TGFBI deficiency predisposes mice to spontaneous tumor development
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Periostin shows increased evolutionary plasticity in its alternatively spliced regionIdentification of PITX1 as a TERT suppressor gene located on human chromosome 5Periostin and TGF-β-induced protein: Two peas in a pod?Role of integrins in the periodontal ligament: organizers and facilitatorsExpression analysis of human pterygium shows a predominance of conjunctival and limbal markers and genes associated with cell migration.Mechanism of radiation carcinogenesis: role of the TGFBI gene and the inflammatory signaling cascade.Transforming growth factor-β-induced protein (TGFBI) suppresses mesothelioma progression through the Akt/mTOR pathwayCD248 facilitates tumor growth via its cytoplasmic domain.Prevention of mutation, cancer, and other age-associated diseases by optimizing micronutrient intake.The role of TGFBI in mesothelioma and breast cancer: association with tumor suppression.Systems analysis of a mouse xenograft model reveals annexin A1 as a regulator of gene expression in tumor stroma.Discoidin domain receptor 1 contributes to tumorigenesis through modulation of TGFBI expression.MicroRNA-mediated regulation of extracellular matrix formation modulates somatic cell reprogrammingTGFB1-induced extracellular expression of TGFBIp and inhibition of TGFBIp expression by RNA interference in a human corneal epithelial cell line.MiR-21 plays an important role in radiation induced carcinogenesis in BALB/c mice by directly targeting the tumor suppressor gene Big-h3.Organized proteomic heterogeneity in colorectal cancer liver metastases and implications for therapies.RecQL4 helicase amplification is involved in human breast tumorigenesis.Transforming properties of 8p11-12 amplified genes in human breast cancer.Association of chromosome damage detected as micronuclei with hematological diseases and micronutrient status.TGFβ-mediated suppression of CD248 in non-cancer cells via canonical Smad-dependent signaling pathways is uncoupled in cancer cellsGallotannin imposes S phase arrest in breast cancer cells and suppresses the growth of triple-negative tumors in vivo.TGFBI expression reduces in vitro and in vivo metastatic potential of lung and breast tumor cells.ADAM17 Promotes Motility, Invasion, and Sprouting of Lymphatic Endothelial Cells.Development of a Transgenic Mouse with R124H Human TGFBI Mutation Associated with Granular Corneal Dystrophy Type 2.Proteomic identification of betaig-h3 as a lysophosphatidic acid-induced secreted protein of human mesenchymal stem cells: paracrine activation of A549 lung adenocarcinoma cells by betaig-h3.Global transcriptome analysis of human bone marrow stromal cells (BMSC) reveals proliferative, mobile and interactive cells that produce abundant extracellular matrix proteins, some of which may affect BMSC potency.SPARC Regulates Transforming Growth Factor Beta Induced (TGFBI) Extracellular Matrix Deposition and Paclitaxel Response in Ovarian Cancer Cells.Proteomic Analysis Implicates Dominant Alterations of RNA Metabolism and the Proteasome Pathway in the Cellular Response to Carbon-Ion IrradiationMiR-9 is overexpressed in spontaneous canine osteosarcoma and promotes a metastatic phenotype including invasion and migration in osteoblasts and osteosarcoma cell lines.Transforming growth Factor-Beta-Induced Protein (TGFBI)/(βig-H3): a matrix protein with dual functions in ovarian cancer.ß3 integrin modulates transforming growth factor beta induced (TGFBI) function and paclitaxel response in ovarian cancer cells.Matrix metalloproteinase 9 (MMP-9)-dependent processing of βig-h3 protein regulates cell migration, invasion, and adhesionRelease of TGFβig-h3 by gastric myofibroblasts slows tumor growth and is decreased with cancer progression.Wnt3 function in the epiblast is required for the maintenance but not the initiation of gastrulation in mice.Intercellular communication of cellular stress monitored by gamma-H2AX induction.Role of TGFBIp in Wound Healing and Mucin Expression in Corneal Epithelial Cells.Bigh3 silencing increases retinoblastoma tumor growth in the murine SV40-TAg-Rb model.TGF-β Signaling in Gastrointestinal Cancers: Progress in Basic and Clinical Research.Transcriptional regulation of Wnt inhibitory factor-1 by Miz-1/c-Myc.Transforming growth factor-beta-induced protein secreted by peritoneal cells increases the metastatic potential of ovarian cancer cells.
P2860
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P2860
TGFBI deficiency predisposes mice to spontaneous tumor development
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on January 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
TGFBI deficiency predisposes mice to spontaneous tumor development
@en
TGFBI deficiency predisposes mice to spontaneous tumor development.
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type
label
TGFBI deficiency predisposes mice to spontaneous tumor development
@en
TGFBI deficiency predisposes mice to spontaneous tumor development.
@nl
prefLabel
TGFBI deficiency predisposes mice to spontaneous tumor development
@en
TGFBI deficiency predisposes mice to spontaneous tumor development.
@nl
P2093
P2860
P1433
P1476
TGFBI deficiency predisposes mice to spontaneous tumor development
@en
P2093
Adayabalam S Balajee
Chyuansheng Lin
Cuidong Wang
Gengyun Wen
Genze Shao
Govind Bhagat
Hongbo Fang
Yongliang Zhao
P2860
P356
10.1158/0008-5472.CAN-08-1648
P407
P577
2009-01-01T00:00:00Z