Increased expression of fibronectin and the alpha 5 beta 1 integrin in angiogenic cerebral blood vessels of mice subject to hypobaric hypoxia.
about
Integrins in the Spotlight of CancerEffect of maternal undernutrition on vascular expression of micro and messenger RNA in newborn and aging offspringMicrovascular sprouting, extension, and creation of new capillary connections with adaptation of the neighboring astrocytes in adult mouse cortex under chronic hypoxia.Human nephrosclerosis triggers a hypoxia-related glomerulopathyEndothelial alpha5 and alphav integrins cooperate in remodeling of the vasculature during developmentIn the hypoxic central nervous system, endothelial cell proliferation is followed by astrocyte activation, proliferation, and increased expression of the alpha 6 beta 4 integrin and dystroglycanPerlecan Domain V induces VEGf secretion in brain endothelial cells through integrin α5β1 and ERK-dependent signaling pathways.Microglial activation state exerts a biphasic influence on brain endothelial cell proliferation by regulating the balance of TNF and TGF-β1.Extensive vascular remodeling in the spinal cord of pre-symptomatic experimental autoimmune encephalomyelitis mice; increased vessel expression of fibronectin and the α5β1 integrin.Juvenile traumatic brain injury induces long-term perivascular matrix changes alongside amyloid-beta accumulation.PlGF knockout delays brain vessel growth and maturation upon systemic hypoxic challenge.Chronic cerebral hypoxia promotes arteriogenic remodeling events that can be identified by reduced endoglin (CD105) expression and a switch in β1 integrinsVulnerability of the developing brain to hypoxic-ischemic damage: contribution of the cerebral vasculature to injury and repair?Upregulation of vascular endothelial growth factor receptor-3 in the spinal cord of Lewis rats with experimental autoimmune encephalomyelitisThe extracellular matrix protein laminin-10 promotes blood-brain barrier repair after hypoxia and inflammation in vitro.TNF-α promotes cerebral pericyte remodeling in vitro, via a switch from α1 to α2 integrins.Upregulation of fibronectin and the α5β1 and αvβ3 integrins on blood vessels within the cerebral ischemic penumbra.An angiogenic role for the α5β1 integrin in promoting endothelial cell proliferation during cerebral hypoxia.Cerebral ischemia-induced angiogenesis is dependent on tumor necrosis factor receptor 1-mediated upregulation of α5β1 and αVβ3 integrins.Endothelial β4 integrin is predominantly expressed in arterioles, where it promotes vascular remodeling in the hypoxic brainInfluence of stress on extracellular matrix and integrin biology.The abluminal endothelial membrane in neurovascular remodeling in health and disease.Perlecan and the blood-brain barrier: beneficial proteolysis?Effects of integrins and integrin αvβ3 inhibitor on angiogenesis in cerebral ischemic stroke.The potential role of perlecan domain V as novel therapy in vascular dementia.Brain endothelial cell specific integrins and ischemic stroke.Neuron-autonomous transcriptome changes upon ischemia/reperfusion injuryThe meta-analytical paradigm in an in silico hybrid: Pathways and networks perturbed during exposure to varying degrees of hypobaric hypoxia.Oxygen-glucose deprivation (OGD) and interleukin-1 (IL-1) differentially modulate cathepsin B/L mediated generation of neuroprotective perlecan LG3 by neurons.Physiological cerebrovascular remodeling in response to chronic mild hypoxia: A role for activated protein C.Control of vascular permeability by adhesion molecules.Hereditary diffuse leukoencephalopathy with spheroids: ultrastructural and immunoelectron microscopic studies.Absence of the alpha v beta 3 integrin dictates the time-course of angiogenesis in the hypoxic central nervous system: accelerated endothelial proliferation correlates with compensatory increases in alpha 5 beta 1 integrin expressionDefining the critical hypoxic threshold that promotes vascular remodeling in the brain.Overexpression of Fibulin-5 Attenuates Ischemia/Reperfusion Injury After Middle Cerebral Artery Occlusion in Rats.Chronic mild hypoxia promotes profound vascular remodeling in spinal cord blood vessels, preferentially in white matter, via an α5β1 integrin-mediated mechanism.EphB3 signaling induces cortical endothelial cell death and disrupts the blood-brain barrier after traumatic brain injury.Hypoxia upregulates integrin gene expression in microvascular endothelial cells and promotes their migration and capillary-like tube formation.Positron emission tomography of cerebral angiogenesis and TSPO expression in a mouse model of chronic hypoxia.Mice deficient in endothelial α5 integrin are profoundly resistant to experimental ischemic stroke.
