Flutamide induces alterations in the cell-cell junction ultrastructure and reduces the expression of Cx43 at the blood-testis barrier with no disturbance in the rat seminiferous tubule morphology

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Flutamide induces alterations in the cell-cell junction ultrastructure and reduces the expression of Cx43 at the blood-testis barrier with no disturbance in the rat seminiferous tubule morphology

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2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

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Flutamide induces alterations ...... seminiferous tubule morphology

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Flutamide induces alterations ...... seminiferous tubule morphology

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Item

label

Flutamide induces alterations ...... seminiferous tubule morphology

@ast

Flutamide induces alterations ...... seminiferous tubule morphology

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Flutamide induces alterations ...... seminiferous tubule morphology

@ast

Flutamide induces alterations ...... seminiferous tubule morphology

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P1476

Flutamide induces alterations ...... seminiferous tubule morphology

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P2093

Alicja Kaminska

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Katarzyna Chojnacka

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Laura Pardyak

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Marta Zarzycka

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Waclaw Tworzydlo

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P2860

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Regulation of blood-testis barrier by actin binding proteins and protein kinases

Selective ablation of the androgen receptor in mouse sertoli cells affects sertoli cell maturation, barrier formation and cytoskeletal development

The Mammalian Blood-Testis Barrier: Its Biology and Regulation

Endocytic processing of connexin43 gap junctions: a morphological study

Sertoli-germ cell junctions in the testis: a review of recent data

Androgen-dependent sertoli cell tight junction remodeling is mediated by multiple tight junction components.

Cytokines, polarity proteins, and endosomal protein trafficking and signaling-the sertoli cell blood-testis barrier system in vitro as a study model

Androgens regulate the permeability of the blood-testis barrier.

Autocrine androgen action is essential for Leydig cell maturation and function, and protects against late-onset Leydig cell apoptosis in both mice and men.

P2888

s12958-016-0144-2

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scholarly article

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10.1186/S12958-016-0144-2

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Barbara Bilińska

Szczepan Biliński

Anna Hejmej

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2016-03-31T00:00:00Z

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1048725046

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27036707

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4818424

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