Human myoblast fusion requires expression of functional inward rectifier Kir2.1 channels
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Inward rectifiers and their regulation by endogenous polyaminesPlasticity of KIR channels in human smooth muscle cells from internal thoracic arteryFunctional expression of Kir2.x in human aortic endothelial cells: the dominant role of Kir2.2Golgi export of the Kir2.1 channel is driven by a trafficking signal located within its tertiary structureVoltage-dependent K+ channel beta subunits in muscle: differential regulation during postnatal development and myogenesisMembrane potential controls adipogenic and osteogenic differentiation of mesenchymal stem cells.The endocannabinoid 2-AG controls skeletal muscle cell differentiation via CB1 receptor-dependent inhibition of Kv7 channelsSpermine is fit to block inward rectifier (Kir) channels.Modulation of endothelial inward-rectifier K+ current by optical isomers of cholesterol.A new role for ion channels in myoblast fusion.Membrane hyperpolarization triggers myogenin and myocyte enhancer factor-2 expression during human myoblast differentiation.Specification of skeletal muscle differentiation by repressor element-1 silencing transcription factor (REST)-regulated Kv7.4 potassium channels.Development of a macromolecular diffusion pathway in the lens.Epidermal growth factor receptor down-regulation triggers human myoblast differentiationEffects of BKCa and Kir2.1 Channels on Cell Cycling Progression and Migration in Human Cardiac c-kit+ Progenitor CellsCdo Regulates Surface Expression of Kir2.1 K+ Channel in Myoblast DifferentiationIon channel development, spontaneous activity, and activity-dependent development in nerve and muscle cells.SDF-1α and LPA modulate microglia potassium channels through rho gtpases to regulate cell morphology.oxLDL antibody inhibits MCP-1 release in monocytes/macrophages by regulating Ca2+ /K+ channel flowKeep Your Friends Close: Cell-Cell Contact and Skeletal Myogenesis.Depolarization alters phenotype, maintains plasticity of predifferentiated mesenchymal stem cells.Mechanisms underlying Andersen's syndrome pathology in skeletal muscle are revealed in human myotubes.Differential voltage-dependent K+ channel responses during proliferation and activation in macrophages.Sustained Depolarization of the Resting Membrane Potential Regulates Muscle Progenitor Cell Growth and Maintains Stem Cell Properties In Vitro.Membrane dysfunction in Andersen-Tawil syndrome assessed by velocity recovery cycles.Potassium currents in human myogenic cells from healthy and congenital myotonic dystrophy foetusesMitochondrial dysfunction reduces the activity of KIR2.1 K channel in myoblasts impaired oxidative phosphorylation
P2860
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P2860
Human myoblast fusion requires expression of functional inward rectifier Kir2.1 channels
description
2001 nî lūn-bûn
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2001年の論文
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2001年学术文章
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2001年学术文章
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2001年学术文章
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2001年学术文章
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2001年学术文章
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2001年學術文章
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2001年學術文章
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name
Human myoblast fusion requires expression of functional inward rectifier Kir2.1 channels
@ast
Human myoblast fusion requires expression of functional inward rectifier Kir2.1 channels
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label
Human myoblast fusion requires expression of functional inward rectifier Kir2.1 channels
@ast
Human myoblast fusion requires expression of functional inward rectifier Kir2.1 channels
@en
prefLabel
Human myoblast fusion requires expression of functional inward rectifier Kir2.1 channels
@ast
Human myoblast fusion requires expression of functional inward rectifier Kir2.1 channels
@en
P2093
P2860
P356
P1476
Human myoblast fusion requires expression of functional inward rectifier Kir2.1 channels
@en
P2093
D Mordasini
J Fischer-Lougheed
L Bernheim
P2860
P304
P356
10.1083/JCB.153.4.677
P407
P577
2001-05-01T00:00:00Z