P2860
Q28066805-DC5E30D6-1BCE-4078-8D6F-6CB71A5525EDQ28576391-3A0EBD28-4807-47C5-A9D8-D42B19220943Q30570982-DFC60F57-82D7-4546-9934-289FE58E9949Q33597077-7B52C7ED-BAD7-447D-A035-7BED0C289B4FQ33929646-32B12BAD-4BBB-480F-AC7B-618680931AECQ34039076-5DB1EB7F-EFC0-4F66-9213-9865CFB2587BQ34428518-757719D2-216E-418C-ABD9-9F95B09CFBA7Q34467962-4E352FC7-A6E7-4A69-ADAE-A6B96AC19E30Q34532833-00A60DF8-9D45-430E-9537-679145B923BDQ34727104-B27B75F3-4268-4504-B383-0F4828274CF4Q35866840-3E916B43-C17E-4CD0-92C0-FAB46A4EFD64Q36211381-08A7A59F-6F0D-4455-AC86-93820FEB1C44Q36385735-0558BBBC-CFD8-4059-A2F1-BB89F0C36C17Q36500155-6CA8B8C9-37CC-4C31-B105-5100A2726FB8Q36530092-A56A3D08-3BDF-443B-9DB5-718EADD5E055Q36741459-F77476A2-4796-473E-8113-EC0BB067CE91Q37106947-EEB0A803-FAFA-4B82-A77A-BA3262673B23Q37106959-1F4689A7-A6E2-434C-BD72-FD0877279BD0Q37228378-74BAF99C-286F-4DB5-9405-953EEFB909C3Q37329753-5560CEFE-197E-46DC-9F81-6C954C5D72DBQ37845642-5572BEB1-809E-4AA3-85E0-AC418C633165Q38032676-BB472FDB-B9F7-4F06-B8BD-8339A798BC7CQ38039410-102ECE6C-4431-42BB-A6B6-D9DB299EC846Q38221073-AA4A989B-FCB6-41A7-B631-DA7DCFC6C7D8Q38223223-33F7B33D-8300-4BAA-B58B-F0788A2333C8Q38254946-6BA3433A-C22A-4A1C-9659-1466C3DFC806Q38431028-074A693E-9DF0-4092-912A-F8ABEF144DB3Q38982578-AFBCBE95-CC1F-493E-BCA1-EEE9BD286318Q40893249-81F281EA-C838-4E9E-9244-BD5FA74199FCQ41486373-3551307F-42FD-4044-A2D4-92F53CDA63E7Q41856569-ECAFC6D5-64AE-471D-BCE0-728AA0F5BF79Q42130117-6412FAAC-33CF-44C4-B0A1-D930A56FAC64Q42414710-211C0C91-B410-43B6-ADB1-BA3143FFC86FQ42556014-D29C0ABD-393A-43A5-B528-455F4AEDC334Q46722580-D8625779-ADB5-48A6-A8F0-154DE73122B9Q47233530-43D5BEFB-7B9B-44EA-966E-6787ADC997F5Q47877363-474773EC-F505-4AB6-8C24-B37D4EDDAD6FQ47916975-7965710F-2E18-446F-BC39-5D830E9A68CCQ48337362-085B9CB6-E9D3-425B-9AF7-9BB7FAA248BEQ49157396-FFC09741-19A9-4255-8CB5-FF5246695245
P2860
Increased expression of fibronectin and the alpha 5 beta 1 integrin in angiogenic cerebral blood vessels of mice subject to hypobaric hypoxia.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Increased expression of fibron ...... subject to hypobaric hypoxia.
@en
type
label
Increased expression of fibron ...... subject to hypobaric hypoxia.
@en
prefLabel
Increased expression of fibron ...... subject to hypobaric hypoxia.
@en
P2093
P2860
P1476
Increased expression of fibron ...... subject to hypobaric hypoxia.
@en
P2093
Bernadette Erokwu
Gregory J del Zoppo
Joseph C LaManna
Richard Milner
Stephanie Hung
P2860
P356
10.1016/J.MCN.2008.01.013
P577
2008-02-13T00:00:00Z