Necdin mediates skeletal muscle regeneration by promoting myoblast survival and differentiation
about
The low-affinity receptor for neurotrophins p75NTR plays a key role for satellite cell function in muscle repair acting via RhoAMaged1, a new regulator of skeletal myogenic differentiation and muscle regeneration.Necdin controls proliferation of white adipocyte progenitor cells.Necdin, a p53-target gene, is an inhibitor of p53-mediated growth arrest.Necdin modulates proliferative cell survival of human cells in response to radiation-induced genotoxic stress.Necdin enhances myoblasts survival by facilitating the degradation of the mediator of apoptosis CCAR1/CARP1Thimerosal-induced apoptosis in mouse C2C12 myoblast cells occurs through suppression of the PI3K/Akt/survivin pathwayDeficient nitric oxide signalling impairs skeletal muscle growth and performance: involvement of mitochondrial dysregulation.Genomic profiling reveals that transient adipogenic activation is a hallmark of mouse models of skeletal muscle regeneration.Antagonistic interplay between necdin and Bmi1 controls proliferation of neural precursor cells in the embryonic mouse neocortex.Insulin/IGF-I regulation of necdin and brown adipocyte differentiation via CREB- and FoxO1-associated pathways.Nitric oxide sustains long-term skeletal muscle regeneration by regulating fate of satellite cells via signaling pathways requiring Vangl2 and cyclic GMP.RNAs of the human chromosome 15q11-q13 imprinted regionProstaglandin F2alpha promotes muscle cell survival and growth through upregulation of the inhibitor of apoptosis protein BRUCE.mTOR regulates skeletal muscle regeneration in vivo through kinase-dependent and kinase-independent mechanisms.Circulating complexes of the vitamin D binding protein with G-actin induce lung inflammation by targeting endothelial cells.Skeletal muscle regeneration in cancer cachexia.Arsenic induces apoptosis in myoblasts through a reactive oxygen species-induced endoplasmic reticulum stress and mitochondrial dysfunction pathway.Transcriptional profile of GTP-mediated differentiation of C2C12 skeletal muscle cells.TIEG1 negatively controls the myoblast pool indispensable for fusion during myogenic differentiation of C2C12 cells.Autophagy as a new therapeutic target in Duchenne muscular dystrophyDual oxidase maturation factor 1 (DUOXA1) overexpression increases reactive oxygen species production and inhibits murine muscle satellite cell differentiation.Necdin enhances muscle reconstitution of dystrophic muscle by vessel-associated progenitors, by promoting cell survival and myogenic differentiation.Recommendations for the investigation of animal models of Prader-Willi syndrome.Cullin E3 Ligase Activity Is Required for Myoblast Differentiation.Muscle dysfunction caused by loss of Magel2 in a mouse model of Prader-Willi and Schaaf-Yang syndromes.
P2860
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P2860
Necdin mediates skeletal muscle regeneration by promoting myoblast survival and differentiation
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Necdin mediates skeletal muscl ...... t survival and differentiation
@ast
Necdin mediates skeletal muscl ...... t survival and differentiation
@en
type
label
Necdin mediates skeletal muscl ...... t survival and differentiation
@ast
Necdin mediates skeletal muscl ...... t survival and differentiation
@en
prefLabel
Necdin mediates skeletal muscl ...... t survival and differentiation
@ast
Necdin mediates skeletal muscl ...... t survival and differentiation
@en
P2093
P2860
P50
P356
P1476
Necdin mediates skeletal muscl ...... t survival and differentiation
@en
P2093
Anna Innocenzi
Daniela Deponti
Giulio Cossu
Raffaella Meneveri
Silvia Baesso
Stéphanie François
P2860
P304
P356
10.1083/JCB.200701027
P407
P577
2007-10-01T00:00:00Z