Gamma-herpesvirus latency is preferentially maintained in splenic germinal center and memory B cells
about
Promotion of tumorigenesis by heterozygous disruption of the beclin 1 autophagy geneGamma-herpesvirus latency requires T cell evasion during episome maintenanceEpstein-Barr virus provides a new paradigm: a requirement for the immediate inhibition of apoptosis.Defining immune engagement thresholds for in vivo control of virus-driven lymphoproliferationMurid herpesvirus-4 exploits dendritic cells to infect B cellsCrystal Structure of the Gamma-2 Herpesvirus LANA DNA Binding Domain Identifies Charged Surface Residues Which Impact Viral LatencyInterplay of Murine Gammaherpesvirus 68 with NF-kappaB Signaling of the HostMurine gammaherpesvirus 68 encoding open reading frame 11 targets TANK binding kinase 1 to negatively regulate the host type I interferon responseA gammaherpesvirus Bcl-2 ortholog blocks B cell receptor-mediated apoptosis and promotes the survival of developing B cells in vivoBats, Primates, and the Evolutionary Origins and Diversification of Mammalian GammaherpesvirusesMurine gamma-herpesvirus 68 latency protein M2 binds to Vav signaling proteins and inhibits B-cell receptor-induced cell cycle arrest and apoptosis in WEHI-231 B cells.Effective control of chronic gamma-herpesvirus infection by unconventional MHC Class Ia-independent CD8 T cells.Antibody-independent control of gamma-herpesvirus latency via B cell induction of anti-viral T cell responses.Inhibition of NF-kappaB activation in vivo impairs establishment of gammaherpesvirus latency.The Gammaherpesvirus m2 protein manipulates the Fyn/Vav pathway through a multidocking mechanism of assemblyA single CD8+ T cell epitope sets the long-term latent load of a murid herpesvirusGammaherpesvirus latency induces antibody-associated thrombocytopenia in mice.Experimental infection of laboratory-bred bank voles (Myodes glareolus) with murid herpesvirus 4.Perturbation of lytic and latent gammaherpesvirus infection in the absence of the inhibitory receptor CEACAM1A novel Cre recombinase imaging system for tracking lymphotropic virus infection in vivo.Mature and functional viral miRNAs transcribed from novel RNA polymerase III promoters.Virus-encoded microRNAs facilitate gammaherpesvirus latency and pathogenesis in vivoEx vivo stimulation of B cells latently infected with gammaherpesvirus 68 triggers reactivation from latencyPathogenesis of a model gammaherpesvirus in a natural hostThe murine gammaherpesvirus 68 M2 gene is required for efficient reactivation from latently infected B cells.Establishment and maintenance of long-term murine gammaherpesvirus 68 latency in B cells in the absence of CD40.Role of B-cell proliferation in the establishment of gammaherpesvirus latency.Murine gammaherpesvirus-68 glycoprotein B presents a difficult neutralization target to monoclonal antibodies derived from infected miceIn vivo imaging of murid herpesvirus-4 infection.Murine gammaherpesvirus 68 LANA is essential for virus reactivation from splenocytes but not long-term carriage of viral genome.The murine gammaherpesvirus immediate-early Rta synergizes with IRF4, targeting expression of the viral M1 superantigen to plasma cellsViral latency and its regulation: lessons from the gamma-herpesviruses.Identification of spliced gammaherpesvirus 68 LANA and v-cyclin transcripts and analysis of their expression in vivo during latent infectionMyeloid infection links epithelial and B cell tropisms of Murid Herpesvirus-4.Infection of dendritic cells by a gamma2-herpesvirus induces functional modulationProtective antigen-independent CD8 T cell memory is maintained during {gamma}-herpesvirus persistenceActivation of Vav by the gammaherpesvirus M2 protein contributes to the establishment of viral latency in B lymphocytesIllumination of murine gammaherpesvirus-68 cycle reveals a sexual transmission route from females to males in laboratory mice.Chemokine binding protein M3 of murine gammaherpesvirus 68 modulates the host response to infection in a natural hostEarly establishment of gamma-herpesvirus latency: implications for immune control.
P2860
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P2860
Gamma-herpesvirus latency is preferentially maintained in splenic germinal center and memory B cells
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
Gamma-herpesvirus latency is p ...... inal center and memory B cells
@ast
Gamma-herpesvirus latency is p ...... inal center and memory B cells
@en
type
label
Gamma-herpesvirus latency is p ...... inal center and memory B cells
@ast
Gamma-herpesvirus latency is p ...... inal center and memory B cells
@en
prefLabel
Gamma-herpesvirus latency is p ...... inal center and memory B cells
@ast
Gamma-herpesvirus latency is p ...... inal center and memory B cells
@en
P2093
P2860
P356
P1476
Gamma-herpesvirus latency is p ...... inal center and memory B cells
@en
P2093
David L Woodland
Emilio Flaño
In-Jeong Kim
Marcia A Blackman
P2860
P304
P356
10.1084/JEM.20020890
P407
P577
2002-11-01T00:00:00Z