Maternal exposure to combustion generated PM inhibits pulmonary Th1 maturation and concomitantly enhances postnatal asthma development in offspring.
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Effects of prenatal inhalation exposure to copper nanoparticles on murine dams and offspringOutdoor air pollution and asthmaEnvironmentally persistent free radicals compromise left ventricular function during ischemia/reperfusion injuryBefore the first breath: prenatal exposures to air pollution and lung development.Exposure to combustion generated environmentally persistent free radicals enhances severity of influenza virus infectionRespiratory Syncytial Virus Disease Is Mediated by Age-Variable IL-33.Exposure to Deepwater Horizon Crude Oil Burnoff Particulate Matter Induces Pulmonary Inflammation and Alters Adaptive Immune ResponseExposure to environmentally persistent free radicals during gestation lowers energy expenditure and impairs skeletal muscle mitochondrial function in adult mice.Autophagy is essential for ultrafine particle-induced inflammation and mucus hyperproduction in airway epithelium.Increased Fetal Thymocytes Apoptosis Contributes to Prenatal Nicotine Exposure-induced Th1/Th2 Imbalance in Male Offspring Mice.Neonatal Immune State Is Influenced by Maternal Allergic Rhinitis and Associated With Regulatory T cellsInhaled Pollutants: The Molecular Scene behind Respiratory and Systemic Diseases Associated with Ultrafine Particulate Matter.Maternal Glucocorticoid Elevation and Associated Fetal Thymocyte Apoptosis are Involved in Immune Disorders of Prenatal Caffeine Exposed Offspring Mice.Mechanistic insight into the impact of nanomaterials on asthma and allergic airway disease.Current understanding of the toxicological risk posed to the fetus following maternal exposure to nanoparticles.Maternal Exposure of BALB/c Mice to Indoor NO2 and Allergic Asthma Syndrome in Offspring at Adulthood with Evaluation of DNA Methylation Associated Th2 Polarization.Early-life exposure to three size-fractionated ultrafine and fine atmospheric particulates in Beijing exacerbates asthma development in mature mice.The Cell Research Trends of Asthma: A Stem Frequency Analysis of the Literature
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P2860
Maternal exposure to combustion generated PM inhibits pulmonary Th1 maturation and concomitantly enhances postnatal asthma development in offspring.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on 16 July 2013
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Maternal exposure to combustio ...... thma development in offspring.
@en
Maternal exposure to combustio ...... thma development in offspring.
@nl
type
label
Maternal exposure to combustio ...... thma development in offspring.
@en
Maternal exposure to combustio ...... thma development in offspring.
@nl
prefLabel
Maternal exposure to combustio ...... thma development in offspring.
@en
Maternal exposure to combustio ...... thma development in offspring.
@nl
P2093
P2860
P356
P1476
Maternal exposure to combustio ...... sthma development in offspring
@en
P2093
Huahao Shen
Pingli Wang
P2860
P2888
P356
10.1186/1743-8977-10-29
P577
2013-07-16T00:00:00Z
P5875
P6179
1048164269