Ketamine-induced neuroapoptosis in the fetal and neonatal rhesus macaque brain. - sprookjes - yvandenbroek - TriplyDB

Ketamine-induced neuroapoptosis in the fetal and neonatal rhesus macaque brain.

Ketamine-induced neuroapoptosis in the fetal and neonatal rhesus macaque brain.

http://www.wikidata.org/entity/Q36206962

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Modeling anesthetic developmental neurotoxicity using human stem cells Early ketamine exposure results in cardiac enlargement and heart dysfunction in Xenopus embryos.Sedation in Critically Ill Children with Respiratory Failure The Fas Ligand/Fas Death Receptor Pathways Contribute to Propofol-Induced Apoptosis and Neuroinflammation in the Brain of Neonatal Rats.Early Exposure to General Anesthesia with Isoflurane Downregulates Inhibitory Synaptic Neurotransmission in the Rat Thalamus.Neonatal exposure to sevoflurane may not cause learning and memory deficits and behavioral abnormality in the childhood of Cynomolgus monkeys Good gas, bad gas: isoflurane, carbon monoxide, and which is which?Anaesthetic neurotoxicity and neuroplasticity: an expert group report and statement based on the BJA Salzburg Seminar Roles of aldosterone and oxytocin in abnormalities caused by sevoflurane anesthesia in neonatal rats Ketamine alters cortical integration of GABAergic interneurons and induces long-term sex-dependent impairments in transgenic Gad67-GFP mice Multiple Anesthetic Exposure in Infant Monkeys Alters Emotional Reactivity to an Acute Stressor.Neonatal ketamine exposure causes impairment of long-term synaptic plasticity in the anterior cingulate cortex of rats Long-term neurocognitive dysfunction in offspring via NGF/ ERK/CREB signaling pathway caused by ketamine exposure during the second trimester of pregnancy in rats.Comparison of neurodegeneration and cognitive impairment in neonatal mice exposed to propofol or isoflurane.Emerging model in anesthetic developmental neurotoxicity: human stem cells.Propofol exposure in pregnant rats induces neurotoxicity and persistent learning deficit in the offspring Focus on apoptosis to decipher how alcohol and many other drugs disrupt brain development.Effects of anesthesia with isoflurane, ketamine, or propofol on physiologic parameters in neonatal rhesus macaques (Macaca mulatta).Brain regional vulnerability to anaesthesia-induced neuroapoptosis shifts with age at exposure and extends into adulthood for some regions.17β-Oestradiol Protects Primary-Cultured Rat Cortical Neurons from Ketamine-Induced Apoptosis by Activating PI3K/Akt/Bcl-2 Signalling.Repeated exposure to ketamine-xylazine during early development impairs motor learning-dependent dendritic spine plasticity in adulthood.MicroRNAs: New Players in Anesthetic-Induced Developmental Neurotoxicity Mechanistic insights into neurotoxicity induced by anesthetics in the developing brain.Efficacy of rutin in inhibiting neuronal apoptosis and cognitive disturbances in sevoflurane or propofol exposed neonatal mice.Neuropathological sequelae of developmental exposure to antiepileptic and anesthetic drugs.Propofol at clinically relevant concentrations increases neuronal differentiation but is not toxic to hippocampal neural precursor cells in vitro.Isoflurane-induced apoptosis of oligodendrocytes in the neonatal primate brain.Selective regulation of neurosteroid biosynthesis under ketamine-induced apoptosis of cortical neurons in vitro.Ketamine enhances human neural stem cell proliferation and induces neuronal apoptosis via reactive oxygen species-mediated mitochondrial pathway Propofol-induced apoptosis of neurones and oligodendrocytes in fetal and neonatal rhesus macaque brain.Ketamine induces toxicity in human neurons differentiated from embryonic stem cells via mitochondrial apoptosis pathway.Prenatal ketamine exposure causes abnormal development of prefrontal cortex in rat Long-term effects of neonatal single or multiple isoflurane exposures on spatial memory in rats.The role of anesthetic drugs in liver apoptosis.Alcohol-induced apoptosis of oligodendrocytes in the fetal macaque brain.Anticonvulsant drug-induced cell death in the developing white matter of the rodent brain.Neonatal pain control and neurologic effects of anesthetics and sedatives in preterm infants.Isoflurane-induced apoptosis of neurons and oligodendrocytes in the fetal rhesus macaque brain.Drug-Induced Apoptosis: Mechanism by which Alcohol and Many Other Drugs Can Disrupt Brain Development.Effect of intravenous patient controlled ketamine analgesiaon postoperative pain in opium abusers.

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Ketamine-induced neuroapoptosis in the fetal and neonatal rhesus macaque brain.

http://www.wikidata.org/entity/Q36206962

description

2012 nî lūn-bûn

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2012年の論文

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2012年论文

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2012年论文

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2012年论文

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name

Ketamine-induced neuroapoptosis in the fetal and neonatal rhesus macaque brain.

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Ketamine-induced neuroapoptosis in the fetal and neonatal rhesus macaque brain.

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Ketamine-induced neuroapoptosis in the fetal and neonatal rhesus macaque brain.

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Ketamine-induced neuroapoptosis in the fetal and neonatal rhesus macaque brain.

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Ketamine-induced neuroapoptosis in the fetal and neonatal rhesus macaque brain.

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Ketamine-induced neuroapoptosis in the fetal and neonatal rhesus macaque brain.

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Ketamine-induced neuroapoptosis in the fetal and neonatal rhesus macaque brain.

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Alex S Evers

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Ansgar M Brambrink

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Catherine E Creeley

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Derek J Smith

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Gregory A Dissen

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John W Olney

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Lauren D Martin

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Michael S Avidan

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Nuri B Farber

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P2860

Antiepileptic drugs and apoptotic neurodegeneration in the developing brain

Ketamine-induced neuronal cell death in the perinatal rhesus monkey

Ethanol-induced neuronal apoptosis in vivo requires BAX in the developing mouse brain

Early exposure to common anesthetic agents causes widespread neurodegeneration in the developing rat brain and persistent learning deficits.

Potential of ketamine and midazolam, individually or in combination, to induce apoptotic neurodegeneration in the infant mouse brain.

Alcohol-induced neuroapoptosis in the fetal macaque brain.

Apoptosis in the in vivo mammalian forebrain.

Ketamine anesthesia during the first week of life can cause long-lasting cognitive deficits in rhesus monkeys.

Fetal alcohol spectrum disorders: an overview with emphasis on changes in brain and behavior.

During pregnancy, recreational drug-using women stop taking ecstasy (3,4-methylenedioxy-N-methylamphetamine) and reduce alcohol consumption, but continue to smoke tobacco and cannabis: initial findings from the Development and Infancy Study.

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