Human polymorphonuclear neutrophil responses to Burkholderia pseudomallei in healthy and diabetic subjects.
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Neutrophils in type 1 diabetesDevelopment of Burkholderia mallei and pseudomallei vaccinesPulmonary tuberculosis and delay in anti-tuberculous treatment are important risk factors for chronic obstructive pulmonary diseaseA transcriptomic reporter assay employing neutrophils to measure immunogenic activity of septic patients' plasmaToll-like receptor 4 region genetic variants are associated with susceptibility to melioidosis.Osteopontin impairs host defense during established gram-negative sepsis caused by Burkholderia pseudomallei (melioidosis).The epidemiology and clinical spectrum of melioidosis: 540 cases from the 20 year Darwin prospective studyReduced neutrophil apoptosis in diabetic mice during staphylococcal infection leads to prolonged Tnfα production and reduced neutrophil clearance.Delineating the importance of serum opsonins and the bacterial capsule in affecting the uptake and killing of Burkholderia pseudomallei by murine neutrophils and macrophagesNeutrophil elastase causes tissue damage that decreases host tolerance to lung infection with burkholderia species.Inflammasome-dependent pyroptosis and IL-18 protect against Burkholderia pseudomallei lung infection while IL-1β is deleteriousThe impact of "omic" and imaging technologies on assessing the host immune response to biodefence agents.Stable, site-specific fluorescent tagging constructs optimized for burkholderia species.The effect of environmental conditions on biofilm formation of Burkholderia pseudomallei clinical isolatesDiabetes and immunity to tuberculosis.Capsule influences the deposition of critical complement C3 levels required for the killing of Burkholderia pseudomallei via NADPH-oxidase induction by human neutrophilsDiabetes-independent increase of factor VII-activating protease activation in patients with Gram-negative sepsis (melioidosis).Identification of a predicted trimeric autotransporter adhesin required for biofilm formation of Burkholderia pseudomallei.Host susceptibility factors to bacterial infections in type 2 diabetes.Macrophage contact dependent and independent TLR4 mechanisms induce β-cell dysfunction and apoptosis in a mouse model of type 2 diabetes.Development of vaccines against burkholderia pseudomallei.T-Cell Responses Are Associated with Survival in Acute Melioidosis Patients.Macroautophagy is essential for killing of intracellular Burkholderia pseudomallei in human neutrophils.Glutathione deficiency in type 2 diabetes impairs cytokine responses and control of intracellular bacteria.Burkholderia pseudomallei triggers altered inflammatory profiles in a whole-blood model of type 2 diabetes-melioidosis comorbidityLow-level laser therapy (904nm) can increase collagen and reduce oxidative and nitrosative stress in diabetic wounded mouse skinExtended loop region of Hcp1 is critical for the assembly and function of type VI secretion system in Burkholderia pseudomallei.Neutrophil extracellular traps exhibit antibacterial activity against burkholderia pseudomallei and are influenced by bacterial and host factors.Neutrophil extracellular traps in the host defense against sepsis induced by Burkholderia pseudomallei (melioidosis)Impaired early cytokine responses at the site of infection in a murine model of type 2 diabetes and melioidosis comorbidity.Production of interleukin-27 by human neutrophils regulates their function during bacterial infection.High-glucose environment enhanced oxidative stress and increased interleukin-8 secretion from keratinocytes: new insights into impaired diabetic wound healingGlibenclamide impairs responses of neutrophils against Burkholderia pseudomallei by reduction of intracellular glutathione.Glibenclamide reduces pro-inflammatory cytokine production by neutrophils of diabetes patients in response to bacterial infectionWhen two is better than one: macrophages and neutrophils work in concert in innate immunity as complementary and cooperative partners of a myeloid phagocyte system.Reduced immune cell infiltration and increased pro-inflammatory mediators in the brain of Type 2 diabetic mouse model infected with West Nile virus.Immunological mechanisms contributing to the double burden of diabetes and intracellular bacterial infections.Acquired immunodeficiencies and tuberculosis: focus on HIV/AIDS and diabetes mellitus.Boosting of post-exposure human T-cell and B-cell recall responses in vivo by Burkholderia pseudomallei-related proteins.Role of Canonical and Non-canonical Inflammasomes During Burkholderia Infection.
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Human polymorphonuclear neutrophil responses to Burkholderia pseudomallei in healthy and diabetic subjects.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 27 October 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Human polymorphonuclear neutro ...... healthy and diabetic subjects.
@en
Human polymorphonuclear neutro ...... healthy and diabetic subjects.
@nl
type
label
Human polymorphonuclear neutro ...... healthy and diabetic subjects.
@en
Human polymorphonuclear neutro ...... healthy and diabetic subjects.
@nl
prefLabel
Human polymorphonuclear neutro ...... healthy and diabetic subjects.
@en
Human polymorphonuclear neutro ...... healthy and diabetic subjects.
@nl
P2093
P2860
P356
P1476
Human polymorphonuclear neutro ...... healthy and diabetic subjects.
@en
P2093
Chidchamai Kewcharoenwong
Manabu Ato
Sujin Chanchamroen
Wattanachai Susaengrat
P2860
P304
P356
10.1128/IAI.00503-08
P407
P577
2008-10-27T00:00:00Z