Zebrafish models flex their muscles to shed light on muscular dystrophies. - sprookjes - yvandenbroek - TriplyDB

Zebrafish models flex their muscles to shed light on muscular dystrophies.

Zebrafish models flex their muscles to shed light on muscular dystrophies.

http://www.wikidata.org/entity/Q36359190

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Nonhuman genetics. Genomic basis for the convergent evolution of electric organs Model organisms in the fight against muscular dystrophy: lessons from drosophila and Zebrafish Swimming into prominence: the zebrafish as a valuable tool for studying human myopathies and muscular dystrophies.Animal models of Duchenne muscular dystrophy: from basic mechanisms to gene therapy Recent advances using zebrafish animal models for muscle disease drug discovery Impaired embryonic motility in dusp27 mutants reveals a developmental defect in myofibril structure Zebrafish as a model system to study toxicology.Loss of Tropomodulin4 in the zebrafish mutant träge causes cytoplasmic rod formation and muscle weakness reminiscent of nemaline myopathy.Swimming-induced exercise promotes hypertrophy and vascularization of fast skeletal muscle fibres and activation of myogenic and angiogenic transcriptional programs in adult zebrafish.Computational approaches for understanding the diagnosis and treatment of Parkinson's disease.Skeletal muscle fiber type: using insights from muscle developmental biology to dissect targets for susceptibility and resistance to muscle disease.Current Translational Research and Murine Models For Duchenne Muscular Dystrophy.Calsequestrins in skeletal and cardiac muscle from adult Danio rerio.Gene therapy in monogenic congenital myopathies.Establishment and Characterization of a New Muscle Cell Line of Zebrafish (Danio rerio) as an In Vitro Model for Gene Expression Studies.Tmem2 regulates cell-matrix interactions that are essential for muscle fiber attachment 503unc, a small and muscle-specific zebrafish promoter.Duchenne and Becker Muscular Dystrophies: A Review of Animal Models, Clinical End Points, and Biomarker Quantification.Facioscapulohumeral Muscular Dystrophy.[Potential of the zebrafish model to study congenital muscular dystrophies].Endotoxin molecule lipopolysaccharide-induced zebrafish inflammation model: a novel screening method for anti-inflammatory drugs.A limb-girdle muscular dystrophy 2I model of muscular dystrophy identifies corrective drug compounds for dystroglycanopathies

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Zebrafish models flex their muscles to shed light on muscular dystrophies.

http://www.wikidata.org/entity/Q36359190

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

@zh-sg

2012年學術文章

@yue

2012年學術文章

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2012年學術文章

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name

Zebrafish models flex their muscles to shed light on muscular dystrophies.

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Zebrafish models flex their muscles to shed light on muscular dystrophies.

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type

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Zebrafish models flex their muscles to shed light on muscular dystrophies.

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Zebrafish models flex their muscles to shed light on muscular dystrophies.

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prefLabel

Zebrafish models flex their muscles to shed light on muscular dystrophies.

@ast

Zebrafish models flex their muscles to shed light on muscular dystrophies.

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Disease Models & Mechanisms

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Zebrafish models flex their muscles to shed light on muscular dystrophies.

@en

P2860

Skeletal muscle laminin and MDC1A: pathogenesis and treatment strategies.

Mutations affecting the cytoplasmic functions of the co-chaperone DNAJB6 cause limb-girdle muscular dystrophy

Mutations in the laminin alpha 2-chain gene (LAMA2) cause merosin-deficient congenital muscular dystrophy

Mutations in ISPD cause Walker-Warburg syndrome and defective glycosylation of α-dystroglycan

Mutations in the integrin alpha7 gene cause congenital myopathy

Type VI collagen mutations in Bethlem myopathy, an autosomal dominant myopathy with contractures

Ullrich scleroatonic muscular dystrophy is caused by recessive mutations in collagen type VI

Local restoration of dystrophin expression with the morpholino oligomer AVI-4658 in Duchenne muscular dystrophy: a single-blind, placebo-controlled, dose-escalation, proof-of-concept study

The zebrafish candyfloss mutant implicates extracellular matrix adhesion failure in laminin alpha2-deficient congenital muscular dystrophy.

Molecular basis for the high-affinity binding and stabilization of firefly luciferase by PTC124

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726-732

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scholarly article

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10.1242/DMM.010082

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6

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5

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Peter D. Currie

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2012-11-01T00:00:00Z

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232745858

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23115202

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Chin-hu̍t seng-bu̍t

biomedical investigative technique

human musculoskeletal system

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3484855

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