Capacity of Mycobacterium avium isolates to grow well or poorly in murine macrophages resides in their ability to induce secretion of tumor necrosis factor.
about
A bone marrow-derived murine macrophage model for evaluating efficacy of antimycobacterial drugs under relevant physiological conditionsTherapeutic effects of benzoxazinorifamycin KRM-1648 administered alone or in combination with a half-sized secretory leukocyte protease inhibitor or the nonsteroidal anti-inflammatory drug diclofenac sodium against Mycobacterium avium complex infecInfection with Mycobacterium avium induces production of interleukin-10 (IL-10), and administration of anti-IL-10 antibody is associated with enhanced resistance to infection in mice.Regulation of murine macrophage effector functions by lipoarabinomannan from mycobacterial strains with different degrees of virulence.Expression of virulence of Mycobacterium tuberculosis within human monocytes: virulence correlates with intracellular growth and induction of tumor necrosis factor alpha but not with evasion of lymphocyte-dependent monocyte effector functions.Host response to nontuberculous mycobacterial infections of current clinical importance.Epidemiology of infection by nontuberculous mycobacteriaRelationship between virulence of Mycobacterium avium strains and induction of tumor necrosis factor alpha production in infected mice and in in vitro-cultured mouse macrophages.Temporal effect of tumor necrosis factor alpha on murine macrophages infected with Mycobacterium aviumThe phagosomal environment protects virulent Mycobacterium avium from killing and destruction by clarithromycin.Correlation of serum tumor necrosis factor-alpha, interleukin-4 and soluble interleukin-2 receptor levels with radiologic and clinical manifestations in active pulmonary tuberculosisSearch for the molecular basis of morphological variation in Mycobacterium avium.Mycolactone-mediated inhibition of tumor necrosis factor production by macrophages infected with Mycobacterium ulcerans has implications for the control of infectionDissemination of enteric Mycobacterium avium infections in mice rendered immunodeficient by thymectomy and CD4 depletion or by prior infection with murine AIDS retroviruses.Virulence and immune response induced by Mycobacterium avium complex strains in a model of progressive pulmonary tuberculosis and subcutaneous infection in BALB/c miceUpregulation of p75 tumor necrosis factor alpha receptor in Mycobacterium avium-infected mice: evidence for a functional role.Cytokine induction in murine macrophages infected with virulent and avirulent Rhodococcus equi.Control of mycobacterial replication in human macrophages: roles of extracellular signal-regulated kinases 1 and 2 and p38 mitogen-activated protein kinase pathways.Common and unique gene expression signatures of human macrophages in response to four strains of Mycobacterium avium that differ in their growth and persistence characteristics.Minor role played by type I tumour necrosis factor receptor in the control of Mycobacterium avium proliferation in infected mice.
P2860
Q28368663-4F892EAA-D0A8-4F3D-BEF0-8E9FFC38C2BDQ28369588-F62F3ACA-8583-4B07-A906-A492B0CBA76AQ33603597-618EA34B-2731-49E7-BA14-BA2090E3BA7EQ33610521-25319642-4720-4E31-9886-06310BBA48E3Q33751770-7D9C315A-252F-4775-9791-CF39042FEDBAQ34298306-947E7437-5F38-4FC4-89A2-E4E58D2DD2A8Q35370226-FD56074C-BE8F-47ED-AE34-E79056183528Q35440803-1E18EC6C-0112-42A1-869B-696903A2C5E9Q35548643-BBC343D7-A0C1-4B51-8FCB-D8B451158A04Q35551106-25E3EB56-1B51-4C15-8B85-682F4483DF4FQ35609246-29ACAB51-A694-495A-980C-6DEBB842A8A1Q35780477-BF0F89BF-E67C-443C-ACEE-8DF6E49BA709Q35949741-A93F3A0C-1B22-44D0-AFE4-B57ADBC99CBFQ36970885-213C421A-0A30-471D-B945-B7E593B78E00Q37264835-D431B728-0ED7-4812-BC38-39F52629F9ECQ39512642-02D6EF29-3567-450D-A700-B4A8233A5E61Q39570944-389A58E6-6F90-448A-9F4F-94D0FCEAD8B8Q39656080-0DF3BCCD-5200-4E14-A51B-B7EF2C04275AQ41939112-7192B2B0-28F7-47D0-B468-83C859488A1DQ52538167-34C14EF2-68B2-4852-B9B4-A04E36264CEB
P2860
Capacity of Mycobacterium avium isolates to grow well or poorly in murine macrophages resides in their ability to induce secretion of tumor necrosis factor.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Capacity of Mycobacterium aviu ...... tion of tumor necrosis factor.
@en
type
label
Capacity of Mycobacterium aviu ...... tion of tumor necrosis factor.
@en
prefLabel
Capacity of Mycobacterium aviu ...... tion of tumor necrosis factor.
@en
P2093
P2860
P1476
Capacity of Mycobacterium aviu ...... etion of tumor necrosis factor
@en
P2093
A D Roberts
P S Skinner
R Appelberg
S K Furney
P2860
P304
P407
P577
1992-10-01T00:00:00Z