Organization of human papillomavirus productive cycle during neoplastic progression provides a basis for selection of diagnostic markers. - sprookjes - yvandenbroek - TriplyDB

Organization of human papillomavirus productive cycle during neoplastic progression provides a basis for selection of diagnostic markers.

Organization of human papillomavirus productive cycle during neoplastic progression provides a basis for selection of diagnostic markers.

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Papillomavirus genome structure, expression, and post-transcriptional regulation Reduced expression of multiple gap junction proteins is a feature of cervical dysplasia.Human Papillomaviruses; Epithelial Tropisms, and the Development of Neoplasia Role of IL-10 and TGF-β1 in local immunosuppression in HPV-associated cervical neoplasia Disruption of Bcl-2 and Bcl-xL by viral proteins as a possible cause of cancer Molecular mechanisms of HPV mediated neoplastic progression Control of human papillomavirus gene expression by alternative splicing E1--E4-mediated keratin phosphorylation and ubiquitylation: a mechanism for keratin depletion in HPV16-infected epithelium.Seroprevalence of 34 human papillomavirus types in the German general population.Human papillomavirus type 16 E1 E4-induced G2 arrest is associated with cytoplasmic retention of active Cdk1/cyclin B1 complexes.Role of the E1--E4 protein in the differentiation-dependent life cycle of human papillomavirus type 31.Nucleic acid sequence-based amplification assay for human papillomavirus mRNA detection and typing: evidence for DNA amplification.Differential methylation of the HPV 16 upstream regulatory region during epithelial differentiation and neoplastic transformation.Human papillomavirus type 16 E1circumflexE4 contributes to multiple facets of the papillomavirus life cycle.Prevalence and type distribution of human papillomavirus infection in women from North Sardinia, Italy Interactions with pocket proteins contribute to the role of human papillomavirus type 16 E7 in the papillomavirus life cycle.Construction of a full transcription map of human papillomavirus type 18 during productive viral infection.The viral E4 protein is required for the completion of the cottontail rabbit papillomavirus productive cycle in vivo AKT1 loss correlates with episomal HPV16 in vulval intraepithelial neoplasia.Human papillomavirus type 8 interferes with a novel C/EBPβ-mediated mechanism of keratinocyte CCL20 chemokine expression and Langerhans cell migration.A humanized mouse model of HPV-associated pathology driven by E7 expression E1 empty set E4 protein of human papillomavirus type 16 associates with mitochondria.E4 antibodies facilitate detection and type-assignment of active HPV infection in cervical disease Presence of E6 and E7 mRNA from human papillomavirus types 16, 18, 31, 33, and 45 in the majority of cervical carcinomas.Persistence of human papillomavirus infection: keys to malignant progression Productive infection of bovine papillomavirus type 2 in the urothelial cells of naturally occurring urinary bladder tumors in cattle and water buffaloes.Assessing the detection of human papillomavirus late mRNA in liquid base cytology samples for risk stratification of cervical disease.Virus transcript levels and cell growth rates after naturally occurring HPV16 integration events in basal cervical keratinocytes.Control of the papillomavirus early-to-late switch by differentially expressed SRp20 E6/E7 expression of human papillomavirus type 20 (HPV-20) and HPV-27 influences proliferation and differentiation of the skin in UV-irradiated SKH-hr1 transgenic mice The E7 open reading frame acts in cis and in trans to mediate differentiation-dependent activities in the human papillomavirus type 16 life cycle.Regulation of cellular miRNA expression by human papillomaviruses.Evolutionary ecology of human papillomavirus: trade-offs, coexistence, and origins of high-risk and low-risk types Stratification of HPV-induced cervical pathology using the virally encoded molecular marker E4 in combination with p16 or MCM HPV status is associated with altered PIWI-interacting RNA expression pattern in head and neck cancer.Construction of a Transcription Map for Papillomaviruses using RACE, RNase Protection, and Primer Extension Assays.Reconstruction of human papillomavirus type 16-mediated early-stage neoplasia implicates E6/E7 deregulation and the loss of contact inhibition in neoplastic progression Investigating Diagnostic Problems of CIN1 and CIN2 Associated With High-risk HPV by Combining the Novel Molecular Biomarker PanHPVE4 With P16INK4a.Functional variants of human papillomavirus type 16 demonstrate host genome integration and transcriptional alterations corresponding to their unique cancer epidemiology.HPV16 and 18 genome amplification show different E4-dependence, with 16E4 enhancing E1 nuclear accumulation and replicative efficiency via its cell cycle arrest and kinase activation functions.

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Organization of human papillomavirus productive cycle during neoplastic progression provides a basis for selection of diagnostic markers.

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Organization of human papillom ...... lection of diagnostic markers.

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Organization of human papillom ...... lection of diagnostic markers.

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JVI.77.19.10186-10201.2003

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Journal of Virology

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Organization of human papillom ...... lection of diagnostic markers.

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Amira El-Sherif

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David Jenkins

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Heather Griffin

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John Doorbar

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Karl Sotlar

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Merilyn Hibma

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Paul Lambert

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P2860

In vivo structure of two divergent promoters at the human PCNA locus. Synthesis of antisense RNA and S phase-dependent binding of E2F complexes in intron 1

Human papillomavirus infection requires cell surface heparan sulfate.

Identification of the human papilloma virus-1a E4 gene products

Identification of the alpha6 integrin as a candidate receptor for papillomaviruses

Cell growth-regulated expression of mammalian MCM5 and MCM6 genes mediated by the transcription factor E2F

Epidemiologic classification of human papillomavirus types associated with cervical cancer

Human papillomavirus is a necessary cause of invasive cervical cancer worldwide

Coexpression of the human papillomavirus type 16 E4 and L1 open reading frames in early cervical neoplasia.

A C-terminal helicase domain of the human papillomavirus E1 protein binds E2 and the DNA polymerase alpha-primase p68 subunit.

p21cip1 Degradation in differentiated keratinocytes is abrogated by costabilization with cyclin E induced by human papillomavirus E7.

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10.1128/JVI.77.19.10186-10201.2003

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77

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2003-10-01T00:00:00Z

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12970404

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228472

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