Effects of K+-deficient diets with and without NaCl supplementation on Na+, K+, and H2O transporters' abundance along the nephron.
about
Considerations when quantitating protein abundance by immunoblotRegulation of NKCC2 activity by inhibitory SPAK isoforms: KS-SPAK is a more potent inhibitor than SPAK2Increasing plasma [K+] by intravenous potassium infusion reduces NCC phosphorylation and drives kaliuresis and natriuresis.Claudin 4 knockout mice: normal physiological phenotype with increased susceptibility to lung injury.STE20/SPS1-related proline/alanine-rich kinase (SPAK) is critical for sodium reabsorption in isolated, perfused thick ascending limb.SPAK-mediated NCC regulation in response to low-K+ dietShort-term nonpressor angiotensin II infusion stimulates sodium transporters in proximal tubule and distal nephron.Intrarenal localization of the plasma membrane ATP channel pannexin1The absence of intrarenal ACE protects against hypertensionNeed to quickly excrete K(+)? Turn off NCC.Impact of Angiotensin Type 1A Receptors in Principal Cells of the Collecting Duct on Blood Pressure and Hypertension.Role of pituitary in K+ homeostasis: impaired renal responses to altered K+ intake in hypophysectomized rats.Paracellular epithelial sodium transport maximizes energy efficiency in the kidney.Differential regulation of Na+ transporters along nephron during ANG II-dependent hypertension: distal stimulation counteracted by proximal inhibitionDietary potassium and the renal control of salt balance and blood pressure.Physiological role of SLC12 family members in the kidney.Potassium Homeostasis: The Knowns, the Unknowns, and the Health Benefits.Potassium Supplementation Prevents Sodium Chloride Cotransporter Stimulation During Angiotensin II Hypertension.Loss of WNK3 is compensated for by the WNK1/SPAK axis in the kidney of the mouse.Nephron segment-specific gene expression using AAV vectors.Renal Tubular Ubiquitin-Protein Ligase NEDD4-2 Is Required for Renal Adaptation during Long-Term Potassium Depletion.Extracellular K+ rapidly controls NaCl cotransporter phosphorylation in the native distal convoluted tubule by Cl- -dependent and independent mechanisms.Hypokalemia: a clinical update.Functional coupling of renal K+ and Na+ handling causes high blood pressure in Na+ replete mice.
P2860
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P2860
Effects of K+-deficient diets with and without NaCl supplementation on Na+, K+, and H2O transporters' abundance along the nephron.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
Effects of K+-deficient diets ...... ' abundance along the nephron.
@ast
Effects of K+-deficient diets ...... ' abundance along the nephron.
@en
type
label
Effects of K+-deficient diets ...... ' abundance along the nephron.
@ast
Effects of K+-deficient diets ...... ' abundance along the nephron.
@en
prefLabel
Effects of K+-deficient diets ...... ' abundance along the nephron.
@ast
Effects of K+-deficient diets ...... ' abundance along the nephron.
@en
P2093
P2860
P1476
Effects of K+-deficient diets ...... ' abundance along the nephron.
@en
P2093
Alicia A McDonough
Donna H Lee
Eric Delpire
Hetal Kocinsky
Mien T X Nguyen
Nicholas K Fletcher
Sebastian Bachmann
P2860
P304
P356
10.1152/AJPRENAL.00032.2012
P577
2012-04-11T00:00:00Z