eonatal intestinal epithelium.

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Amniotic fluid inhibits Toll-l ...... eonatal intestinal epithelium.

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PNAS.1200856109

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Proceedings of the National Academy of Sciences of the United States of America

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Amniotic fluid inhibits Toll-l ...... neonatal intestinal epithelium

@en

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Amin Afrazi

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Charlotte E Egan

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Chhinder P Sodhi

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Congrong Ma

http://www.w3.org/2001/XMLSchema#string

David J Hackam

http://www.w3.org/2001/XMLSchema#string

Dennis Slagle

http://www.w3.org/2001/XMLSchema#string

Feras Alkhudari

http://www.w3.org/2001/XMLSchema#string

George K Gittes

http://www.w3.org/2001/XMLSchema#string

Hongpeng Jia

http://www.w3.org/2001/XMLSchema#string

Ibrahim Yazji

http://www.w3.org/2001/XMLSchema#string

P2860

Unravelling the pathogenesis of inflammatory bowel disease

The roles of bacteria and TLR4 in rat and murine models of necrotizing enterocolitis

Tomato lycopene extract prevents lipopolysaccharide-induced NF-kappaB signaling but worsens dextran sulfate sodium-induced colitis in NF-kappaBEGFP mice.

Toll-like receptor-4 inhibits enterocyte proliferation via impaired beta-catenin signaling in necrotizing enterocolitis.

Nucleotide-binding oligomerization domain-2 inhibits toll-like receptor-4 signaling in the intestinal epithelium.

New insights into the pathogenesis and treatment of necrotizing enterocolitis: Toll-like receptors and beyond

Specific epidermal growth factor receptor autophosphorylation sites promote mouse colon epithelial cell chemotaxis and restitution.

Intracellular heat shock protein-70 negatively regulates TLR4 signaling in the newborn intestinal epithelium.

Reciprocal expression and signaling of TLR4 and TLR9 in the pathogenesis and treatment of necrotizing enterocolitis

Diversity of microbes in amniotic fluid.

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11330-11335

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10.1073/PNAS.1200856109

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109

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2012-06-25T00:00:00Z

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228067008

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22733781

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Toll-like receptor

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3396489

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