Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

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Alpha-lipoic acid mitigates toxic-induced demyelination in the corpus callosum by lessening of oxidative stress and stimulation of polydendrocytes proliferation.Correction: Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

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Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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2016年學術文章

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Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

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Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

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Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

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Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

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Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

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Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

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P1476

Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

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P2093

Claudia Kemming

http://www.w3.org/2001/XMLSchema#string

Karin Hagemeier

http://www.w3.org/2001/XMLSchema#string

Marc Ehrlich

http://www.w3.org/2001/XMLSchema#string

Stefanie Albrecht

http://www.w3.org/2001/XMLSchema#string

Tanja Kuhlmann

http://www.w3.org/2001/XMLSchema#string

P2860

NMDA receptor couples Rac1-GEF Tiam1 to direct oligodendrocyte precursor cell migration

Oligodendrocyte precursor cells modulate the neuronal network by activity-dependent ectodomain cleavage of glial NG2.

Differential arterial/venous expression of NG2 proteoglycan in perivascular cells along microvessels: identifying a venule-specific phenotype.

Golli myelin basic proteins regulate oligodendroglial progenitor cell migration through voltage-gated Ca2+ influx

PDGF (alpha)-receptor is unresponsive to PDGF-AA in aortic smooth muscle cells from the NG2 knockout mouse.

Spatiotemporally different origins of NG2 progenitors produce cortical interneurons versus glia in the mammalian forebrain.

The NG2 proteoglycan promotes oligodendrocyte progenitor proliferation and developmental myelination

Neuron-glia synapses in the brain.

Glutamatergic synapses on oligodendrocyte precursor cells in the hippocampus.

The neurotoxicant, cuprizone, as a model to study demyelination and remyelination in the central nervous system.

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e0163841

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scholarly article

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10.1371/JOURNAL.PONE.0163841

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English

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P478

http://www.w3.org/2001/XMLSchema#string

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Jacqueline Trotter

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2016-10-18T00:00:00Z

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27755537

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P932

5068753

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Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

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P2860

P2860

Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

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P1476

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P2860

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P698

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P1476

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P407

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