Hemophagocytic macrophages in murine typhoid fever have an anti-inflammatory phenotype.
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Replication of Salmonella enterica Serovar Typhimurium in Human Monocyte-Derived MacrophagesTH1/TH2 paradigm extended: macrophage polarization as an unappreciated pathogen-driven escape mechanism?Exploiting host immunity: the Salmonella paradigmMalaria parasite infection compromises control of concurrent systemic non-typhoidal Salmonella infection via IL-10-mediated alteration of myeloid cell function.Salmonella acquires ferrous iron from haemophagocytic macrophagesTemporal and anatomical host resistance to chronic Salmonella infection is quantitatively dictated by Nramp1 and influenced by host genetic background.Salmonella enterica infection stimulates macrophages to hemophagocytosePractical murine hematopathology: a comparative review and implications for research.Increased ferroportin-1 expression and rapid splenic iron loss occur with anemia caused by Salmonella enterica Serovar Typhimurium infection in miceInfection with Salmonella enterica Serovar Typhimurium Leads to Increased Proportions of F4/80+ Red Pulp Macrophages and Decreased Proportions of B and T Lymphocytes in the Spleen.Macrophage cell death upon intracellular bacterial infection.Bacterial Stimulation of Toll-Like Receptor 4 Drives Macrophages To Hemophagocytose.Salmonella enterica causes more severe inflammatory disease in C57/BL6 Nramp1G169 mice than Sv129S6 mice.Salmonella require the fatty acid regulator PPARδ for the establishment of a metabolic environment essential for long-term persistence.Salmonella Infection Enhances Erythropoietin Production by the Kidney and Liver, Which Correlates with Elevated Bacterial BurdensChanges in macrophage function modulated by the lipid environment.Emerging Roles for RIPK1 and RIPK3 in Pathogen-Induced Cell Death and Host Immunity.HIF1A is a critical downstream mediator for hemophagocytic lymphohistiocytosis.Salmonella and the Inflammasome: Battle for Intracellular Dominance.Disseminated infections with antibiotic-resistant non-typhoidal Salmonella strains: contributions of host and pathogen factors.Siderophores in Iron Metabolism: From Mechanism to Therapy Potential.Single-cell RNA-seq ties macrophage polarization to growth rate of intracellular Salmonella.Dysbiosis-Associated Change in Host Metabolism Generates Lactate to Support Salmonella Growth.Modulation of Iron Metabolism in Response to Infection: Twists for All Tastes
P2860
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P2860
Hemophagocytic macrophages in murine typhoid fever have an anti-inflammatory phenotype.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
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2012年學術文章
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name
Hemophagocytic macrophages in murine typhoid fever have an anti-inflammatory phenotype.
@ast
Hemophagocytic macrophages in murine typhoid fever have an anti-inflammatory phenotype.
@en
type
label
Hemophagocytic macrophages in murine typhoid fever have an anti-inflammatory phenotype.
@ast
Hemophagocytic macrophages in murine typhoid fever have an anti-inflammatory phenotype.
@en
prefLabel
Hemophagocytic macrophages in murine typhoid fever have an anti-inflammatory phenotype.
@ast
Hemophagocytic macrophages in murine typhoid fever have an anti-inflammatory phenotype.
@en
P2093
P2860
P356
P1476
Hemophagocytic macrophages in murine typhoid fever have an anti-inflammatory phenotype.
@en
P2093
Corrella S Detweiler
Melissa W McCoy
Sarah M Moreland
P2860
P304
P356
10.1128/IAI.00656-12
P407
P577
2012-08-06T00:00:00Z