Selective corticospinal tract injury in the rat induces primary afferent fiber sprouting in the spinal cord and hyperreflexia
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Motor System Reorganization After Stroke: Stimulating and Training Toward PerfectionMotor cortex electrical stimulation augments sprouting of the corticospinal tract and promotes recovery of motor functionMotor Experience Reprograms Development of a Genetically-Altered Bilateral Corticospinal Motor CircuitOverexpression of BDNF increases excitability of the lumbar spinal network and leads to robust early locomotor recovery in completely spinalized ratsWeakened rate-dependent depression of Hoffmann's reflex and increased motoneuron hyperactivity after motor cortical infarction in mice.Focal Stroke in the Developing Rat Motor Cortex Induces Age- and Experience-Dependent Maladaptive Plasticity of Corticospinal System.Down-regulation of KCC2 expression and phosphorylation in motoneurons, and increases the number of in primary afferent projections to motoneurons in mice with post-stroke spasticity.Dendritic spine dysgenesis contributes to hyperreflexia after spinal cord injury.Transspinal direct current stimulation immediately modifies motor cortex sensorimotor maps.Participatory design in the development of an early therapy intervention for perinatal stroke.Competition with Primary Sensory Afferents Drives Remodeling of Corticospinal Axons in Mature Spinal Motor Circuits.Molecular and Cellular Mechanisms of Axonal Regeneration After Spinal Cord Injury.Role of DSCAM in the development of the spinal locomotor and sensorimotor circuits.Awake behaving electrophysiological correlates of forelimb hyperreflexia, weakness and disrupted muscular synchronization following cervical spinal cord injury in the rat.Therapeutic intraspinal microstimulation improves forelimb function after cervical contusion injury.Motoneuron intrinsic properties, but not their receptive fields, recover in chronic spinal injury.Intramuscular Neurotrophin-3 normalizes low threshold spinal reflexes, reduces spasms and improves mobility after bilateral corticospinal tract injury in rats.Rapid and persistent impairments of the forelimb motor representations following cervical deafferentation in rats.Electrical stimulation of motor cortex in the uninjured hemisphere after chronic unilateral injury promotes recovery of skilled locomotion through ipsilateral control.Alterations in the neural circuits from peripheral afferents to the spinal cord: possible implications for diabetic polyneuropathy in streptozotocin-induced type 1 diabetic rats.Hindlimb spasticity after unilateral motor cortex lesion in rats is reduced by contralateral nerve root transfer.EphA4-mediated ipsilateral corticospinal tract misprojections are necessary for bilateral voluntary movements but not bilateral stereotypic locomotion.Activity-Based Therapies for Repair of the Corticospinal System Injured during Development.Preclinical models of muscle spasticity: valuable tools in the development of novel treatment for neurological diseases and conditions.Deletion of the Fractalkine Receptor, CX3CR1, Improves Endogenous Repair, Axon Sprouting, and Synaptogenesis after Spinal Cord Injury in Mice.Opposite, bidirectional shifts in excitation and inhibition in specific types of dorsal horn interneurons are associated with spasticity and pain post-SCI.Defining recovery neurobiology of injured spinal cord by synthetic matrix-assisted hMSC implantation.Paired motor cortex and cervical epidural electrical stimulation timed to converge in the spinal cord promotes lasting increases in motor responses.Undirected compensatory plasticity contributes to neuronal dysfunction after severe spinal cord injury.Strategies to augment volitional and reflex function may improve locomotor capacity following incomplete spinal cord injury.Interlimb Dynamic after Unilateral Focal Lesion of the Cervical Dorsal Corticospinal Tract with Endothelin-1.Motor system plasticity after unilateral injury in the developing brain.Spinal cord direct current stimulation differentially modulates neuronal activity in the dorsal and ventral spinal cord.Plasticity in One Hemisphere, Control From Two: Adaptation in Descending Motor Pathways After Unilateral Corticospinal Injury in Neonatal Rats.Cerebral Ischemia Changed the Effect of Metabosensitive Muscle Afferents on Somatic Reflex Without Affecting Thalamic Activity.Simultaneous Assessment of Homonymous and Heteronymous Monosynaptic Reflex Excitability in the Adult Rat
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P2860
Selective corticospinal tract injury in the rat induces primary afferent fiber sprouting in the spinal cord and hyperreflexia
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2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
@wuu
2012年学术文章
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2012年学术文章
@zh-hans
2012年学术文章
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2012年学术文章
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2012年學術文章
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name
Selective corticospinal tract ...... spinal cord and hyperreflexia
@ast
Selective corticospinal tract ...... spinal cord and hyperreflexia
@en
Selective corticospinal tract ...... spinal cord and hyperreflexia
@en-gb
type
label
Selective corticospinal tract ...... spinal cord and hyperreflexia
@ast
Selective corticospinal tract ...... spinal cord and hyperreflexia
@en
Selective corticospinal tract ...... spinal cord and hyperreflexia
@en-gb
prefLabel
Selective corticospinal tract ...... spinal cord and hyperreflexia
@ast
Selective corticospinal tract ...... spinal cord and hyperreflexia
@en
Selective corticospinal tract ...... spinal cord and hyperreflexia
@en-gb
P2093
P2860
P1476
Selective corticospinal tract ...... spinal cord and hyperreflexia
@en
P2093
Andrew M Tan
Hiroki Kimura
John H Martin
P2860
P304
12896-12908
P356
10.1523/JNEUROSCI.6451-11.2012
P407
P577
2012-09-01T00:00:00Z