Wood smoke particles from different combustion phases induce similar pro-inflammatory effects in a co-culture of monocyte and pneumocyte cell lines. - sprookjes - yvandenbroek - TriplyDB

Wood smoke particles from different combustion phases induce similar pro-inflammatory effects in a co-culture of monocyte and pneumocyte cell lines.

Wood smoke particles from different combustion phases induce similar pro-inflammatory effects in a co-culture of monocyte and pneumocyte cell lines.

http://www.wikidata.org/entity/Q36531518

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Highway proximity and black carbon from cookstoves as a risk factor for higher blood pressure in rural China Particulate matter beyond mass: recent health evidence on the role of fractions, chemical constituents and sources of emission Acute exposure to wood smoke from incomplete combustion--indications of cytotoxicity.AMPK is required for PM2.5-induced autophagy in human lung epithelial A549 cells.Associations between ambient wood smoke and other particulate pollutants and biomarkers of systemic inflammation, coagulation and thrombosis in cardiac patients Short-term chamber exposure to low doses of two kinds of wood smoke does not induce systemic inflammation, coagulation or oxidative stress in healthy humans.Association Between Health Symptoms and Particulate Matter from Traffic and Residential Heating - Results from RHINE III in Tartu.Public health and components of particulate matter: the changing assessment of black carbon.Review of the health effects of wildland fire smoke on wildland firefighters and the public.Automated clean-up, separation and detection of polycyclic aromatic hydrocarbons in particulate matter extracts using a 2D-LC/2D-GC system: a method translation from two FIDs to two MS detectors.Wood combustion particles induce adverse effects to normal and diseased airway epithelia.Particulate matter-mediated release of long pentraxin 3 (PTX3) and vascular endothelial growth factor (VEGF) in vitro: Limited importance of endotoxin and organic content.Pro-inflammatory response and oxidative stress induced by specific components in ambient particulate matter in human bronchial epithelial cells.Wildfire smoke exposure and human health: Significant gaps in research for a growing public health issue.Toxicity of wood smoke particles in human A549 lung epithelial cells: the role of PAHs, soot and zinc.Mutagenicity and Lung Toxicity of Smoldering vs. Flaming Emissions from Various Biomass Fuels: Implications for Health Effects from Wildland Fires.Toxicity of Urban PM10 and Relation with Tracers of Biomass Burning.Changes in HPBMC markers of immmune function following controlled short-term inhalation exposures of humans to hardwood smoke.Differences between co-cultures and monocultures in testing the toxicity of particulate matter derived from log wood and pellet combustion.

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P2860

Wood smoke particles from different combustion phases induce similar pro-inflammatory effects in a co-culture of monocyte and pneumocyte cell lines.

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Roger Westerholm

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P2860

Concordance between in vitro and in vivo dosimetry in the proinflammatory effects of low-toxicity, low-solubility particles: the key role of the proximal alveolar region

Reduced lung cell proliferation following short-term exposure to ultrafine soot and iron particles in neonatal rats: key to impaired lung growth

Cytokine release from alveolar macrophages exposed to ambient particulate matter: heterogeneity in relation to size, city and season.

Physicochemical characterisation of combustion particles from vehicle exhaust and residential wood smoke.

Pathways governing G1/S transition and their response to DNA damage

Antioxidant airway responses following experimental exposure to wood smoke in man

Health effects of fine particulate air pollution: lines that connect

Woodsmoke health effects: a review

Airway epithelial cells, cytokines, and pollutants.

Regulation of interleukin-8 expression by tumor-associated stress factors.

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