Recurrent Sleep Fragmentation Induces Insulin and Neuroprotective Mechanisms in Middle-Aged Flies. - sprookjes - yvandenbroek - TriplyDB

Recurrent Sleep Fragmentation Induces Insulin and Neuroprotective Mechanisms in Middle-Aged Flies.

Recurrent Sleep Fragmentation Induces Insulin and Neuroprotective Mechanisms in Middle-Aged Flies.

http://www.wikidata.org/entity/Q37143865

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Sleep mediates the association between homocysteine and oxidative status in mild cognitive impairment.Ingestion of green tea with lowered caffeine improves sleep quality of the elderly via suppression of stress.

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Recurrent Sleep Fragmentation Induces Insulin and Neuroprotective Mechanisms in Middle-Aged Flies.

http://www.wikidata.org/entity/Q37143865

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 02 August 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Recurrent Sleep Fragmentation ...... chanisms in Middle-Aged Flies.

@en

Recurrent Sleep Fragmentation ...... chanisms in Middle-Aged Flies.

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type

label

Recurrent Sleep Fragmentation ...... chanisms in Middle-Aged Flies.

@en

Recurrent Sleep Fragmentation ...... chanisms in Middle-Aged Flies.

@nl

prefLabel

Recurrent Sleep Fragmentation ...... chanisms in Middle-Aged Flies.

@en

Recurrent Sleep Fragmentation ...... chanisms in Middle-Aged Flies.

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FNAGI.2016.00180

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Frontiers in Aging Neuroscience

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Recurrent Sleep Fragmentation ...... chanisms in Middle-Aged Flies.

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Daniel Erlandsson

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Ella Potter

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Josef Carlsson

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Loora Laan

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Michael J Williams

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Robert Frediksson

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Tabusi Mahebali

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Accurate normalization of real-time quantitative RT-PCR data by geometric averaging of multiple internal control genes

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Glatiramer acetate fights against Alzheimer's disease by inducing dendritic-like microglia expressing insulin-like growth factor 1

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

ER stress-induced cell death mechanisms

The circadian clock mutation alters sleep homeostasis in the mouse

Cytochrome C is tyrosine 97 phosphorylated by neuroprotective insulin treatment

Changes in components of energy regulation in mouse cortex with increases in wakefulness

Changes in endoplasmic reticulum stress proteins and aldolase A in cells exposed to dopamine

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180

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scholarly article

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10.3389/FNAGI.2016.00180

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8

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Helgi B. Schiöth

Mikaela M Ceder

Christian Benedict

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2016-08-02T00:00:00Z

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27531979

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