Major histocompatibility complex class I allele-specific cooperative and competitive interactions between immune evasion proteins of cytomegalovirus.
about
Human cytomegalovirus glycoprotein UL16 causes intracellular sequestration of NKG2D ligands, protecting against natural killer cell cytotoxicityInhibition of MHC class I is a virulence factor in herpes simplex virus infection of mice.The evolution of natural killer cell receptorsMechanisms of virus immune evasion lead to development from chronic inflammation to cancer formation associated with human papillomavirus infectionCytomegalovirus m154 hinders CD48 cell-surface expression and promotes viral escape from host natural killer cell controlNodular inflammatory foci are sites of T cell priming and control of murine cytomegalovirus infection in the neonatal lungNK cell receptor/H2-Dk-dependent host resistance to viral infection is quantitatively modulated by H2q inhibitory signalsStructural elucidation of the m157 mouse cytomegalovirus ligand for Ly49 natural killer cell receptorsDirect interaction of the mouse cytomegalovirus m152/gp40 immunoevasin with RAE-1 isoformsSignal peptide-dependent inhibition of MHC class I heavy chain translation by rhesus cytomegalovirusIdentification of a mouse cytomegalovirus gene selectively targeting CD86 expression on antigen-presenting cells.Know Thyself: NK-Cell Inhibitory Receptors Prompt Self-Tolerance, Education, and Viral Control.Deficient major histocompatibility complex-linked innate murine cytomegalovirus immunity in MA/My.L-H2b mice and viral downregulation of H-2k class I proteins.Comprehensive mutational analysis of a herpesvirus gene in the viral genome context reveals a region essential for virus replicationOptimal conjugation of catechol group onto hyaluronic acid in coronary stent substrate coating for the prevention of restenosis.Mouse cytomegalovirus microRNAs dominate the cellular small RNA profile during lytic infection and show features of posttranscriptional regulation.Systemic priming-boosting immunization with a trivalent plasmid DNA and inactivated murine cytomegalovirus (MCMV) vaccine provides long-term protection against viral replication following systemic or mucosal MCMV challengeImmune evasion proteins of murine cytomegalovirus preferentially affect cell surface display of recently generated peptide presentation complexes.Human cytomegalovirus Fcγ binding proteins gp34 and gp68 antagonize Fcγ receptors I, II and IIISelective down-regulation of the NKG2D ligand H60 by mouse cytomegalovirus m155 glycoproteinFrequent coinfection of cells explains functional in vivo complementation between cytomegalovirus variants in the multiply infected hostAbsence of cross-presenting cells in the salivary gland and viral immune evasion confine cytomegalovirus immune control to effector CD4 T cells.Kaposi's sarcoma-associated herpesvirus K3 and K5 ubiquitin E3 ligases have stage-specific immune evasion roles during lytic replicationHuman cytomegalovirus protein pp71 disrupts major histocompatibility complex class I cell surface expression.Cytomegalovirus immunoevasin reveals the physiological role of "missing self" recognition in natural killer cell dependent virus control in vivo.Recombinant mouse cytomegalovirus expressing a ligand for the NKG2D receptor is attenuated and has improved vaccine properties.Reverse genetics modification of cytomegalovirus antigenicity and immunogenicity by CD8 T-cell epitope deletion and insertion.Murine cytomegalovirus interference with antigen presentation contributes to the inability of CD8 T cells to control virus in the salivary gland.Cytomegalovirus encodes a positive regulator of antigen presentationGain of virulence caused by loss of a gene in murine cytomegalovirusNatural killer cell receptors for major histocompatibility complex class I and related molecules in cytomegalovirus infection.Evaluating Human T-Cell Therapy of Cytomegalovirus Organ Disease in HLA-Transgenic Mice.Investigation of the impact of the common animal facility contaminant murine norovirus on experimental murine cytomegalovirus infectionViral modulation of NK cell immunity.Herpesviruses in periodontal diseases.MHC immunoevasins: protecting the pathogen reservoir in infection.The herpesviral Fc receptor fcr-1 down-regulates the NKG2D ligands MULT-1 and H60.Targeted deletion of regions rich in immune-evasive genes from the cytomegalovirus genome as a novel vaccine strategy.The role of NKG2D signaling in inhibition of cytotoxic T-lymphocyte lysis by the Murine cytomegalovirus immunoevasin m152/gp40A Novel MHC-I Surface Targeted for Binding by the MCMV m06 Immunoevasin Revealed by Solution NMR.
