Recruitment of the inflammatory subset of monocytes to sites of ischemia induces angiogenesis in a monocyte chemoattractant protein-1-dependent fashion.
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Communication in the heart: the role of the innate immune system in coordinating cellular responses to ischemic injuryPhagocyte-myocyte interactions and consequences during hypoxic wound healing.Vascular growth responses to chronic arterial occlusion are unaffected by myeloid specific focal adhesion kinase (FAK) deletion.Arteriolar and venular remodeling are differentially regulated by bone marrow-derived cell-specific CX3CR1 and CCR2 expressionSpatial and temporal coordination of bone marrow-derived cell activity during arteriogenesis: regulation of the endogenous response and therapeutic implications.Capillary arterialization requires the bone-marrow-derived cell (BMC)-specific expression of chemokine (C-C motif) receptor-2, but BMCs do not transdifferentiate into microvascular smooth muscle.Molecular imaging of the paracrine proangiogenic effects of progenitor cell therapy in limb ischemia.Dysregulated selectin expression and monocyte recruitment during ischemia-related vascular remodeling in diabetes mellitusUnderlying chronic inflammation alters the profile and mechanisms of acute neutrophil recruitment.Dual delivery of VEGF and MCP-1 to support endothelial cell transplantation for therapeutic vascularizationPulmonary hypertension associated with HIV infection: pulmonary vascular disease: the global perspectiveSystemic levels of G-CSF and interleukin-6 determine the angiogenic potential of bone marrow resident monocytes.The C terminus of mouse monocyte chemoattractant protein 1 (MCP1) mediates MCP1 dimerization while blocking its chemotactic potency.PEG-albumin plasma expansion increases expression of MCP-1 evidencing increased circulatory wall shear stress: an experimental study.The diverse identity of angiogenic monocytes.Protease-activated receptor (PAR)2, but not PAR1, is involved in collateral formation and anti-inflammatory monocyte polarization in a mouse hind limb ischemia model.Vascular remodeling after ischemic stroke: mechanisms and therapeutic potentialsIn vitro and in vivo effects of human monocytes and their subsets on new vessel formation.TLR4 accessory molecule RP105 (CD180) regulates monocyte-driven arteriogenesis in a murine hind limb ischemia model.Collaterals: Implications in cerebral ischemic diseases and therapeutic interventionsHorizontal gene transfer from macrophages to ischemic muscles upon delivery of naked DNA with Pluronic block copolymersThe effects of weekly augmentation therapy in patients with PiZZ α1-antitrypsin deficiency.MicroRNAs as potential novel therapeutic targets and tools for regulating paracrine function of endothelial progenitor cells.Macrophage migration arrest due to a winning balance of Rac2/Sp1 repression over β-catenin-induced PLD expressionMCP-1-induced protein promotes endothelial-like and angiogenic properties in human bone marrow monocytic cellsMonocyte transplantation for neural and cardiovascular ischemia repairMonocytes in heart failure: relationship to a deteriorating immune overreaction or a desperate attempt for tissue repair?Angiogenesis in the infarcted myocardium.Neutrophils in local and systemic antibody-dependent inflammatory and anaphylactic reactions.Monocyte chemoattractant protein-1 and the blood-brain barrier.Chemokines and their receptors in intracerebral hemorrhage.Cellular and molecular mechanisms of inflammation-induced angiogenesis.Glucose and acute exercise influence factors secreted by circulating angiogenic cells in vitro.Mouse monocyte chemoattractant protein 1 (MCP1) functions as a monomer.Immature myeloid cells are critical for enhancing bone fracture healing through angiogenic cascade.Monocytes and monocyte chemoattractant protein 1 (MCP-1) as early predictors of disease outcome in patients with cerebral ischemic stroke.Lysine acetyltransferase PCAF is a key regulator of arteriogenesis.Bone marrow-derived cells and their conditioned medium induce microvascular repair in uremic rats by stimulation of endogenous repair mechanisms.Truncation of monocyte chemoattractant protein 1 by plasmin promotes blood-brain barrier disruption.A novel flow cytometry-based assay to study leukocyte-endothelial cell interactions in vitro.
P2860
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P2860
Recruitment of the inflammatory subset of monocytes to sites of ischemia induces angiogenesis in a monocyte chemoattractant protein-1-dependent fashion.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Recruitment of the inflammator ...... t protein-1-dependent fashion.
@en
type
label
Recruitment of the inflammator ...... t protein-1-dependent fashion.
@en
prefLabel
Recruitment of the inflammator ...... t protein-1-dependent fashion.
@en
P2093
P2860
P356
P1476
Recruitment of the inflammator ...... t protein-1-dependent fashion.
@en
P2093
Alyssa D Gregory
Benjamin J Capoccia
Daniel C Link
P2860
P304
P356
10.1189/JLB.1107756
P577
2008-06-11T00:00:00Z