Chronic human immunodeficiency virus type 1 infection stimulates distinct NF-kappa B/rel DNA binding activities in myelomonoblastic cells.

Chronic human immunodeficiency virus type 1 infection stimulates distinct NF-kappa B/rel DNA binding activities in myelomonoblastic cells.

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http://www.wikidata.org/entity/Q36651776
Regulation of human immunodeficiency virus type 1 and cytokine gene expression in myeloid cells by NF-kappa B/Rel transcription factorsTransdominant mutants of I kappa B alpha block Tat-tumor necrosis factor synergistic activation of human immunodeficiency virus type 1 gene expression and virus multiplicationExtracellular human immunodeficiency virus type 1 Tat protein is associated with an increase in both NF-kappa B binding and protein kinase C activity in primary human astrocytesDiazepam-mediated inhibition of human immunodeficiency virus type 1 expression in human brain cellsRegulation of HIV-1 transcription in cells of the monocyte-macrophage lineage.The human immunodeficiency virus type 1 Tat protein up-regulates the promoter activity of the beta-chemokine monocyte chemoattractant protein 1 in the human astrocytoma cell line U-87 MG: role of SP-1, AP-1, and NF-kappaB consensus sites.NF-kappaB cis-acting motifs of the human immunodeficiency virus (HIV) long terminal repeat regulate HIV transcription in human macrophages.Chronic human immunodeficiency virus type 1 infection of myeloid cells disrupts the autoregulatory control of the NF-kappaB/Rel pathway via enhanced IkappaBalpha degradationConstitutive expression of p50 homodimer in freshly isolated human monocytes decreases with in vitro and in vivo differentiation: a possible mechanism influencing human immunodeficiency virus replication in monocytes and mature macrophages.The kappaB sites in the human immunodeficiency virus type 1 long terminal repeat enhance virus replication yet are not absolutely required for viral growth.A family of serine proteases expressed exclusively in myelo-monocytic cells specifically processes the nuclear factor-kappa B subunit p65 in vitro and may impair human immunodeficiency virus replication in these cells.Viral induction of the human beta interferon promoter: modulation of transcription by NF-kappa B/rel proteins and interferon regulatory factorsCaveolin-1 suppresses human immunodeficiency virus-1 replication by inhibiting acetylation of NF-κB.Permanent occupancy of the human immunodeficiency virus type 1 enhancer by NF-kappa B is needed for persistent viral replication in monocytes.Protein kinase C-zeta mediates NF-kappa B activation in human immunodeficiency virus-infected monocytes.The Ras-Raf pathway is activated in human immunodeficiency virus-infected monocytes and particpates in the activation of NF-kappa B.Transcriptional activation of the interleukin-8 gene by respiratory syncytial virus infection in alveolar epithelial cells: nuclear translocation of the RelA transcription factor as a mechanism producing airway mucosal inflammation.
P2860

Chronic human immunodeficiency virus type 1 infection stimulates distinct NF-kappa B/rel DNA binding activities in myelomonoblastic cells.

Item
http://www.wikidata.org/entity/Q36651776
description
1993 nî lūn-bûn
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1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
Chronic human immunodeficiency ...... ies in myelomonoblastic cells.
@ast
Chronic human immunodeficiency ...... ies in myelomonoblastic cells.
@en
type
label
Chronic human immunodeficiency ...... ies in myelomonoblastic cells.
@ast
Chronic human immunodeficiency ...... ies in myelomonoblastic cells.
@en
prefLabel
Chronic human immunodeficiency ...... ies in myelomonoblastic cells.
@ast
Chronic human immunodeficiency ...... ies in myelomonoblastic cells.
@en
P1433
P1476
P2093
P2860
P304
P31
P407
P433
P478
P577
P698
P921
P932
P1433
P1476
Chronic human immunodeficiency ...... ties in myelomonoblastic cells
@en
P2093
P2860
P304
P31
P407
P433
P478
P577
P698
P921
P932