P2860
Q24303742-D4869AD9-1D18-4132-AFE9-A1DF837879D6Q24817190-64BF03CF-B386-4BB1-B9FC-D8E6231B3E32Q26782592-7200A104-E4BC-4951-B4E7-1826BE6A231FQ27002467-9BB2D897-33A6-49AD-81E3-ED6E73FBAF8BQ27329902-7E54FD39-F2FD-4235-B1E8-71CFBBFB11CDQ27331441-977215DB-3A15-484F-B3E4-BEB50EECBACAQ27342452-F95483FC-6020-4662-B597-CFAD2C9C4843Q27645060-D6ADA16F-7463-4D4C-9C36-CBD25F7B8D9DQ28594342-DCDE68D5-020A-445D-8F19-CB66628A4C14Q28757096-4C377721-7362-4A75-88BC-A783795FBDD3Q30320331-87B5390F-A81C-436F-B5E0-F340F9AD25B8Q30413217-2D00F8F4-FF9C-4E52-94D5-68CC14F4F6B1Q30441926-1304A9B0-7F87-44F4-B6FD-6011DFED0449Q31094095-0D68D66A-C901-4020-838F-1B3DD3D2037FQ31164958-12AAB2B0-2C27-4A67-BC2D-3F75D1E1CE0CQ33303003-22749A1E-4E1A-497C-AC41-2E69442F3221Q33551402-55C13F7A-7013-45A0-8378-51A60C3C9F9EQ33614398-F62E29FD-12C4-4E69-98CC-C34FF14CFE47Q33618817-18BFED0E-3A12-430C-85AF-4B86117C4BF0Q33834018-39B073DE-9D91-4E49-B6AA-B6B22AF37260Q33908684-EC6E74CC-1924-457F-8770-72FA82ED6140Q34013634-42351612-F401-4D4E-807E-672E5B10E9BCQ34059252-1C3BADDE-0ED9-47E4-B428-9392B18C2277Q34301906-D1693153-B585-477F-A7D7-F994EB5178D7Q34340605-174F7F56-84E4-4A65-893B-E125A0859081Q34360228-C7F8DEAF-5004-4641-84C7-EADFF19CF90FQ34495529-D2A401CD-9DF3-4D20-AA42-4C41D0A074CAQ34546047-852B723E-D2BC-405F-9A92-0527E907B0ECQ35023830-0DA480A8-1C28-4EF1-A53F-A04F7AE39C3BQ35123311-94E67BE1-E653-42D3-9EE2-B0D7304E4050Q35678024-52ACD0FF-316C-4B32-8CAE-8E14A180FD41Q35693349-43AB3D5D-50CD-476B-8512-90FAEFBE35A5Q35786621-1DC7BC22-FE77-45C1-97A0-EA208BFA166BQ35988761-56A5D434-B438-4CBF-9E61-60AD38BCAAD7Q36107779-5E3D774B-FA2D-47A3-9123-EE20829583D4Q36176408-974BBAD1-F3C9-48F0-AD44-20152783F686Q36228791-4325617E-069D-47D6-8DD4-B5B469F473D1Q36315171-4FAB6144-9C1E-4DC2-BC1D-01840CAD696DQ36315486-809482BB-D140-4C57-BEA1-78B8E3405FC3Q36323466-059AAB6C-0847-4AFD-9E2D-B089E7B6C4B6
P2860
Major histocompatibility complex class I allele-specific cooperative and competitive interactions between immune evasion proteins of cytomegalovirus.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
Major histocompatibility compl ...... n proteins of cytomegalovirus.
@ast
Major histocompatibility compl ...... n proteins of cytomegalovirus.
@en
type
label
Major histocompatibility compl ...... n proteins of cytomegalovirus.
@ast
Major histocompatibility compl ...... n proteins of cytomegalovirus.
@en
prefLabel
Major histocompatibility compl ...... n proteins of cytomegalovirus.
@ast
Major histocompatibility compl ...... n proteins of cytomegalovirus.
@en
P2093
P2860
P356
P1476
Major histocompatibility compl ...... n proteins of cytomegalovirus.
@en
P2093
Anja Gutermann
Jürgen Podlech
Markus Wagner
Matthias J Reddehase
Ulrich H Koszinowski
P2860
P304
P356
10.1084/JEM.20020811
P407
P577
2002-09-01T00:00:00Z